Robin Schneider‐Broussard

3.2k total citations · 2 hit papers
18 papers, 2.6k citations indexed

About

Robin Schneider‐Broussard is a scholar working on Oncology, Molecular Biology and Genetics. According to data from OpenAlex, Robin Schneider‐Broussard has authored 18 papers receiving a total of 2.6k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Oncology, 9 papers in Molecular Biology and 7 papers in Genetics. Recurrent topics in Robin Schneider‐Broussard's work include Cancer Cells and Metastasis (5 papers), Cancer-related Molecular Pathways (5 papers) and RNA Interference and Gene Delivery (3 papers). Robin Schneider‐Broussard is often cited by papers focused on Cancer Cells and Metastasis (5 papers), Cancer-related Molecular Pathways (5 papers) and RNA Interference and Gene Delivery (3 papers). Robin Schneider‐Broussard collaborates with scholars based in United States. Robin Schneider‐Broussard's co-authors include Dean G. Tang, Lubna Patrawala, Kent Claypool, Jianjun Zhou, Bobby Bhatia, D. Gale Johnson, Shaohua Tang, Tammy Calhoun‐Davis, Dhyan Chandra and Jiancheng Zhou and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Molecular and Cellular Biology.

In The Last Decade

Robin Schneider‐Broussard

18 papers receiving 2.6k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Highly purified CD44+ prostate cancer cells from xenograft human tumors are enriched in tumorigenic and metastatic progenitor cells

2006 770 citations Lubna Patrawala, Robin Schneider‐Broussard et al. Oncogene profile →
Side Population Is Enriched in Tumorigenic, Stem-Like Cancer Cells, whereas ABCG2+ and ABCG2− Cancer Cells Are Similarly Tumorigenic

2005 737 citations Lubna Patrawala, Robin Schneider‐Broussard et al. Cancer Research profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Robin Schneider‐Broussard United States	15	1.8k	1.5k	657	522	249	18	2.6k
Frank Bartel Germany	31	1.9k 1.0×	2.5k 1.6×	841 1.3×	424 0.8×	234 0.9×	65	3.6k
René Villadsen Denmark	23	1.6k 0.9×	1.3k 0.9×	605 0.9×	271 0.5×	347 1.4×	40	2.5k
Sharmila A. Bapat India	20	1.5k 0.8×	1.6k 1.1×	741 1.1×	169 0.3×	232 0.9×	53	2.5k
Purificacı́on Muñoz Spain	29	1.1k 0.6×	2.5k 1.7×	755 1.1×	195 0.4×	189 0.8×	35	3.8k
Matthew V. Lorenzi United States	28	920 0.5×	1.5k 1.0×	367 0.6×	586 1.1×	200 0.8×	64	2.6k
Yoko Katsuno Japan	17	1.1k 0.6×	1.6k 1.0×	489 0.7×	239 0.5×	162 0.7×	22	2.3k
Peter Eirew Canada	17	1.7k 0.9×	1.5k 1.0×	681 1.0×	265 0.5×	151 0.6×	26	2.6k
Pedro A. Pérez–Mancera United Kingdom	24	1.0k 0.6×	2.1k 1.4×	935 1.4×	225 0.4×	295 1.2×	38	3.1k
Kou-Juey Wu Taiwan	22	1.1k 0.6×	2.2k 1.4×	1.2k 1.8×	389 0.7×	212 0.9×	25	3.0k
Paul Meltzer United States	7	768 0.4×	1.8k 1.2×	824 1.3×	235 0.5×	190 0.8×	10	2.4k

Countries citing papers authored by Robin Schneider‐Broussard

Since Specialization

Citations

This map shows the geographic impact of Robin Schneider‐Broussard's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Robin Schneider‐Broussard with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Robin Schneider‐Broussard more than expected).

Fields of papers citing papers by Robin Schneider‐Broussard

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Robin Schneider‐Broussard. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Robin Schneider‐Broussard. The network helps show where Robin Schneider‐Broussard may publish in the future.

Co-authorship network of co-authors of Robin Schneider‐Broussard

This figure shows the co-authorship network connecting the top 25 collaborators of Robin Schneider‐Broussard. A scholar is included among the top collaborators of Robin Schneider‐Broussard based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Robin Schneider‐Broussard. Robin Schneider‐Broussard is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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18 of 18 papers shown

Smith, Karen, et al.. (2019). Development of a biosafety level-2 facility for irradiation of biological cells using MeV ions. Nuclear Instruments and Methods in Physics Research Section B Beam Interactions with Materials and Atoms. 447. 50–54. 1 indexed citations

Suraneni, Mahipal, Robin Schneider‐Broussard, John R. Moore, et al.. (2010). Transgenic expression of 15-lipoxygenase 2 (15-LOX2) in mouse prostate leads to hyperplasia and cell senescence. Oncogene. 29(30). 4261–4275. 37 indexed citations

Chen, Xin, Robin Schneider‐Broussard, Mark J. McArthur, et al.. (2009). Abnormal differentiation, hyperplasia and embryonic/perinatal lethality in BK5-T/t transgenic mice. Differentiation. 77(3). 324–334. 4 indexed citations

Bhatia, Bobby, Ming Jiang, Mahipal Suraneni, et al.. (2008). Critical and Distinct Roles of p16 and Telomerase in Regulating the Proliferative Life Span of Normal Human Prostate Epithelial Progenitor Cells. Journal of Biological Chemistry. 283(41). 27957–27972. 31 indexed citations

Patrawala, Lubna, Tammy Calhoun‐Davis, Robin Schneider‐Broussard, & Dean G. Tang. (2007). Hierarchical Organization of Prostate Cancer Cells in Xenograft Tumors: The CD44+α2β1+ Cell Population Is Enriched in Tumor-Initiating Cells. Cancer Research. 67(14). 6796–6805. 279 indexed citations

Bhatia, Bobby, Asha S. Multani, Lubna Patrawala, et al.. (2007). Evidence that senescent human prostate epithelial cells enhance tumorigenicity: Cell fusion as a potential mechanism and inhibition by p16INK4a and hTERT. International Journal of Cancer. 122(7). 1483–1495. 38 indexed citations

Tang, Dean G., Lubna Patrawala, Bobby Bhatia, et al.. (2006). Prostate cancer stem/progenitor cells: Identification, characterization, and implications. Molecular Carcinogenesis. 46(1). 1–14. 178 indexed citations

Patrawala, Lubna, Robin Schneider‐Broussard, Heng Li, et al.. (2006). Highly purified CD44+ prostate cancer cells from xenograft human tumors are enriched in tumorigenic and metastatic progenitor cells. Oncogene. 25(12). 1696–1708. 770 indexed citations breakdown →

Tang, Dean G., Bobby Bhatia, Shaohua Tang, & Robin Schneider‐Broussard. (2006). 15-Lipoxygenase 2 (15-LOX2) is a functional tumor suppressor that regulates human prostate epithelial cell differentiation, senescence, and growth (size). Prostaglandins & Other Lipid Mediators. 82(1-4). 135–146. 47 indexed citations

10.

Patrawala, Lubna, et al.. (2005). Side Population Is Enriched in Tumorigenic, Stem-Like Cancer Cells, whereas ABCG2+ and ABCG2− Cancer Cells Are Similarly Tumorigenic. Cancer Research. 65(14). 6207–6219. 737 indexed citations breakdown →

11.

Rounbehler, Robert J., Robin Schneider‐Broussard, Claudio J. Conti, & D. Gale Johnson. (2001). Myc lacks E2F1's ability to suppress skin carcinogenesis. Oncogene. 20(38). 5341–5349. 35 indexed citations

12.

Wang, Aijin, Robin Schneider‐Broussard, Addanki P. Kumar, Michael C. MacLeod, & D. Gale Johnson. (2000). Regulation of BRCA1 Expression by the Rb-E2F Pathway. Journal of Biological Chemistry. 275(6). 4532–4536. 101 indexed citations

13.

Pierce, Angela, Robin Schneider‐Broussard, Irma Gimenez‐Conti, et al.. (1999). E2F1 Has Both Oncogenic and Tumor-Suppressive Properties in a Transgenic Model. Molecular and Cellular Biology. 19(9). 6408–6414. 137 indexed citations

14.

Pierce, Angela, Irma Gimenez‐Conti, Robin Schneider‐Broussard, et al.. (1998). Increased E2F1 activity induces skin tumors in mice heterozygous and nullizygous for p53. Proceedings of the National Academy of Sciences. 95(15). 8858–8863. 135 indexed citations

15.

Pierce, Angela, et al.. (1998). Differential activities of E2F family members: Unique functions in regulating transcription. Molecular Carcinogenesis. 22(3). 190–198. 30 indexed citations

16.

Schneider‐Broussard, Robin, Darryl L. Felder, Caryl A. Chlan, & Joseph E. Neigel. (1998). TESTS OF PHYLOGEOGRAPHIC MODELS WITH NUCLEAR AND MITOCHONDRIAL DNA SEQUENCE VARIATION IN THE STONE CRABS, MENIPPE ADINA AND MENIPPE MERCENARIA. Evolution. 52(6). 1671–1678. 30 indexed citations

17.

Schneider‐Broussard, Robin, Darryl L. Felder, Caryl A. Chlan, & Joseph E. Neigel. (1998). Tests of Phylogeographic Models with Nuclear and Mitochondrial DNA Sequence Variation in the Stone Crabs, Menippe adina and Menippe mercenaria. Evolution. 52(6). 1671–1671. 9 indexed citations

18.

Schneider‐Broussard, Robin & Joseph E. Neigel. (1997). A large-subunit mitochondrial ribosomal DNA sequence translocated to the nuclear genome of two stone crabs (Menippe). Molecular Biology and Evolution. 14(2). 156–165. 27 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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