Sumia A. Bageghni
Impact in
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- Cardiac Fibrosis and Remodeling
- Cardiovascular Function and Risk Factors
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- Cancer-related molecular mechanisms research
Papers in
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- Signaling Pathways in Disease 2
- Pluripotent Stem Cells Research 1
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- Cardiac Fibrosis and Remodeling 3
- Co-authors
- Karen E. Porter (5 shared papers)Neil A. Turner (5 shared papers)Justin Ainscough (4 shared papers)Mark J. Drinkhill (4 shared papers)Christopher P. Denton (2 shared papers)Karen E. Hemmings (3 shared papers)David O’Regan (1 shared paper)Kirsten Riches‐Suman (1 shared paper)
- Journals
- Cardiovascular Pathology (1 paper)Matrix Biology (1 paper)The FASEB Journal (1 paper)Genes & Development (1 paper)Journal of Endocrinology (1 paper)
- Partner nations
- United KingdomPanamaSaudi Arabia
In The Last Decade
Sumia A. Bageghni
7 papers receiving 292 citations
Peers
Comparison fields: 5 of 52
- Cardiology and Cardiovascular Medicine 118
- Cancer Research 45
- Molecular Biology 167
- Immunology 42
- Genetics 45
Countries citing papers authored by Sumia A. Bageghni
This map shows the geographic impact of Sumia A. Bageghni's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sumia A. Bageghni with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sumia A. Bageghni more than expected).
Fields of papers citing papers by Sumia A. Bageghni
This network shows the impact of papers produced by Sumia A. Bageghni. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sumia A. Bageghni. The network helps show where Sumia A. Bageghni may publish in the future.
Co-authors
The 25 scholars most cited alongside Sumia A. Bageghni, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2017 | 92 | |
| 2 | 2019 | 60 | |
| 3 | 2018 | 59 | |
| 4 | 2014 | 45 | |
| 5 | 2017 | 30 | |
| 6 | 2024 | 5 | |
| 7 | 2016 | 4 |
About Sumia A. Bageghni
Sumia A. Bageghni is a scholar working on Molecular Biology, Cardiology and Cardiovascular Medicine, Genetics, Surgery and Pharmacology, having authored 7 papers that have together received 295 indexed citations. Recurring topics across this work include Cardiac Fibrosis and Remodeling (3 papers), Genetics and Neurodevelopmental Disorders (2 papers), Signaling Pathways in Disease (2 papers), Genetic and Clinical Aspects of Sex Determination and Chromosomal Abnormalities (1 paper), Apelin-related biomedical research (1 paper), Blood properties and coagulation (1 paper), Pluripotent Stem Cells Research (1 paper) and Endoplasmic Reticulum Stress and Disease (1 paper). The work is most often cited by research in Cardiology and Cardiovascular Medicine (118 citations), Cancer Research (45 citations), Molecular Biology (167 citations), Immunology (42 citations) and Genetics (45 citations). Sumia A. Bageghni has collaborated with scholars based in United Kingdom, Panama and Saudi Arabia. Frequent co-authors include Karen E. Porter, Neil A. Turner, Justin Ainscough, Mark J. Drinkhill, Christopher P. Denton, Karen E. Hemmings, David O’Regan, Kirsten Riches‐Suman, W Mansfield and Dawn Coverley. Their work appears in journals such as Cardiovascular Pathology, Matrix Biology, The FASEB Journal, Genes & Development and Journal of Endocrinology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.