Stefan G. Lechner

2.8k total citations
48 papers, 1.9k citations indexed

About

Stefan G. Lechner is a scholar working on Physiology, Molecular Biology and Sensory Systems. According to data from OpenAlex, Stefan G. Lechner has authored 48 papers receiving a total of 1.9k indexed citations (citations by other indexed papers that have themselves been cited), including 29 papers in Physiology, 24 papers in Molecular Biology and 14 papers in Sensory Systems. Recurrent topics in Stefan G. Lechner's work include Ion channel regulation and function (19 papers), Pain Mechanisms and Treatments (15 papers) and Erythrocyte Function and Pathophysiology (12 papers). Stefan G. Lechner is often cited by papers focused on Ion channel regulation and function (19 papers), Pain Mechanisms and Treatments (15 papers) and Erythrocyte Function and Pathophysiology (12 papers). Stefan G. Lechner collaborates with scholars based in Germany, Austria and Italy. Stefan G. Lechner's co-authors include Gary R. Lewin, Francisco J. Taberner, Ewan St. John Smith, Vincenzo Prato, Stefan Boehm, Paul A. Heppenstall, Hagen Wende, Carmen Birchmeier, Werner Zwerschke and Maria C. Mitterberger and has published in prestigious journals such as Nature, Science and Proceedings of the National Academy of Sciences.

In The Last Decade

Stefan G. Lechner

47 papers receiving 1.9k citations

Peers

Stefan G. Lechner

PHYSI

CMN

Reza Sharif‐Naeini Canada

Michael P. Jankowski United States

Andy Weyer United States

Hagen Wende Germany

Yoshiyuki Shibukawa Japan

Atsushi Yoshida Japan

Chuansong Wang United States

Francesca Pagani Italy

Giulia Bolasco Italy

Reza Sharif‐Naeini Canada

Stefan G. Lechner

984 ×1.0

PHYSI

1.0k ×1.1

710 ×1.3

CMN

421 ×1.3

134 ×0.8

Citations per year, relative to Stefan G. Lechner Stefan G. Lechner (= 1×) peers Reza Sharif‐Naeini

Countries citing papers authored by Stefan G. Lechner

Since Specialization

Citations

This map shows the geographic impact of Stefan G. Lechner's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stefan G. Lechner with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stefan G. Lechner more than expected).

Fields of papers citing papers by Stefan G. Lechner

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stefan G. Lechner. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stefan G. Lechner. The network helps show where Stefan G. Lechner may publish in the future.

Co-authorship network of co-authors of Stefan G. Lechner

This figure shows the co-authorship network connecting the top 25 collaborators of Stefan G. Lechner. A scholar is included among the top collaborators of Stefan G. Lechner based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stefan G. Lechner. Stefan G. Lechner is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Verkest, Clément, et al.. (2025). Cluster nanoarchitecture and structural diversity of PIEZO1 at rest and during activation in intact cells. Science Advances. 11(43). eady8052–eady8052. 1 indexed citations

Verkest, Clément, et al.. (2025). 3D-MINFLUX nanoscopy reveals distinct allosteric mechanisms for activation and modulation of PIEZO1 by Yoda1. Nature Communications. 16(1). 11192–11192.

Lechner, Stefan G., et al.. (2024). The activation thresholds and inactivation kinetics of poking-evoked PIEZO1 and PIEZO2 currents are sensitive to subtle variations in mechanical stimulation parameters. Channels. 18(1). 2355123–2355123. 7 indexed citations

Sliwinski, Christopher, Francisco J. Taberner, Lisa Weiss, et al.. (2023). Contribution of mechanoreceptors to spinal cord injury–induced mechanical allodynia. Pain. 165(6). 1336–1347. 3 indexed citations

Verkest, Clément, et al.. (2023). PKA mediates modality-specific modulation of the mechanically gated ion channel PIEZO2. Journal of Biological Chemistry. 299(6). 104782–104782. 9 indexed citations

Gangadharan, Vijayan, Hongwei Zheng, Francisco J. Taberner, et al.. (2022). Neuropathic pain caused by miswiring and abnormal end organ targeting. Nature. 606(7912). 137–145. 68 indexed citations

Prato, Vincenzo, Stefan G. Lechner, Gerbrand J. Groen, et al.. (2020). TTX-Resistant Sodium Channels Functionally Separate Silent From Polymodal C-nociceptors. Frontiers in Cellular Neuroscience. 14. 13–13. 7 indexed citations

Arokiaraj, Cynthia M., Francisco J. Taberner, Linda Nocchi, et al.. (2018). Control of mechanical pain hypersensitivity in mice through ligand-targeted photoablation of TrkB-positive sensory neurons. Nature Communications. 9(1). 1640–1640. 79 indexed citations

Prato, Vincenzo, Francisco J. Taberner, James R.F. Hockley, et al.. (2017). Functional and Molecular Characterization of Mechanoinsensitive “Silent” Nociceptors. Cell Reports. 21(11). 3102–3115. 124 indexed citations

10.

Prato, Vincenzo, Francisco J. Taberner, Hagen Wende, et al.. (2016). Touch Receptor-Derived Sensory Information Alleviates Acute Pain Signaling and Fine-Tunes Nociceptive Reflex Coordination. Neuron. 93(1). 179–193. 152 indexed citations

11.

Lewin, Gary R., Stefan G. Lechner, & Ewan St. John Smith. (2014). Nerve Growth Factor and Nociception: From Experimental Embryology to New Analgesic Therapy. Handbook of experimental pharmacology. 220. 251–282. 54 indexed citations

12.

Wende, Hagen, Stefan G. Lechner, Cyril Chéret, et al.. (2012). The Transcription Factor c-Maf Controls Touch Receptor Development and Function. Science. 335(6074). 1373–1376. 124 indexed citations

13.

Mitterberger, Maria C., Stefan G. Lechner, Monika Mattesich, et al.. (2012). DLK1(PREF1) is a negative regulator of adipogenesis in CD105+/CD90+/CD34+/CD31−/FABP4− adipose-derived stromal cells from subcutaneous abdominal fat pats of adult women. Stem Cell Research. 9(1). 35–48. 66 indexed citations

14.

Wende, Hagen, Stefan G. Lechner, & Carmen Birchmeier. (2012). The transcription factor c-Maf in sensory neuron development. Transcription. 3(6). 285–289. 7 indexed citations

15.

Frenzel, H, Jörg E. Bohlender, Jens Tank, et al.. (2012). A Genetic Basis for Mechanosensory Traits in Humans. PLoS Biology. 10(5). e1001318–e1001318. 61 indexed citations

16.

Heidenreich, Matthias, Stefan G. Lechner, Vitya Vardanyan, et al.. (2011). KCNQ4 K+ channels tune mechanoreceptors for normal touch sensation in mouse and man. Nature Neuroscience. 15(1). 138–145. 82 indexed citations

17.

Kaiser, Andreas, Steffi Herold, Stefan G. Lechner, et al.. (2011). The human papillomavirus type 16 E7 oncoprotein targets Myc-interacting zinc-finger protein-1. Virology. 422(2). 242–253. 10 indexed citations

18.

Lechner, Stefan G., et al.. (2005). Presynaptic inhibition of transmitter release from rat sympathetic neurons by bradykinin. Journal of Neurochemistry. 93(5). 1110–1121. 13 indexed citations

19.

Lechner, Stefan G. & Stefan Boehm. (2004). Regulation of neuronal ion channels via P2Y receptors. Purinergic Signalling. 1(1). 31–41. 20 indexed citations

20.

Lechner, Stefan G., et al.. (2003). Activation of M₁ Muscarinic Receptors Triggers Transmitter Release from Rat Sympathetic Neurons Through an Inhibition of M‐Type K⁺ Channels. The Journal of Physiology. 553(3). 789–802. 31 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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