Stephen P. Fink
- Molecular Biology
- Cancer Research top 10%
- Oncology top 10%
- Pharmacology top 10%
- Cell Biology top 10%
- Co-authors
- Lawrence J. MarnettSanford D. MarkowitzG. Ramachandra ReddyRobley C. WilliamsJames K. V. WillsonDebra MikkolaJoseph WillisJames Lutterbaugh
- Topics
- Inflammatory mediators and NSAID effects (10 papers)Genetic factors in colorectal cancer (8 papers)DNA Repair Mechanisms (7 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryJournal of Clinical Investigation
- Partner nations
- United StatesSouth KoreaCanada
In The Last Decade
Stephen P. Fink
29 papers receiving 1.0k citations
Peers
Comparison fields: 5 of 96
- Molecular Biology 646
- Cancer Research 292
- Oncology 256
- Pharmacology 163
- Cell Biology 160
Countries citing papers authored by Stephen P. Fink
This map shows the geographic impact of Stephen P. Fink's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stephen P. Fink with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stephen P. Fink more than expected).
Fields of papers citing papers by Stephen P. Fink
This network shows the impact of papers produced by Stephen P. Fink. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stephen P. Fink. The network helps show where Stephen P. Fink may publish in the future.
Co-authorship network of co-authors of Stephen P. Fink
This figure shows the co-authorship network connecting the top 25 collaborators of Stephen P. Fink. A scholar is included among the top collaborators of Stephen P. Fink based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stephen P. Fink. Stephen P. Fink is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 7 | |
| 2 | 2 | |
| 3 | 8 | |
| 4 | 0 | |
| 5 | 9 | |
| 6 | 16 | |
| 7 | 27 | |
| 8 | 13 | |
| 9 | 11 | |
| 10 | 6 | |
| 11 | 10 | |
| 12 | 23 | |
| 13 | 40 | |
| 14 | 23 | |
| 15 | 79 | |
| 16 | 1 | |
| 17 | 7 | |
| 18 | 30 | |
| 19 | 119 | |
| 20 | 80 |
About Stephen P. Fink
Stephen P. Fink is a scholar working on Pharmacology, Oncology and Pathology and Forensic Medicine, having authored 31 papers that have together received 1.1k indexed citations. Recurring topics across this work include Inflammatory mediators and NSAID effects (10 papers), Genetic factors in colorectal cancer (8 papers) and DNA Repair Mechanisms (7 papers). The work is most often cited by research in Cancer Research (292 citations), Cell Biology (160 citations) and Pharmacology (163 citations). Stephen P. Fink has collaborated with scholars based in United States, South Korea and Canada. Frequent co-authors include Lawrence J. Marnett, Sanford D. Markowitz, G. Ramachandra Reddy, Robley C. Williams, James K. V. Willson, Debra Mikkola, Joseph Willis, James Lutterbaugh, Petra Platzer and Jill S. Barnholtz‐Sloan. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.