Thomas D. Pugh

4.7k total citations · 1 hit paper
50 papers, 3.8k citations indexed

About

Thomas D. Pugh is a scholar working on Molecular Biology, Oncology and Physiology. According to data from OpenAlex, Thomas D. Pugh has authored 50 papers receiving a total of 3.8k indexed citations (citations by other indexed papers that have themselves been cited), including 18 papers in Molecular Biology, 14 papers in Oncology and 14 papers in Physiology. Recurrent topics in Thomas D. Pugh's work include Genetics, Aging, and Longevity in Model Organisms (12 papers), Adipose Tissue and Metabolism (8 papers) and Cancer-related Molecular Pathways (6 papers). Thomas D. Pugh is often cited by papers focused on Genetics, Aging, and Longevity in Model Organisms (12 papers), Adipose Tissue and Metabolism (8 papers) and Cancer-related Molecular Pathways (6 papers). Thomas D. Pugh collaborates with scholars based in United States, Japan and Norway. Thomas D. Pugh's co-authors include Richard Weindruch, Stanley Goldfarb, Tomas A. Prolla, Shinichi Someya, Tatsuya Yamasoba, Masaru Tanokura, Gregory C. Kujoth, Jason D. Morrow, Rachel Sullivan and Stephanie E. Wohlgemuth and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Thomas D. Pugh

49 papers receiving 3.6k citations

Hit Papers

Mitochondrial DNA Mutations, Oxidative Stress, and Apopto... 2005 2026 2012 2019 2005 500 1000 1.5k

Peers

Thomas D. Pugh
Comparison fields: 5 of 137
  • Molecular Biology 2.1k
  • Physiology 1.1k
  • Aging 554
  • Epidemiology 380
  • Clinical Biochemistry 363
Replace Nicolle Sitte with:
Nicolle Sitte Germany
Simon C. Johnson United States
Thomas J. Corydon Denmark
Weiwei Fan United States
Yves Malthièry France
Tsuyoshi Kayo Japan
Axel Kowald Germany
Vladislav Petyuk United States
Hao Li China
Yuji Ikeno United States
Nicolle Sitte Germany View profile →
Citations per field, relative to Thomas D. Pugh
Thomas D. Pugh · 1×
Citations per year, relative to Thomas D. Pugh
Thomas D. Pugh · 1×

Countries citing papers authored by Thomas D. Pugh

Since Specialization
Citations

This map shows the geographic impact of Thomas D. Pugh's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Thomas D. Pugh with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Thomas D. Pugh more than expected).

Fields of papers citing papers by Thomas D. Pugh

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Thomas D. Pugh. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Thomas D. Pugh. The network helps show where Thomas D. Pugh may publish in the future.

Co-authorship network of co-authors of Thomas D. Pugh

This figure shows the co-authorship network connecting the top 25 collaborators of Thomas D. Pugh. A scholar is included among the top collaborators of Thomas D. Pugh based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Thomas D. Pugh. Thomas D. Pugh is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
# Work Indexed citations
1 33
2 22
3 35
4 69
5 78
6 108
7 72
8 146
9 43
10 7
11 43
12 15
13
Growth kinetics of microscopic hepatocellular neoplasms in carcinogen-resistant and carcinogen-responsive strains of mice.
13
14
Ovariectomy accelerates the growth of microscopic hepatocellular neoplasms in the mouse: possible association with whole body growth and fat deposition.
19
15
Focal impairment of growth in hepatocellular neoplasms of C57BL/6 mice: a possible explanation for the strain's resistance to hepatocarcinogenesis.
15
16 14
17 53
18
gamma-Glutamyl transpeptidase and malignant transformation of cultured liver cells.
38
19
Quantitative histochemical and autoradiographic studies of hepatocarcinogenesis in rats fed 2-acetylaminofluorene followed by phenobarbital.
132
20 13

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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