Greg H. Enders

3.0k total citations · 1 hit paper
31 papers, 2.4k citations indexed

About

Greg H. Enders is a scholar working on Oncology, Molecular Biology and Pathology and Forensic Medicine. According to data from OpenAlex, Greg H. Enders has authored 31 papers receiving a total of 2.4k indexed citations (citations by other indexed papers that have themselves been cited), including 21 papers in Oncology, 18 papers in Molecular Biology and 8 papers in Pathology and Forensic Medicine. Recurrent topics in Greg H. Enders's work include Cancer-related Molecular Pathways (16 papers), Epigenetics and DNA Methylation (9 papers) and Genetic factors in colorectal cancer (8 papers). Greg H. Enders is often cited by papers focused on Cancer-related Molecular Pathways (16 papers), Epigenetics and DNA Methylation (9 papers) and Genetic factors in colorectal cancer (8 papers). Greg H. Enders collaborates with scholars based in United States, United Kingdom and Spain. Greg H. Enders's co-authors include James Koh, Brian David Dynlacht, Ed Harlow, Emma E. Furth, Charlotte Y. Dai, Jayashree Mitra, Joshua T. Mendell, Danielle Murphy, Michael Dews and Duonan Yu and has published in prestigious journals such as Nature, Journal of Biological Chemistry and Nature Genetics.

In The Last Decade

Greg H. Enders

30 papers receiving 2.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Augmentation of tumor angiogenesis by a Myc-activated microRNA cluster

2006 851 citations Michael Dews, Duonan Yu et al. Nature Genetics profile →

Peers

Greg H. Enders

MB

ONCOL

CR

CB

PHYSI

Pedro A. Pérez–Mancera United Kingdom

Rónán C. O’Hagan United States

Ann M. Killary United States

Aleksandra Franovic United States

Nikolaos G. Kastrinakis Greece

M. Phillip DeYoung United States

Mario Mikula Austria

Lucia Riccardi United States

Charles Catzavelos Canada

Qiongqing Wang United States

Pedro A. Pérez–Mancera United Kingdom

Greg H. Enders

2.1k ×1.1

MB

1.0k ×1.1

ONCOL

935 ×1.0

CR

295 ×1.0

CB

446 ×1.9

PHYSI

Citations per year, relative to Greg H. Enders Greg H. Enders (= 1×) peers Pedro A. Pérez–Mancera

Countries citing papers authored by Greg H. Enders

Since Specialization

Citations

This map shows the geographic impact of Greg H. Enders's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Greg H. Enders with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Greg H. Enders more than expected).

Fields of papers citing papers by Greg H. Enders

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Greg H. Enders. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Greg H. Enders. The network helps show where Greg H. Enders may publish in the future.

Co-authorship network of co-authors of Greg H. Enders

This figure shows the co-authorship network connecting the top 25 collaborators of Greg H. Enders. A scholar is included among the top collaborators of Greg H. Enders based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Greg H. Enders. Greg H. Enders is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

1.

Goyal, Hemant, Rupak Desai, Mark M. Aloysius, et al.. (2019). Young-onset colorectal cancer: hospitalization trends and gender disparities in the United States 2010–2014. International Journal of Colorectal Disease. 34(9). 1611–1615. 5 indexed citations

2.

Arora, Sanjeevani, Hong Yan, Il‐Taeg Cho, et al.. (2015). Genetic Variants That Predispose to DNA Double-Strand Breaks in Lymphocytes From a Subset of Patients With Familial Colorectal Carcinomas. Gastroenterology. 149(7). 1872–1883.e9. 30 indexed citations

3.

Holzen, Urs von & Greg H. Enders. (2012). A surprise cell of origin for Barrett’s esophagus. Cancer Biology & Therapy. 13(8). 588–591. 8 indexed citations

4.

Kennedy, Alyssa L., Jennifer P. Morton, David M. Nelson, et al.. (2011). Activation of the PIK3CA/AKT Pathway Suppresses Senescence Induced by an Activated RAS Oncogene to Promote Tumorigenesis. Molecular Cell. 42(1). 36–49. 160 indexed citations

5.

Kennedy, Alyssa L., Tony McBryan, Greg H. Enders, et al.. (2010). Senescent mouse cells fail to overtly regulate the HIRA histone chaperone and do not form robust Senescence Associated Heterochromatin Foci. Cell Division. 5(1). 16–16. 53 indexed citations

6.

Holzen, Urs von, Joel E. Richter, Gary W. Falk, et al.. (2010). Evidence for DNA Damage Checkpoint Activation in Barrett Esophagus. Translational Oncology. 3(1). 33–42. 6 indexed citations

7.

Enders, Greg H., et al.. (2009). Chemoprevention of Mouse Intestinal Tumorigenesis by the Cyclin-Dependent Kinase Inhibitor SNS-032. Cancer Prevention Research. 2(9). 800–806. 17 indexed citations

8.

Payá, Artemio, Cristina Alenda, Lucía Pérez–Carbonell, et al.. (2009). Utility of p16 Immunohistochemistry for the Identification of Lynch Syndrome. Clinical Cancer Research. 15(9). 3156–3162. 15 indexed citations

9.

Enders, Greg H.. (2008). Expanded Roles for Chk1 in Genome Maintenance. Journal of Biological Chemistry. 283(26). 17749–17752. 49 indexed citations

10.

Adams, Peter D. & Greg H. Enders. (2008). Wnt signaling and senescence: A tug of war in early neoplasia?. Cancer Biology & Therapy. 7(11). 1706–1711. 19 indexed citations

11.

Dews, Michael, Duonan Yu, Danielle Murphy, et al.. (2006). Augmentation of tumor angiogenesis by a Myc-activated microRNA cluster. Nature Genetics. 38(9). 1060–1065. 851 indexed citations breakdown →

12.

Enders, Greg H. & Shannon L. Maude. (2006). Traffic safety for the cell: Influence of cyclin-dependent kinase activity on genomic stability. Gene. 371(1). 1–6. 14 indexed citations

13.

Furth, Emma E., Karen S. Gustafson, Charlotte Y. Dai, et al.. (2006). Induction of the Tumor-Suppressor p16INK4a within Regenerative Epithelial Crypts in Ulcerative Colitis. Neoplasia. 8(6). 429–436. 18 indexed citations

14.

Sharpless, Norman E., et al.. (2005). p16Ink4a inhibits histologic progression and angiogenic signaling in min colon tumors. Cancer Biology & Therapy. 4(12). 1389–1394. 15 indexed citations

15.

Mitra, Jayashree & Greg H. Enders. (2004). Cyclin A/Cdk2 complexes regulate activation of Cdk1 and Cdc25 phosphatases in human cells. Oncogene. 23(19). 3361–3367. 70 indexed citations

16.

Dai, Charlotte Y., Ronald A. DePinho, Michael S. Gee, et al.. (2003). Inhibition of colon tumor progression and angiogenesis by the Ink4a/Arf locus.. PubMed. 63(4). 742–6. 34 indexed citations

17.

Wexler, Roseanne S., et al.. (2001). Regulation of gene expression and cell growth in vivo by tetracycline using the hollow fiber assay.. PubMed. 21(2A). 869–72. 8 indexed citations

18.

Dai, Charlotte Y. & Greg H. Enders. (2000). p16INK4a can initiate an autonomous senescence program. Oncogene. 19(13). 1613–1622. 117 indexed citations

19.

Dai, Charlotte Y., Emma E. Furth, Rosemarie Mick, et al.. (2000). p16INK4a expression begins early in human colon neoplasia and correlates inversely with markers of cell proliferation. Gastroenterology. 119(4). 929–942. 74 indexed citations

20.

Koh, James, Greg H. Enders, Brian David Dynlacht, & Ed Harlow. (1995). Tumour-derived p16 alleles encoding proteins defective in cell-cycle inhibition. Nature. 375(6531). 506–510. 444 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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