Noriyasu Seki
About
Noriyasu Seki is a scholar working on Immunology, Molecular Biology and Rheumatology.
According to data from OpenAlex, Noriyasu Seki has authored 26 papers receiving a total of 693 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Immunology, 12 papers in Molecular Biology and 10 papers in Rheumatology. Recurrent topics in Noriyasu Seki's work include Immune Cell Function and Interaction (9 papers), Sphingolipid Metabolism and Signaling (9 papers) and T-cell and B-cell Immunology (7 papers). Noriyasu Seki is often cited by papers focused on Immune Cell Function and Interaction (9 papers), Sphingolipid Metabolism and Signaling (9 papers) and T-cell and B-cell Immunology (7 papers). Noriyasu Seki collaborates with scholars based in Japan, United States and South Africa. Noriyasu Seki's co-authors include Masato Kubo, Kenji Chiba, Katsuhiko Hayashi, Kunio Sugahara, Hirotoshi Kataoka, Tsutomu Takeuchi, Jun Tsukada, Akira Matsumoto, Tadamitsu Kishimoto and Akihiko Yoshimura and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Experimental Medicine and SHILAP Revista de lepidopterología.
In The Last Decade
Noriyasu Seki
26 papers receiving 682 citations
Peers
Noriyasu Seki
Nathalie Castéran France
Graeme P. Sullivan Ireland
Paola Pittoni Italy
Laurent Frenzel France
Nicolas Chamberlain United States
Etsuko Fujii United States
Kursteen S. Price United States
Shuji Kaga Japan
Elizabeth R. Walsh United States
Wondossen Sime Sweden
Citations per year, relative to Noriyasu Seki Noriyasu Seki (= 1×)
peers
Nathalie Castéran
Countries citing papers authored by Noriyasu Seki
Since
Specialization
Citations
This map shows the geographic impact of Noriyasu Seki's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Noriyasu Seki with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Noriyasu Seki more than expected).
Fields of papers citing papers by Noriyasu Seki
Since
Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences
This network shows the impact of papers produced by Noriyasu Seki. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Noriyasu Seki. The network helps show where Noriyasu Seki may publish in the future.
Co-authorship network of co-authors of Noriyasu Seki
This figure shows the co-authorship network connecting the top 25 collaborators of Noriyasu Seki. A scholar is included among the top collaborators of Noriyasu Seki based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Noriyasu Seki. Noriyasu Seki is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
1.
Ono, Yūichi, Ryuta Saito, Noriyasu Seki, et al.. (2024). Interleukin-17A is a potential therapeutic target predicted by proteomics for systemic sclerosis patients at high risk of pulmonary arterial hypertension. Scientific Reports. 14(1). 29484–29484.
2.
Seki, Noriyasu, Sho Ishigaki, Hiroshi Takei, et al.. (2024). Th17/IL-17A axis is critical for pulmonary arterial hypertension (PAH) in systemic sclerosis (SSc): SSc patients with high levels of serum IL-17A exhibit reduced lung functions and increased prevalence of PAH. Cytokine. 176. 156534–156534.
3.
Seki, Noriyasu, Shuntaro Saito, Jun Kikuchi, et al.. (2022). Role of interferons (IFNs) in the differentiation of T peripheral helper (Tph) cells. International Immunology. 34(10). 533–544.
4.
Seki, Noriyasu, Fumihiko Miyoshi, Kunio Sugahara, et al.. (2022). Role of interferons (IFNs) in the differentiation of T peripheral helper (Tph) cells. International Immunology. 34(10). 519–532.
5.
Hanaoka, Hironari, Jun Kikuchi, Yuko Kaneko, et al.. (2021). Proton Pump Inhibitor and Tacrolimus Uses are Associated With Hypomagnesemia in Connective Tissue Disease: a Potential Link With Renal Dysfunction and Recurrent Infection. Frontiers in Pharmacology. 12. 616719–616719.
6.
Matsumoto, Kotaro, Katsuya Suzuki, Keiko Yoshimoto, et al.. (2020). Longitudinal immune cell monitoring identified CD14++ CD16+ intermediate monocyte as a marker of relapse in patients with ANCA-associated vasculitis. Arthritis Research & Therapy. 22(1). 145–145.
7.
Saito, Shuntaro, Jun Kikuchi, Masaru Takeshita, et al.. (2020). Milk fat globule epidermal growth factor 8 (MFG-E8) on monocytes is a novel biomarker of disease activity in systemic lupus erythematosus. Lupus. 30(1). 61–69.
8.
Matsumoto, Kotaro, Katsuya Suzuki, Keiko Yoshimoto, et al.. (2019). Significant association between clinical characteristics and changes in peripheral immuno-phenotype in large vessel vasculitis. Arthritis Research & Therapy. 21(1). 304–304.
9.
Maeda, Yasuhiro, Noriyasu Seki, Hirotoshi Kataoka, et al.. (2015). IL-17–Producing Vγ4+ γδ T Cells Require Sphingosine 1-Phosphate Receptor 1 for Their Egress from the Lymph Nodes under Homeostatic and Inflammatory Conditions. The Journal of Immunology. 195(4). 1408–1416.
10.
Chiba, Kenji, Yasuhiro Maeda, Noriyasu Seki, Hirotoshi Kataoka, & Kunio Sugahara. (2014). Role of sphingosine 1-phosphate (S1P) and effects of fingolimod, an S1P receptor 1 functional antagonist in lymphocyte circulation and autoimmune diseases. SHILAP Revista de lepidopterología. 1(4). 162–182.
11.
Ishibuchi, Seigo, Koichi Oshita, Noriyasu Seki, et al.. (2013). A New Phenylpyrazoleanilide, Y-320, Inhibits Interleukin 17 Production and Ameliorates Collagen-Induced Arthritis in Mice and Cynomolgus Monkeys. Pharmaceuticals. 7(1). 1–17.
12.
Seki, Noriyasu, Hirotoshi Kataoka, Kunio Sugahara, Atsushi Fukunari, & Kenji Chiba. (2013). Role of Sphingosine 1-Phosphate (S1P) Receptor 1 in Experimental Autoimmune Encephalomyelitis —II. Pharmacology & Pharmacy. 4(8). 638–646.
13.
Chiba, Kenji, Hirotoshi Kataoka, Noriyasu Seki, et al.. (2010). Fingolimod (FTY720), sphingosine 1-phosphate receptor modulator, shows superior efficacy as compared with interferon-β in mouse experimental autoimmune encephalomyelitis. International Immunopharmacology. 11(3). 366–372.
14.
Kataoka, Hirotoshi, Kyoko Shimano, Noriyasu Seki, et al.. (2010). Fingolimod (FTY720) ameliorates experimental autoimmune encephalomyelitis (EAE): I. Oral administration of FTY720 effectively inhibits relapse of EAE. Inflammation and Regeneration. 30(5). 451–457.
15.
Seki, Noriyasu, et al.. (2010). Fingolimod (FTY720) ameliorates experimental autoimmune encephalomyelitis (EAE): II. FTY720 decreases infiltration of Th17 and Th1 cells into the central nervous system in EAE. Inflammation and Regeneration. 30(6). 542–548.
16.
Chiba, Kenji, Hirotoshi Kataoka, Yasuhiro Maeda, Noriyasu Seki, & Kunio Sugahara. (2010). Sphingosine 1-phosphate receptor modulator, fingolimod (FTY720), provides a new therapeutic approach for autoimmune diseases. Inflammation and Regeneration. 30(5). 419–424.
17.
Seki, Noriyasu, Wataru Suzuki, Katsuhiko Hayashi, et al.. (2004). IL-4-Induced GATA-3 Expression Is a Time-Restricted Instruction Switch for Th2 Cell Differentiation. The Journal of Immunology. 172(10). 6158–6166.
18.
Hayashi, Keitaro, Waka Natsume, Toshio Watanabe, et al.. (2003). The Runx1 Transcription Factor Inhibits the Differentiation of Naive CD4 + T Cells into the Th2 Lineage by Repressing GATA3 Expression. The Journal of Experimental Medicine. 198(1). 51–61.
19.
Yagi, Ryoji, Wataru Suzuki, Noriyasu Seki, et al.. (2002). The IL-4 production capability of different strains of naive CD4+ T cells controls the direction of the Th cell response. International Immunology. 14(1). 1–11.
20.
Hayashi, Katsuhiko, Akira Matsumoto, Noriyasu Seki, et al.. (2002). Expression of the suppressor of cytokine signaling-5 (SOCS5) negatively regulates IL-4-dependent STAT6 activation and Th2 differentiation. Proceedings of the National Academy of Sciences. 99(20). 13003–13008.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.
Explore authors with similar magnitude of impact
Breakdown of academic impact, for papers by Nathalie Castéran Breakdown of academic impact, for papers by Graeme P. Sullivan Breakdown of academic impact, for papers by Paola Pittoni Breakdown of academic impact, for papers by Laurent Frenzel Breakdown of academic impact, for papers by Nicolas Chamberlain Breakdown of academic impact, for papers by Etsuko Fujii Breakdown of academic impact, for papers by Kursteen S. Price Breakdown of academic impact, for papers by Shuji Kaga Breakdown of academic impact, for papers by Elizabeth R. Walsh Breakdown of academic impact, for papers by Wondossen Sime