Takeru Hayashi

866 total citations
14 papers, 641 citations indexed

About

Takeru Hayashi is a scholar working on Molecular Biology, Surgery and Immunology. According to data from OpenAlex, Takeru Hayashi has authored 14 papers receiving a total of 641 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 8 papers in Surgery and 8 papers in Immunology. Recurrent topics in Takeru Hayashi's work include Helicobacter pylori-related gastroenterology studies (8 papers), Galectins and Cancer Biology (7 papers) and Protein Tyrosine Phosphatases (5 papers). Takeru Hayashi is often cited by papers focused on Helicobacter pylori-related gastroenterology studies (8 papers), Galectins and Cancer Biology (7 papers) and Protein Tyrosine Phosphatases (5 papers). Takeru Hayashi collaborates with scholars based in Japan and United States. Takeru Hayashi's co-authors include Masanori Hatakeyama, Hideaki Higashi, Naoko Murata‐Kamiya, Toshiya Senda, Lisa Nagase, Kenji Kikuchi, Miki Senda, Atsushi Takahashi, Masanori Taira and Ippei Kikuchi and has published in prestigious journals such as Journal of Biological Chemistry, Scientific Reports and Biochemical and Biophysical Research Communications.

In The Last Decade

Takeru Hayashi

14 papers receiving 635 citations

Peers

Takeru Hayashi

SURGE

IMMUN

ONCOL

Yumiko Fujii Japan

Garrett Z. Ng Australia

Judith Lind Austria

Malvika Pompaiah Germany

Wende Tong China

Isabella M. Toller Switzerland

Aki Imai‐Matsushima Germany

Virginie Debailleul France

Monika Semmrich France

Séverine Nollet France

Yumiko Fujii Japan

Takeru Hayashi

501 ×1.2

SURGE

402 ×1.2

IMMUN

242 ×1.0

78 ×1.0

ONCOL

95 ×1.3

Citations per year, relative to Takeru Hayashi Takeru Hayashi (= 1×) peers Yumiko Fujii

Countries citing papers authored by Takeru Hayashi

Since Specialization

Citations

This map shows the geographic impact of Takeru Hayashi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Takeru Hayashi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Takeru Hayashi more than expected).

Fields of papers citing papers by Takeru Hayashi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Takeru Hayashi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Takeru Hayashi. The network helps show where Takeru Hayashi may publish in the future.

Co-authorship network of co-authors of Takeru Hayashi

This figure shows the co-authorship network connecting the top 25 collaborators of Takeru Hayashi. A scholar is included among the top collaborators of Takeru Hayashi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Takeru Hayashi. Takeru Hayashi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Hayashi, Takeru, et al.. (2022). Kinase Activity of PAR1b, Which Mediates Nuclear Translocation of the BRCA1 Tumor Suppressor, Is Potentiated by Nucleic Acid-Mediated PAR1b Multimerization. International Journal of Molecular Sciences. 23(12). 6634–6634. 1 indexed citations

Wu, Xiaojing, et al.. (2020). Alternative splicing reverses the cell-intrinsic and cell-extrinsic pro-oncogenic potentials of YAP1. Journal of Biological Chemistry. 295(41). 13965–13980. 6 indexed citations

Yahara, Koji, Takeru Hayashi, Takeshi Azuma, et al.. (2018). Evaluating the origin and virulence of a Helicobacter pylori cagA-positive strain isolated from a non-human primate. Scientific Reports. 8(1). 15981–15981. 14 indexed citations

Hayashi, Takeru, Miki Senda, Nobuhiro Suzuki, et al.. (2017). Differential Mechanisms for SHP2 Binding and Activation Are Exploited by Geographically Distinct Helicobacter pylori CagA Oncoproteins. Cell Reports. 20(12). 2876–2890. 68 indexed citations

Hayashi, Takeru, et al.. (2016). Impact of structural polymorphism for the Helicobacter pylori CagA oncoprotein on binding to polarity-regulating kinase PAR1b. Scientific Reports. 6(1). 30031–30031. 22 indexed citations

Murata‐Kamiya, Naoko, Takeru Hayashi, Lisa Nagase, et al.. (2016). Host SHP1 phosphatase antagonizes Helicobacter pylori CagA and can be downregulated by Epstein–Barr virus. Nature Microbiology. 1(4). 16026–16026. 76 indexed citations

Senda, Miki, Takeru Hayashi, Masanori Hatakeyama, et al.. (2016). Use of Multiple Cryoprotectants to Improve Diffraction Quality from Protein Crystals. Crystal Growth & Design. 16(3). 1565–1571. 6 indexed citations

Kameoka, Shoichiro, Takeshi Kameyama, Seiichi Sato, et al.. (2016). Helicobacter pylori induces IL-1β protein through the inflammasome activation in differentiated macrophagic cells . Biomedical Research. 37(1). 21–27. 29 indexed citations

Nagase, Lisa, Takeru Hayashi, Toshiya Senda, & Masanori Hatakeyama. (2015). Dramatic increase in SHP2 binding activity of Helicobacter pylori Western CagA by EPIYA-C duplication: its implications in gastric carcinogenesis. Scientific Reports. 5(1). 15749–15749. 37 indexed citations

10.

Takahashi, Atsushi, et al.. (2015). Determination of the catalytic activity of LEOPARD syndrome-associated SHP2 mutants toward parafibromin, a bona fide SHP2 substrate involved in Wnt signaling. Biochemical and Biophysical Research Communications. 469(4). 1133–1139. 17 indexed citations

11.

Tsutsumi, Ryouhei, Atsushi Takahashi, Yumiko Fujii, et al.. (2013). YAP and TAZ, Hippo Signaling Targets, Act as a Rheostat for Nuclear SHP2 Function. Developmental Cell. 26(6). 658–665. 79 indexed citations

12.

Hayashi, Takeru, et al.. (2012). Bacterial EPIYA effectors - Where do they come from? What are they? Where are they going?. Cellular Microbiology. 15(3). 377–385. 43 indexed citations

13.

Hayashi, Takeru, Miki Senda, Hideaki Higashi, et al.. (2012). Tertiary Structure-Function Analysis Reveals the Pathogenic Signaling Potentiation Mechanism of Helicobacter pylori Oncogenic Effector CagA. Cell Host & Microbe. 12(1). 20–33. 118 indexed citations

14.

Murata‐Kamiya, Naoko, Kenji Kikuchi, Takeru Hayashi, Hideaki Higashi, & Masanori Hatakeyama. (2010). Helicobacter pylori Exploits Host Membrane Phosphatidylserine for Delivery, Localization, and Pathophysiological Action of the CagA Oncoprotein. Cell Host & Microbe. 7(5). 399–411. 125 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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