Tammy Calhoun‐Davis

4.0k total citations · 1 hit paper
17 papers, 3.0k citations indexed

About

Tammy Calhoun‐Davis is a scholar working on Oncology, Molecular Biology and Pulmonary and Respiratory Medicine. According to data from OpenAlex, Tammy Calhoun‐Davis has authored 17 papers receiving a total of 3.0k indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Oncology, 8 papers in Molecular Biology and 7 papers in Pulmonary and Respiratory Medicine. Recurrent topics in Tammy Calhoun‐Davis's work include Cancer Cells and Metastasis (12 papers), Prostate Cancer Treatment and Research (7 papers) and Immunotherapy and Immune Responses (4 papers). Tammy Calhoun‐Davis is often cited by papers focused on Cancer Cells and Metastasis (12 papers), Prostate Cancer Treatment and Research (7 papers) and Immunotherapy and Immune Responses (4 papers). Tammy Calhoun‐Davis collaborates with scholars based in United States, China and Germany. Tammy Calhoun‐Davis's co-authors include Dean G. Tang, Xin Chen, Lubna Patrawala, Collene Jeter, Can Liu, Hangwen Li, Bigang Liu, Sofia Honorio, Kevin Kelnar and Jason F. Wiggins and has published in prestigious journals such as Journal of Biological Chemistry, Nature Medicine and Nature Communications.

In The Last Decade

Tammy Calhoun‐Davis

17 papers receiving 3.0k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

The microRNA miR-34a inhibits prostate cancer stem cells and metastasis by directly repressing CD44

2011 1.1k citations Can Liu, Kevin Kelnar et al. Nature Medicine profile →

Peers

Tammy Calhoun‐Davis

ONCOL

PRM

IMMUN

Collene Jeter United States

Kurt W. Evans United States

Lubna Patrawala United States

Hyejin Choi United States

Anne‐Marie Cleton‐Jansen Netherlands

Rachel Tsan United States

María Soledad Sosa United States

Sharmila A. Bapat India

Lynn M.G. Gardner United States

Xiaoguang Fang United States

Collene Jeter United States

Tammy Calhoun‐Davis

2.2k ×1.1

1.3k ×1.0

ONCOL

1.4k ×1.0

673 ×0.9

PRM

215 ×0.9

IMMUN

Citations per year, relative to Tammy Calhoun‐Davis Tammy Calhoun‐Davis (= 1×) peers Collene Jeter

Countries citing papers authored by Tammy Calhoun‐Davis

Since Specialization

Citations

This map shows the geographic impact of Tammy Calhoun‐Davis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Tammy Calhoun‐Davis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Tammy Calhoun‐Davis more than expected).

Fields of papers citing papers by Tammy Calhoun‐Davis

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Tammy Calhoun‐Davis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Tammy Calhoun‐Davis. The network helps show where Tammy Calhoun‐Davis may publish in the future.

Co-authorship network of co-authors of Tammy Calhoun‐Davis

This figure shows the co-authorship network connecting the top 25 collaborators of Tammy Calhoun‐Davis. A scholar is included among the top collaborators of Tammy Calhoun‐Davis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Tammy Calhoun‐Davis. Tammy Calhoun‐Davis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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17 of 17 papers shown

Chen, Xin, Qiuhui Li, Xin Liu, et al.. (2016). Defining a Population of Stem-like Human Prostate Cancer Cells That Can Generate and Propagate Castration-Resistant Prostate Cancer. Clinical Cancer Research. 22(17). 4505–4516. 75 indexed citations

Jeter, Collene, Bigang Liu, Yue Lu, et al.. (2016). NANOG reprograms prostate cancer cells to castration resistance via dynamically repressing and engaging the AR/FOXA1 signaling axis. Cell Discovery. 2(1). 16041–16041. 42 indexed citations

Zhang, Dingxiao, Daechan Park, Yi Zhong, et al.. (2016). Stem cell and neurogenic gene-expression profiles link prostate basal cells to aggressive prostate cancer. Nature Communications. 7(1). 10798–10798. 148 indexed citations

Atanassov, Boyko S., Ryan D. Mohan, Xianjiang Lan, et al.. (2016). ATXN7L3 and ENY2 Coordinate Activity of Multiple H2B Deubiquitinases Important for Cellular Proliferation and Tumor Growth. Molecular Cell. 62(4). 558–571. 96 indexed citations

Liu, Ruifang, Can Liu, Dingxiao Zhang, et al.. (2016). miR-199a-3p targets stemness-related and mitogenic signaling pathways to suppress the expansion and tumorigenic capabilities of prostate cancer stem cells. Oncotarget. 7(35). 56628–56642. 45 indexed citations

Zhang, Dingxiao, Kevin Lin, Yue Lu, et al.. (2016). Developing a Novel Two-Dimensional Culture System to Enrich Human Prostate Luminal Progenitors that Can Function as a Cell of Origin for Prostate Cancer. Stem Cells Translational Medicine. 6(3). 748–760. 18 indexed citations

Chen, Xin, Bigang Liu, Qiuhui Li, et al.. (2013). Dissociated Primary Human Prostate Cancer Cells Coinjected with the Immortalized Hs5 Bone Marrow Stromal Cells Generate Undifferentiated Tumors in NOD/SCID-γ Mice. PLoS ONE. 8(2). e56903–e56903. 10 indexed citations

Qin, Jichao, Xin Liu, Brian Laffin, et al.. (2012). The PSA−/lo Prostate Cancer Cell Population Harbors Self-Renewing Long-Term Tumor-Propagating Cells that Resist Castration. Cell stem cell. 10(5). 556–569. 243 indexed citations

Liu, Can, Kevin Kelnar, Bigang Liu, et al.. (2011). The microRNA miR-34a inhibits prostate cancer stem cells and metastasis by directly repressing CD44. Nature Medicine. 17(2). 211–215. 1136 indexed citations breakdown →

10.

Yan, Hong, Xin Chen, Qiuping Zhang, et al.. (2011). Drug-Tolerant Cancer Cells Show Reduced Tumor-Initiating Capacity: Depletion of CD44+ Cells and Evidence for Epigenetic Mechanisms. PLoS ONE. 6(9). e24397–e24397. 42 indexed citations

11.

Jeter, Collene, Bigang Liu, Xin Liu, et al.. (2011). NANOG promotes cancer stem cell characteristics and prostate cancer resistance to androgen deprivation. Oncogene. 30(36). 3833–3845. 302 indexed citations

12.

Li, Hangwen, Ming Jiang, Sofia Honorio, et al.. (2009). Methodologies in Assaying Prostate Cancer Stem Cells. Methods in molecular biology. 568. 85–138. 30 indexed citations

13.

Jeter, Collene, Mark Badeaux, Grace Choy, et al.. (2009). Functional Evidence that the Self-Renewal Gene NANOG Regulates Human Tumor Development. Stem Cells. 27(5). 993–1005. 280 indexed citations

14.

Li, Hangwen, Xin Chen, Tammy Calhoun‐Davis, Kent Claypool, & Dean G. Tang. (2008). PC3 Human Prostate Carcinoma Cell Holoclones Contain Self-renewing Tumor-Initiating Cells. Cancer Research. 68(6). 1820–1825. 181 indexed citations

15.

Bhatia, Bobby, Ming Jiang, Mahipal Suraneni, et al.. (2008). Critical and Distinct Roles of p16 and Telomerase in Regulating the Proliferative Life Span of Normal Human Prostate Epithelial Progenitor Cells. Journal of Biological Chemistry. 283(41). 27957–27972. 31 indexed citations

16.

Patrawala, Lubna, Tammy Calhoun‐Davis, Robin Schneider‐Broussard, & Dean G. Tang. (2007). Hierarchical Organization of Prostate Cancer Cells in Xenograft Tumors: The CD44+α2β1+ Cell Population Is Enriched in Tumor-Initiating Cells. Cancer Research. 67(14). 6796–6805. 279 indexed citations

17.

Bhatia, Bobby, Asha S. Multani, Lubna Patrawala, et al.. (2007). Evidence that senescent human prostate epithelial cells enhance tumorigenicity: Cell fusion as a potential mechanism and inhibition by p16INK4a and hTERT. International Journal of Cancer. 122(7). 1483–1495. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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