Anetta Härtlová
- Molecular Biology top 10%
- Immunology top 5%
- Oncology top 10%
- Infectious Diseases top 5%
- Epidemiology
- Co-authors
- Nelson O. GekaraSaskia F. ErttmannMatthias TrostTorben EkUlrike ReschJiří StulíkLisa M. NilssonJonas A. Nilsson
- Topics
- Immunotherapy and Immune Responses (8 papers)Phagocytosis and Immune Regulation (7 papers)Immune cells in cancer (7 papers)
- Journals
- Nucleic Acids ResearchSHILAP Revista de lepidopterologíaThe EMBO Journal
- Partner nations
- SwedenUnited KingdomCzechia
In The Last Decade
Anetta Härtlová
34 papers receiving 2.0k citations
Hit Papers
Peers
Comparison fields: 5 of 104
- Molecular Biology 1.1k
- Immunology 787
- Oncology 291
- Infectious Diseases 246
- Epidemiology 241
Countries citing papers authored by Anetta Härtlová
This map shows the geographic impact of Anetta Härtlová's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Anetta Härtlová with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Anetta Härtlová more than expected).
Fields of papers citing papers by Anetta Härtlová
This network shows the impact of papers produced by Anetta Härtlová. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Anetta Härtlová. The network helps show where Anetta Härtlová may publish in the future.
Co-authorship network of co-authors of Anetta Härtlová
This figure shows the co-authorship network connecting the top 25 collaborators of Anetta Härtlová. A scholar is included among the top collaborators of Anetta Härtlová based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Anetta Härtlová. Anetta Härtlová is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 4 | |
| 3 | 8 | |
| 4 | 0 | |
| 5 | 14 | |
| 6 | 7 | |
| 7 | 17 | |
| 8 | 6 | |
| 9 | 14 | |
| 10 | 74 | |
| 11 | 18 | |
| 12 | 126 | |
| 13 | 80 | |
| 14 | 19 | |
| 15 | 4 | |
| 16 | 37 | |
| 17 | 3 | |
| 18 | DNA Damage Primes the Type I Interferon System via the Cytosolic DNA Sensor STING to Promote Anti-Microbial Innate Immunitybreakdown → | 537 |
| 19 | 42 | |
| 20 | 68 |
About Anetta Härtlová
Anetta Härtlová is a scholar working on Immunology, Endocrinology and Molecular Medicine, having authored 36 papers that have together received 2.0k indexed citations. Recurring topics across this work include Immunotherapy and Immune Responses (8 papers), Phagocytosis and Immune Regulation (7 papers) and Immune cells in cancer (7 papers). The work is most often cited by research in Immunology (787 citations), Molecular Biology (1.1k citations) and Infectious Diseases (246 citations). Anetta Härtlová has collaborated with scholars based in Sweden, United Kingdom and Czechia. Frequent co-authors include Nelson O. Gekara, Saskia F. Erttmann, Matthias Trost, Torben Ek, Ulrike Resch, Jiří Stulík, Lisa M. Nilsson, Jonas A. Nilsson, Stefan Lienenklaus and Andrea Kröger. Their work appears in journals such as Nucleic Acids Research, SHILAP Revista de lepidopterología and The EMBO Journal.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.