Sunita Gulati
- Microbiology top 0.1%
- Immunology top 2%
- Epidemiology top 5%
- Molecular Biology
- Infectious Diseases top 5%
- Co-authors
- Peter A. RiceSanjay RamDaniel P. McQuillenMichael K. PangburnChristopher A. ElkinsLisa A. LewisJutamas NgampasutadolAjay Sharma
- Topics
- Bacterial Infections and Vaccines (58 papers)Reproductive tract infections research (40 papers)Complement system in diseases (22 papers)
- Cited by
- MicrobiologyImmunologyEndocrinology
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryJournal of Clinical Investigation
- Partner nations
- United StatesUnited KingdomCanada
In The Last Decade
Sunita Gulati
69 papers receiving 3.0k citations
Peers
Comparison fields: 5 of 107
- Microbiology 1.8k
- Immunology 1.0k
- Epidemiology 859
- Molecular Biology 510
- Infectious Diseases 421
Countries citing papers authored by Sunita Gulati
This map shows the geographic impact of Sunita Gulati's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sunita Gulati with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sunita Gulati more than expected).
Fields of papers citing papers by Sunita Gulati
This network shows the impact of papers produced by Sunita Gulati. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sunita Gulati. The network helps show where Sunita Gulati may publish in the future.
Co-authorship network of co-authors of Sunita Gulati
This figure shows the co-authorship network connecting the top 25 collaborators of Sunita Gulati. A scholar is included among the top collaborators of Sunita Gulati based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sunita Gulati. Sunita Gulati is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 0 | |
| 3 | 0 | |
| 4 | 4 | |
| 5 | 12 | |
| 6 | 62 | |
| 7 | 12 | |
| 8 | 33 | |
| 9 | 29 | |
| 10 | 30 | |
| 11 | 22 | |
| 12 | 64 | |
| 13 | 17 | |
| 14 | 75 | |
| 15 | 81 | |
| 16 | 41 | |
| 17 | 40 | |
| 18 | 39 | |
| 19 | 57 | |
| 20 | 66 |
About Sunita Gulati
Sunita Gulati is a scholar working on Microbiology, Immunology and Hematology, having authored 72 papers that have together received 3.0k indexed citations. Recurring topics across this work include Bacterial Infections and Vaccines (58 papers), Reproductive tract infections research (40 papers) and Complement system in diseases (22 papers). The work is most often cited by research in Microbiology (1.8k citations), Immunology (1.0k citations) and Endocrinology (164 citations). Sunita Gulati has collaborated with scholars based in United States, United Kingdom and Canada. Frequent co-authors include Peter A. Rice, Sanjay Ram, Daniel P. McQuillen, Michael K. Pangburn, Christopher A. Elkins, Lisa A. Lewis, Jutamas Ngampasutadol, Ajay Sharma, Scott D. Simpson and Bo Zheng. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.