Steve E. Humphries

107.9k total citations · 3 hit papers
872 papers, 42.3k citations indexed

About

Steve E. Humphries is a scholar working on Surgery, Endocrinology, Diabetes and Metabolism and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Steve E. Humphries has authored 872 papers receiving a total of 42.3k indexed citations (citations by other indexed papers that have themselves been cited), including 372 papers in Surgery, 224 papers in Endocrinology, Diabetes and Metabolism and 210 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Steve E. Humphries's work include Lipoproteins and Cardiovascular Health (326 papers), Diabetes, Cardiovascular Risks, and Lipoproteins (178 papers) and Genetic Associations and Epidemiology (127 papers). Steve E. Humphries is often cited by papers focused on Lipoproteins and Cardiovascular Health (326 papers), Diabetes, Cardiovascular Risks, and Lipoproteins (178 papers) and Genetic Associations and Epidemiology (127 papers). Steve E. Humphries collaborates with scholars based in United Kingdom, United States and France. Steve E. Humphries's co-authors include Philippa J. Talmud, Vidya Mohamed‐Ali, Hugh Montgomery, Meena Kumari, Anders Hamsten, Jackie A. Cooper, John Yudkin, George J. Miller, Adriano Henney and Aroon D. Hingorani and has published in prestigious journals such as Nature, New England Journal of Medicine and Cell.

In The Last Decade

Steve E. Humphries

863 papers receiving 41.0k citations

Hit Papers

The effect of novel polymorphisms in the interleukin-6 (I... 1998 2026 2007 2016 1998 2000 2013 500 1000 1.5k
What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

  1. The effect of novel polymorphisms in the interleukin-6 (IL-6) gene on IL-6 transcription and plasma IL-6 levels, and an association with systemic-onset juvenile chronic arthritis.
    1998 1.9k citations Steve E. Humphries et al. profile →
  2. Inflammation, obesity, stress and coronary heart disease: is interleukin-6 the link?
    2000 1.4k citations Meena Kumari, Steve E. Humphries et al. Atherosclerosis profile →
  3. Use of low-density lipoprotein cholesterol gene score to distinguish patients with polygenic and monogenic familial hypercholesterolaemia: a case-control study
    2013 411 citations Philippa J. Talmud, Sonia Shah et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Steve E. Humphries United Kingdom 94 14.3k 10.0k 10.0k 7.9k 7.7k 872 42.3k
Eric Boerwinkle United States 95 11.1k 0.8× 10.4k 1.0× 11.0k 1.1× 13.9k 1.8× 13.1k 1.7× 811 47.6k
Anne Tybjærg‐Hansen Denmark 81 11.0k 0.8× 9.3k 0.9× 7.4k 0.7× 5.1k 0.6× 4.8k 0.6× 345 28.3k
Børge G. Nordestgaard Denmark 120 27.1k 1.9× 18.8k 1.9× 16.7k 1.7× 7.8k 1.0× 4.9k 0.6× 953 65.2k
L. Adrienne Cupples United States 112 6.6k 0.5× 7.0k 0.7× 8.7k 0.9× 7.9k 1.0× 7.1k 0.9× 539 43.4k
Jacques Genest Canada 87 16.0k 1.1× 10.5k 1.1× 13.0k 1.3× 8.6k 1.1× 1.4k 0.2× 709 40.4k
Takashi Kadowaki Japan 119 10.4k 0.7× 14.1k 1.4× 7.6k 0.8× 25.1k 3.2× 5.3k 0.7× 869 62.0k
Stephen Burgess United Kingdom 78 5.5k 0.4× 4.7k 0.5× 4.4k 0.4× 11.2k 1.4× 22.5k 2.9× 334 48.2k
John Danesh United Kingdom 59 5.8k 0.4× 4.0k 0.4× 5.5k 0.5× 3.9k 0.5× 4.6k 0.6× 129 29.9k
Ulf dé Fairé Sweden 81 4.4k 0.3× 8.1k 0.8× 13.6k 1.4× 3.7k 0.5× 2.3k 0.3× 381 30.4k
Kenneth Walsh United States 113 7.2k 0.5× 2.7k 0.3× 9.8k 1.0× 28.3k 3.6× 2.9k 0.4× 462 54.0k

Countries citing papers authored by Steve E. Humphries

Since Specialization
Citations

This map shows the geographic impact of Steve E. Humphries's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Steve E. Humphries with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Steve E. Humphries more than expected).

Fields of papers citing papers by Steve E. Humphries

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Steve E. Humphries. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Steve E. Humphries. The network helps show where Steve E. Humphries may publish in the future.

Co-authorship network of co-authors of Steve E. Humphries

This figure shows the co-authorship network connecting the top 25 collaborators of Steve E. Humphries. A scholar is included among the top collaborators of Steve E. Humphries based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Steve E. Humphries. Steve E. Humphries is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Humphries, Steve E. & Marta Futema. (2025). Genetic Determinants of the Familial Hypercholesterolaemia Phenotype. Annals of Human Genetics. 89(5). 293–304. 1 indexed citations
2.
3.
Gratton, Jasmine, Steve E. Humphries, Amand F. Schmidt, et al.. (2023). Modelling a two-stage adult population screen for autosomal dominant familial hypercholesterolaemia: cross-sectional analysis within the UK Biobank. SHILAP Revista de lepidopterología. 1(1). e000021–e000021. 2 indexed citations
4.
Alves, Ana Catarina, Ana Margarida Medeiros, Joana Rita Chora, et al.. (2019). The familial hypercholesterolaemia phenotype: Monogenic familial hypercholesterolaemia, polygenic hypercholesterolaemia and other causes. Clinical Genetics. 97(3). 457–466. 19 indexed citations
5.
Shabana, Shabana, et al.. (2016). Association of ACE and NOS3 Gene Polymorphisms with Blood Pressure in a Case Control Study of Coronary Artery Disease in Punjab, Pakistan. UCL Discovery (University College London). 3 indexed citations
6.
Ramaswami, Uma, Jackie A. Cooper, & Steve E. Humphries. (2016). The UK Paediatric Familial Hypercholesterolaemia Register: preliminary data. Archives of Disease in Childhood. 102(3). 255–260. 35 indexed citations
7.
Yiannakouris, Nikos, Jackie A. Cooper, Sonia Shah, et al.. (2011). IRS1 gene variants, dysglycaemic metabolic changes and type-2 diabetes risk. Nutrition Metabolism and Cardiovascular Diseases. 22(12). 1024–1030. 20 indexed citations
8.
Stephens, Jeffrey W., et al.. (2004). The APOA4 T347S variant is associated with reduced plasma TAOS in subjects with diabetes mellitus and cardiovascular disease. Journal of Lipid Research. 45(8). 1565–1571. 19 indexed citations
9.
Serrano, Norma C., Francesco Paolo Zito, Lyle V. Jones, et al.. (2003). A polymorphism in the human CRP gene influences basal and stimulated CRP levels: Implications for the prediction and pathogenesis of coronary heart disease. Atherosclerosis. 168(1). 192. 4 indexed citations
10.
Harding, David, S Dhamrait, Hugh Montgomery, et al.. (2002). Does IL6-174 genotype predict the development of septicaemia in preterm infants?. UCL Discovery (University College London). 1 indexed citations
11.
Humphries, Steve E., Lars Berglund, Carmen R. Isasi, et al.. (2002). Loci for CETP, LPL, LIPC, and APOC3 affect plasma lipoprotein size and sub-population distribution in Hispanic and non-Hispanic white subjects: the Columbia University BioMarkers Study.. PubMed. 12(4). 163–72. 18 indexed citations
12.
Piťha, Jan, et al.. (2001). Polymorphism in the lipoprotein lipase and hepatic lipase genes and plasma lipid values in the Czech population. Physiological Research. 345–351. 8 indexed citations
13.
Xu, Weiming, Steve E. Humphries, Masafumi Tomita, et al.. (2000). Survey of the Allelic Frequency of a NOS2A Promoter Microsatellite in Human Populations: Assessment of the NOS2A Gene and Predisposition to Infectious Disease. Nitric Oxide. 4(4). 379–383. 46 indexed citations
14.
Bolla, Manjeet K., George J. Miller, Derek M. Yellon, et al.. (1998). Analysis of the Association of A Heat Shock Protein70‐1 Gene Promoter Polymorphism With Myocardial Infarction and Coronary Risk Traits. Disease Markers. 13(4). 227–235. 20 indexed citations
15.
Talmud, Philippa J., et al.. (1996). Lipoprotein lipase -93T/G transition is associated with lower plasma triglyceride levels and increases promoter activity in vitro. UCL Discovery (University College London). 1 indexed citations
16.
17.
Ramakrishnan, R, Philippa J. Talmud, Steve E. Humphries, et al.. (1995). RELATIONSHIP OF APOPROTEIN-B AND APOPROTEIN-CIII POLYMORPHISMS WITH POSTPRANDIAL LIPEMIA. UCL Discovery (University College London). 1 indexed citations
18.
Peacock, R., Gerald F. Watts, Sundhiya Mandalia, et al.. (1994). Associations between genotypes of the apolipoprotein E, B, AI-CIII-AIV and lipoprotein lipase genes and coronary artery disease in STARS. Nutrition Metabolism and Cardiovascular Diseases. 44. 128–136. 1 indexed citations
19.
Humphries, Steve E., et al.. (1993). A labour of love : the experience of parenthood in Britain 1900-1950. 8 indexed citations
20.
Humphries, Steve E., et al.. (1992). Out of sight : the experience of disability, 1900-1950. 40 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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