Stuart W. Hicks
- Endocrinology top 5%
- Cell Biology top 5%
- Endoplasmic Reticulum Stress and Disease 6
- Cellular transport and secretion 6
- Physiology top 5%
- Calcium signaling and nucleotide metabolism 3
- Virology top 10%
- HIV Research and Treatment 2
- Immunology top 10%
- Immune Cell Function and Interaction 5
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- Monoclonal and Polyclonal Antibodies Research 8
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- CAR-T cell therapy research 7
- HER2/EGFR in Cancer Research 6
- Co-authors
- Carolyn E. MachamerJorge E. GalánCindy M. QuezadaC. Erec StebbinsGuillaume CharronFederica PericleGiuseppe SconocchiaDavid M. Segal
- Cited by
- EndocrinologyCell BiologyPhysiology
- Partner nations
- United StatesSwitzerlandUkraine
In The Last Decade
Stuart W. Hicks
26 papers receiving 1.1k citations
Peers
Comparison fields: 5 of 83
- Endocrinology 119
- Cell Biology 305
- Physiology 58
- Virology 60
- Immunology 247
Countries citing papers authored by Stuart W. Hicks
This map shows the geographic impact of Stuart W. Hicks's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stuart W. Hicks with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stuart W. Hicks more than expected).
Fields of papers citing papers by Stuart W. Hicks
This network shows the impact of papers produced by Stuart W. Hicks. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stuart W. Hicks. The network helps show where Stuart W. Hicks may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Stuart W. Hicks, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2020 | 4 | |
| 3 | 2019 | 18 | |
| 4 | 2017 | 27 | |
| 5 | 2016 | 17 | |
| 6 | 2014 | 3 | |
| 7 | 2013 | 96 | |
| 8 | 2011 | 34 | |
| 9 | 2011 | 72 | |
| 10 | 2009 | 150 | |
| 11 | 2009 | 73 | |
| 12 | 2006 | 51 | |
| 13 | 2006 | 43 | |
| 14 | 2006 | 40 | |
| 15 | 2005 | 152 | |
| 16 | 2005 | 39 | |
| 17 | 2003 | 69 | |
| 18 | 2002 | 66 | |
| 19 | 1999 | 62 | |
| 20 | 1998 | 46 |
About Stuart W. Hicks
Stuart W. Hicks is a scholar working on Physiology, Cell Biology, Oncology, Virology and Endocrinology, having authored 28 papers that have together received 1.1k indexed citations. Recurring topics across this work include Monoclonal and Polyclonal Antibodies Research (8 papers), CAR-T cell therapy research (7 papers), Endoplasmic Reticulum Stress and Disease (6 papers), Cellular transport and secretion (6 papers), HER2/EGFR in Cancer Research (6 papers), Immune Cell Function and Interaction (5 papers), Calcium signaling and nucleotide metabolism (3 papers) and HIV Research and Treatment (2 papers). The work is most often cited by research in Endocrinology (119 citations), Cell Biology (305 citations), Physiology (58 citations), Virology (60 citations) and Immunology (247 citations). Stuart W. Hicks has collaborated with scholars based in United States, Switzerland and Ukraine. Frequent co-authors include Carolyn E. Machamer, Jorge E. Galán, Cindy M. Quezada, C. Erec Stebbins, Guillaume Charron, Federica Pericle, Giuseppe Sconocchia, David M. Segal, Howard C. Hang and David M. Zuckerman. Their work appears in journals such as Cancer Research, Journal of Biological Chemistry, mAbs, Blood and Traffic.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.