Stefan Feske

20.4k total citations · 6 hit papers
115 papers, 16.0k citations indexed

About

Stefan Feske is a scholar working on Sensory Systems, Molecular Biology and Immunology. According to data from OpenAlex, Stefan Feske has authored 115 papers receiving a total of 16.0k indexed citations (citations by other indexed papers that have themselves been cited), including 79 papers in Sensory Systems, 51 papers in Molecular Biology and 41 papers in Immunology. Recurrent topics in Stefan Feske's work include Ion Channels and Receptors (79 papers), Ion channel regulation and function (28 papers) and Immune Cell Function and Interaction (24 papers). Stefan Feske is often cited by papers focused on Ion Channels and Receptors (79 papers), Ion channel regulation and function (28 papers) and Immune Cell Function and Interaction (24 papers). Stefan Feske collaborates with scholars based in United States, Germany and Canada. Stefan Feske's co-authors include Anjana Rao, Murali Prakriya, Patrick G. Hogan, Sonal Srikanth, Yousang Gwack, Edward Y. Skolnik, Richard S. Lewis, Rodrigo S. Lacruz, Bogdan Tanasă and Heike Wulff and has published in prestigious journals such as Nature, New England Journal of Medicine and Cell.

In The Last Decade

Stefan Feske

112 papers receiving 15.8k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

A mutation in Orai1 causes immune deficiency by abrogating CRAC channel function

2006 1.8k citations Stefan Feske, Murali Prakriya et al. profile →
Orai1 is an essential pore subunit of the CRAC channel

2006 1.1k citations Murali Prakriya, Stefan Feske et al. profile →
Phosphoenolpyruvate Is a Metabolic Checkpoint of Anti-tumor T Cell Responses

2015 1.1k citations Stefan Feske et al. profile →
Calcium signalling in lymphocyte activation and disease

2007 828 citations Stefan Feske profile →
Ion Channels in Innate and Adaptive Immunity

2015 573 citations Stefan Feske et al. profile →
Diseases caused by mutations in ORAI1 and STIM1

2015 327 citations Rodrigo S. Lacruz, Stefan Feske profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Stefan Feske United States	61	8.0k	7.0k	4.4k	3.4k	1.7k	115	16.0k
Jean-Pierre Kinet United States	37	1.9k 0.2×	3.0k 0.4×	4.7k 1.1×	594 0.2×	260 0.2×	50	9.6k
Andrew M. Scharenberg United States	57	2.6k 0.3×	5.6k 0.8×	3.8k 0.9×	563 0.2×	142 0.1×	126	12.7k
Michael A. Beaven United States	66	529 0.1×	6.7k 1.0×	6.3k 1.4×	1.2k 0.3×	179 0.1×	227	14.7k
Zui Pan United States	39	893 0.1×	3.4k 0.5×	442 0.1×	822 0.2×	255 0.2×	116	6.1k
Ole Thastrup Denmark	34	1.2k 0.1×	5.3k 0.8×	624 0.1×	2.6k 0.8×	62 0.0×	76	7.9k
Wei‐Guo Zhu China	59	828 0.1×	7.3k 1.1×	607 0.1×	703 0.2×	83 0.0×	253	11.5k
Young Yang South Korea	45	626 0.1×	4.4k 0.6×	1.3k 0.3×	699 0.2×	113 0.1×	193	7.6k
Hideki Sumimoto Japan	56	488 0.1×	4.5k 0.6×	4.1k 0.9×	441 0.1×	95 0.1×	177	10.3k
C. Michael Knudson United States	47	238 0.0×	7.0k 1.0×	1.4k 0.3×	2.1k 0.6×	140 0.1×	82	10.7k
Timothy F. Walseth United States	54	2.9k 0.4×	3.7k 0.5×	713 0.2×	895 0.3×	20 0.0×	137	9.7k

Countries citing papers authored by Stefan Feske

Since Specialization

Citations

This map shows the geographic impact of Stefan Feske's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Stefan Feske with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Stefan Feske more than expected).

Fields of papers citing papers by Stefan Feske

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Stefan Feske. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Stefan Feske. The network helps show where Stefan Feske may publish in the future.

Co-authorship network of co-authors of Stefan Feske

This figure shows the co-authorship network connecting the top 25 collaborators of Stefan Feske. A scholar is included among the top collaborators of Stefan Feske based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Stefan Feske. Stefan Feske is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Zhong, Li, Yin‐Hu Wang, Sascha Kahlfuß, et al.. (2025). STIM1-mediated NFAT signaling synergizes with STAT1 to control T-bet expression and TH1 differentiation. Nature Immunology. 26(3). 484–496. 1 indexed citations

Wang, Yin‐Hu, et al.. (2025). Machine learning approach to single cell transcriptomic analysis of Sjogren's disease reveals altered activation states of B and T lymphocytes. Journal of Autoimmunity. 154. 103419–103419.

Wang, Yin‐Hu, George Maiti, Fang Zhou, et al.. (2024). IFN-γ–producing T _H 1 cells and dysfunctional regulatory T cells contribute to the pathogenesis of Sjögren’s disease. Science Translational Medicine. 16(778). eado4856–eado4856. 15 indexed citations

Feske, Stefan, Francesco Colucci, & William A. Coetzee. (2024). Do KATP channels have a role in immunity?. Frontiers in Immunology. 15. 1484971–1484971. 1 indexed citations

Wei, Dongyu, Hareram Birla, Yannong Dou, et al.. (2023). PGE2 Potentiates Orai1-Mediated Calcium Entry Contributing to Peripheral Sensitization. Journal of Neuroscience. 44(1). e0329232023–e0329232023. 2 indexed citations

Emrich, Scott M., Ryan E. Yoast, Xuexin Zhang, et al.. (2023). Orai3 and Orai1 mediate CRAC channel function and metabolic reprogramming in B cells. eLife. 12. 13 indexed citations

Ito-Kureha, Taku, Cristina Leoni, Kayla Borland, et al.. (2022). The function of Wtap in N6-adenosine methylation of mRNAs controls T cell receptor signaling and survival of T cells. Nature Immunology. 23(8). 1208–1221. 64 indexed citations

Wang, Yin‐Hu, Lucile Noyer, Sascha Kahlfuß, et al.. (2022). Distinct roles of ORAI1 in T cell–mediated allergic airway inflammation and immunity to influenza A virus infection. Science Advances. 8(40). eabn6552–eabn6552. 15 indexed citations

Wang, Liwei, Lucile Noyer, Yin‐Hu Wang, et al.. (2022). ORAI3 is dispensable for store-operated Ca2+ entry and immune responses by lymphocytes and macrophages. The Journal of General Physiology. 154(10). 7 indexed citations

10.

Behrens, Gesine, Stephanie L. Edelmann, Thomas Monecke, et al.. (2021). Disrupting Roquin-1 interaction with Regnase-1 induces autoimmunity and enhances antitumor responses. Nature Immunology. 22(12). 1563–1576. 36 indexed citations

11.

Subedi, Krishna Prasad, Hwei Ling Ong, Lucile Noyer, et al.. (2020). STIM2 targets Orai1/STIM1 to the AKAP79 signaling complex and confers coupling of Ca ²⁺ entry with NFAT1 activation. Proceedings of the National Academy of Sciences. 117(28). 16638–16648. 37 indexed citations

12.

Eckstein, Miriam, Martin Vaeth, Francisco J. Aulestia, et al.. (2019). Differential regulation of Ca ²⁺ influx by ORAI channels mediates enamel mineralization. Science Signaling. 12(578). 42 indexed citations

13.

Maus, Máté, Mario Ćuk, Bindi Patel, et al.. (2017). Store-Operated Ca 2+ Entry Controls Induction of Lipolysis and the Transcriptional Reprogramming to Lipid Metabolism. Cell Metabolism. 25(3). 698–712. 126 indexed citations

14.

Maus, Máté, Amit Jairaman, Peter B. Stathopulos, et al.. (2015). Missense mutation in immunodeficient patients shows the multifunctional roles of coiled-coil domain 3 (CC3) in STIM1 activation. Proceedings of the National Academy of Sciences. 112(19). 6206–6211. 50 indexed citations

15.

Ong, Hwei Ling, Lorena Brito de Souza, Changyu Zheng, et al.. (2015). STIM2 enhances receptor-stimulated Ca ²⁺ signaling by promoting recruitment of STIM1 to the endoplasmic reticulum–plasma membrane junctions. Science Signaling. 8(359). ra3–ra3. 81 indexed citations

16.

Maul‐Pavicic, Andrea, Samuel C. C. Chiang, Anne Rensing‐Ehl, et al.. (2011). ORAI1-mediated calcium influx is required for human cytotoxic lymphocyte degranulation and target cell lysis. Proceedings of the National Academy of Sciences. 108(8). 3324–3329. 165 indexed citations

17.

Feske, Stefan. (2010). CRAC channelopathies. Pflügers Archiv - European Journal of Physiology. 460(2). 417–435. 168 indexed citations

18.

Foerster, Christian, Nadine Voelxen, Mirzokhid Rakhmanov, et al.. (2010). B Cell Receptor-Mediated Calcium Signaling Is Impaired in B Lymphocytes of Type Ia Patients with Common Variable Immunodeficiency. The Journal of Immunology. 184(12). 7305–7313. 56 indexed citations

19.

Lür, György, Lee P. Haynes, Ian A. Prior, et al.. (2009). Ribosome-free Terminals of Rough ER Allow Formation of STIM1 Puncta and Segregation of STIM1 from IP3 Receptors. Current Biology. 19(19). 1648–1653. 108 indexed citations

20.

McCarl, Christie‐Ann, Capucine Pïcard, Sara Khalil, et al.. (2009). ORAI1 deficiency and lack of store-operated Ca2+ entry cause immunodeficiency, myopathy, and ectodermal dysplasia. Journal of Allergy and Clinical Immunology. 124(6). 1311–1318.e7. 256 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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