Miriam Eckstein

20 papers receiving 1.2k citations

Hit Papers

Mitochondrial dysfunction promotes the transition of precursor to terminally exhausted T cells through HIF-1α-mediated glycolytic reprogramming 2023 · 135 citations
13520222026202320244080120

Peers

Miriam Eckstein
Comparison fields: 5 of 89
  • Sensory Systems 323
  • Immunology 391
  • Physiology 58
  • Biochemistry 55
  • Rheumatology 133
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Fernando de Miguel United States
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Citations per field
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Citations per year

Countries citing papers authored by Miriam Eckstein

Since Specialization
Citations

This map shows the geographic impact of Miriam Eckstein's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Miriam Eckstein with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Miriam Eckstein more than expected).

Fields of papers citing papers by Miriam Eckstein

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Miriam Eckstein. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Miriam Eckstein. The network helps show where Miriam Eckstein may publish in the future.

Co-authors

The 25 scholars most cited alongside Miriam Eckstein, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.

Border = papers with Miriam Eckstein Line = papers co-authored together Miriam Eckstein links everyone, so they are left out of the graph.

All Works

20 of 20 papers shown
#Work
1 2017201
2 2017167
3
The glucose transporter GLUT3 controls T helper 17 cell responses through glycolytic-epigenetic reprogramming
Hit paper breakdown →
2022140
4
Mitochondrial dysfunction promotes the transition of precursor to terminally exhausted T cells through HIF-1α-mediated glycolytic reprogramming
Hit paper breakdown →
2023135
5 201689
6 201668
7 201764
8 201946
9 201743
10 201942
11 201536
12 201633
13 201330
14 201522
15 201818
16 202316
17 201815
18 201813
19 20217
20 20092

About Miriam Eckstein

Miriam Eckstein is a scholar working on Sensory Systems, Toxicology, Rheumatology, Cell Biology and Immunology, having authored 20 papers that have together received 1.2k indexed citations. Recurring topics across this work include Ion Channels and Receptors (8 papers), Connexins and lens biology (6 papers), Immune Cell Function and Interaction (4 papers), Bone and Dental Protein Studies (4 papers), Cancer, Hypoxia, and Metabolism (3 papers), Aldose Reductase and Taurine (3 papers), T-cell and B-cell Immunology (2 papers) and Ion channel regulation and function (2 papers). The work is most often cited by research in Sensory Systems (323 citations), Immunology (391 citations), Physiology (58 citations), Biochemistry (55 citations) and Rheumatology (133 citations). Miriam Eckstein has collaborated with scholars based in United States, Germany and Canada. Frequent co-authors include Martin Vaeth, Stefan Feske, Rodrigo S. Lacruz, Jun J. Yang, Friederike Berberich‐Siebelt, Meerim K. Nurbaeva, Stuart E. Turvey, Scott B. Cameron, Richard Possemato and Sophia M. Hochrein. Their work appears in journals such as Nature Communications, Immunity, Cell Metabolism, Scientific Reports and Cell Calcium.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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