Suzanne E. Hickman is a scholar working on Neurology, Physiology and Immunology.
According to data from OpenAlex, Suzanne E. Hickman has authored 27 papers receiving a total of 6.6k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Neurology, 13 papers in Physiology and 11 papers in Immunology. Recurrent topics in Suzanne E. Hickman's work include Neuroinflammation and Neurodegeneration Mechanisms (18 papers), Alzheimer's disease research and treatments (13 papers) and Immune cells in cancer (7 papers). Suzanne E. Hickman is often cited by papers focused on Neuroinflammation and Neurodegeneration Mechanisms (18 papers), Alzheimer's disease research and treatments (13 papers) and Immune cells in cancer (7 papers). Suzanne E. Hickman collaborates with scholars based in United States, Netherlands and China. Suzanne E. Hickman's co-authors include Joseph El Khoury, Elizabeth Allison, Terry K. Means, Liza Morsett, Saef Izzy, Pritha Sen, Mark L. Borowsky, Li-chong Wang, Toshiro K. Ohsumi and Andrew D. Luster and has published in prestigious journals such as Nature, Nature Medicine and Nature Communications.
In The Last Decade
Suzanne E. Hickman
27 papers
receiving
6.5k citations
Hit Papers
What are hit papers?
Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies
if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the
same subfield and year (this is the minimum needed to enter the top 1%, not the average
within it), or reaches the top citation threshold in at least one of its specific research
topics.
Microglia in neurodegeneration
20181.2k citationsSuzanne E. Hickman, Saef Izzy et al.Nature Neuroscienceprofile →
The microglial sensome revealed by direct RNA sequencing
20131.2k citationsSuzanne E. Hickman, Toshiro K. Ohsumi et al.Nature Neuroscienceprofile →
Microglial Dysfunction and Defective β-Amyloid Clearance Pathways in Aging Alzheimer's Disease Mice
20081.1k citationsSuzanne E. Hickman, Elizabeth Allison et al.Journal of Neuroscienceprofile →
Ccr2 deficiency impairs microglial accumulation and accelerates progression of Alzheimer-like disease
2007740 citationsJoseph El Khoury, Suzanne E. Hickman et al.Nature Medicineprofile →
Scavenger receptor-mediated adhesion of microglia to β-amyloid fibrils
1996636 citationsJoseph El Khoury, Suzanne E. Hickman et al.Natureprofile →
Peers — A (Enhanced Table)
Peers by citation overlap · career bar shows stage (early→late)
cites ·
hero ref
Countries citing papers authored by Suzanne E. Hickman
Since
Specialization
Citations
This map shows the geographic impact of Suzanne E. Hickman's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Suzanne E. Hickman with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Suzanne E. Hickman more than expected).
Fields of papers citing papers by Suzanne E. Hickman
This network shows the impact of papers produced by Suzanne E. Hickman. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Suzanne E. Hickman. The network helps show where Suzanne E. Hickman may publish in the future.
Co-authorship network of co-authors of Suzanne E. Hickman
This figure shows the co-authorship network connecting the top 25 collaborators of Suzanne E. Hickman.
A scholar is included among the top collaborators of Suzanne E. Hickman based on the total number of
citations received by their joint publications. Widths of edges
represent the number of papers authors have co-authored together.
Node borders
signify the number of papers an author published with Suzanne E. Hickman. Suzanne E. Hickman is excluded from
the visualization to improve readability, since they are connected to all nodes in the network.
Hickman, Suzanne E. & Joseph El Khoury. (2019). Analysis of the Microglial Sensome. Methods in molecular biology. 2034. 305–323.26 indexed citations
10.
Hickman, Suzanne E., Saef Izzy, Pritha Sen, Liza Morsett, & Joseph El Khoury. (2018). Microglia in neurodegeneration. Nature Neuroscience. 21(10). 1359–1369.1195 indexed citations breakdown →
Hickman, Suzanne E., Toshiro K. Ohsumi, Mark L. Borowsky, et al.. (2013). The microglial sensome revealed by direct RNA sequencing. Nature Neuroscience. 16(12). 1896–1905.1151 indexed citations breakdown →
16.
Tampakakis, Emmanouil, Lindsay Puckett, Melissa Tai, et al.. (2009). Evolutionarily Conserved Recognition and Innate Immunity to Fungal Pathogens by the Scavenger Receptors SCARF1 and CD36. Digital Access to Scholarship at Harvard (DASH) (Harvard University).195 indexed citations
Hickman, Suzanne E., Elizabeth Allison, & Joseph El Khoury. (2008). Microglial Dysfunction and Defective β-Amyloid Clearance Pathways in Aging Alzheimer's Disease Mice. Journal of Neuroscience. 28(33). 8354–8360.1067 indexed citations breakdown →
19.
Khoury, Joseph El, Suzanne E. Hickman, Terry K. Means, et al.. (2007). Ccr2 deficiency impairs microglial accumulation and accelerates progression of Alzheimer-like disease. Nature Medicine. 13(4). 432–438.740 indexed citations breakdown →
20.
Khoury, Joseph El, Suzanne E. Hickman, Christian A. Thomas, et al.. (1996). Scavenger receptor-mediated adhesion of microglia to β-amyloid fibrils. Nature. 382(6593). 716–719.636 indexed citations breakdown →
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive
bibliographic database. While OpenAlex provides broad and valuable coverage of the global
research landscape, it—like all bibliographic datasets—has inherent limitations. These include
incomplete records, variations in author disambiguation, differences in journal indexing, and
delays in data updates. As a result, some metrics and network relationships displayed in
Rankless may not fully capture the entirety of a scholar's output or impact.