Stephen Baylin
- Oncology top 1%
- Pancreatic and Hepatic Oncology Research 2
- Cancer-related Molecular Pathways 1
- Cancer Research top 2%
- Cancer-related molecular mechanisms research 1
- Biotechnology top 1%
- Cancer Research and Treatments 1
- Molecular Biology top 10%
- RNA modifications and cancer 2
- Epigenetics and DNA Methylation 2
- Cancer-related gene regulation 2
-
- Neuroendocrine Tumor Research Advances 2
- Co-authors
- Nancy E. DavidsonPeter DevileeBert VogelsteinJ. Milburn JessupKaren R. ClearyRama ModaliFrancis S. CollinsAntonette C. Preisinger
- Cited by
- OncologyCancer ResearchBiotechnology
- Partner nations
- United StatesSpainNetherlands
In The Last Decade
Stephen Baylin
6 papers receiving 2.5k citations
Hit Papers
Peers
Comparison fields: 5 of 91
- Oncology 1.7k
- Cancer Research 687
- Biotechnology 332
- Pathology and Forensic Medicine 423
- Molecular Biology 1.2k
Countries citing papers authored by Stephen Baylin
This map shows the geographic impact of Stephen Baylin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stephen Baylin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stephen Baylin more than expected).
Fields of papers citing papers by Stephen Baylin
This network shows the impact of papers produced by Stephen Baylin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stephen Baylin. The network helps show where Stephen Baylin may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Stephen Baylin, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2023 | 0 | |
| 2 | 2021 | 2 | |
| 3 | 2016 | 17 | |
| 4 | 2015 | 1 | |
| 5 | 2014 | 34 | |
| 6 | Mechanisms of inactivation of E-cadherin in breast cancer cell lines. | 1998 | 125 |
| 7 | Mutations in the p53 gene occur in diverse human tumour typesbreakdown → | 1989 | 2370 |
About Stephen Baylin
Stephen Baylin is a scholar working on Cancer Research, Biotechnology, Oncology, Molecular Biology and Epidemiology, having authored 7 papers that have together received 2.5k indexed citations. Recurring topics across this work include Pancreatic and Hepatic Oncology Research (2 papers), RNA modifications and cancer (2 papers), Neuroendocrine Tumor Research Advances (2 papers), Epigenetics and DNA Methylation (2 papers), Cancer-related gene regulation (2 papers), Cancer Research and Treatments (1 paper), Cancer-related Molecular Pathways (1 paper) and Cancer-related molecular mechanisms research (1 paper). The work is most often cited by research in Oncology (1.7k citations), Cancer Research (687 citations), Biotechnology (332 citations), Pathology and Forensic Medicine (423 citations) and Molecular Biology (1.2k citations). Stephen Baylin has collaborated with scholars based in United States, Spain and Netherlands. Frequent co-authors include Nancy E. Davidson, Peter Devilee, Bert Vogelstein, J. Milburn Jessup, Karen R. Cleary, Rama Modali, Francis S. Collins, Antonette C. Preisinger, Curtis C. Harris and Thomas W. Glover. Their work appears in journals such as Journal of Clinical Oncology, Nature, Carcinogenesis, Oncotarget and Cancer Research.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.