Srinivas Ghatta

1.5k total citations
21 papers, 942 citations indexed

About

Srinivas Ghatta is a scholar working on Molecular Biology, Sensory Systems and Cellular and Molecular Neuroscience. According to data from OpenAlex, Srinivas Ghatta has authored 21 papers receiving a total of 942 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Molecular Biology, 6 papers in Sensory Systems and 5 papers in Cellular and Molecular Neuroscience. Recurrent topics in Srinivas Ghatta's work include Ion Channels and Receptors (6 papers), Renal cell carcinoma treatment (2 papers) and Birth, Development, and Health (2 papers). Srinivas Ghatta is often cited by papers focused on Ion Channels and Receptors (6 papers), Renal cell carcinoma treatment (2 papers) and Birth, Development, and Health (2 papers). Srinivas Ghatta collaborates with scholars based in United States, United Kingdom and Denmark. Srinivas Ghatta's co-authors include Thomas E. Taylor‐Clark, Bradley J. Undem, Stephen T. O’Rourke, Xiaoping Xu, Michael Carr, M. Allen McAlexander, Donald W. MacGlashan, Praveen Shukla, Poduri Ramarao and P Bakke and has published in prestigious journals such as Journal of Clinical Oncology, Blood and The FASEB Journal.

In The Last Decade

Srinivas Ghatta

21 papers receiving 928 citations

Peers

Srinivas Ghatta
Ana I. Caceres United States
Raffaele Gatti Italy
Bret F. Bessac United States
Lihuan Liang United Kingdom
Kenjiro Matsumoto Japan
David J Hele United Kingdom
Krisztina Pohóczky Hungary
Hong Wei China
Ana I. Caceres United States
Srinivas Ghatta
Citations per year, relative to Srinivas Ghatta Srinivas Ghatta (= 1×) peers Ana I. Caceres

Countries citing papers authored by Srinivas Ghatta

Since Specialization
Citations

This map shows the geographic impact of Srinivas Ghatta's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Srinivas Ghatta with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Srinivas Ghatta more than expected).

Fields of papers citing papers by Srinivas Ghatta

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Srinivas Ghatta. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Srinivas Ghatta. The network helps show where Srinivas Ghatta may publish in the future.

Co-authorship network of co-authors of Srinivas Ghatta

This figure shows the co-authorship network connecting the top 25 collaborators of Srinivas Ghatta. A scholar is included among the top collaborators of Srinivas Ghatta based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Srinivas Ghatta. Srinivas Ghatta is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Srour, Samer A., Jad Chahoud, Alexandra Drakaki, et al.. (2025). ALLO-316 in advanced clear cell renal cell carcinoma (ccRCC): Updated results from the phase 1 TRAVERSE study.. Journal of Clinical Oncology. 43(16_suppl). 4508–4508. 4 indexed citations
2.
Srour, Samer A., Jad Chahoud, Alexandra Drakaki, et al.. (2024). 322 ALLO-316 in patients with advanced or metastatic clear cell renal cell carcinoma (ccRCC): updated safety and efficacy from the phase 1 TRAVERSE multicenter study. Regular and Young Investigator Award Abstracts. A373–A373. 5 indexed citations
3.
Mailankody, Sham, Jeffrey Matous, Michaela Liedtke, et al.. (2022). Universal Updated Phase 1 Data Highlights Role of Allogeneic Anti-BCMA ALLO-715 Therapy for Relapsed/Refractory Multiple Myeloma. Blood. 140(Supplement 1). 4620–4622. 7 indexed citations
4.
5.
Concin, Nicole, Ignace Vergote, Ulrik Lassen, et al.. (2018). A phase IIa study of tisotumab vedotin in patients with previously treated recurrent or metastatic cervical cancer: Updated analysis of full cervical expansion cohort. Annals of Oncology. 29. viii344–viii344. 1 indexed citations
6.
Shukla, Praveen, Srinivas Ghatta, Caleb O Lemley, et al.. (2014). Maternal nutrient restriction during pregnancy impairs an endothelium-derived hyperpolarizing factor-like pathway in sheep fetal coronary arteries. American Journal of Physiology-Heart and Circulatory Physiology. 307(2). H134–H142. 18 indexed citations
7.
Shukla, Praveen, et al.. (2011). Curcumin ((E,E)-1,7-bis(4-hydroxy-3-methoxyphenyl)-1,6-heptadiene-3,5-dione) activates and desensitizes the nociceptor ion channel TRPA1. Neuroscience Letters. 503(3). 157–162. 60 indexed citations
8.
9.
Zhu, Guohua, Amund Gulsvik, P Bakke, et al.. (2009). Association of TRPV4 gene polymorphisms with chronic obstructive pulmonary disease. Human Molecular Genetics. 18(11). 2053–2062. 88 indexed citations
10.
Taylor‐Clark, Thomas E., et al.. (2009). Nitrooleic Acid, an Endogenous Product of Nitrative Stress, Activates Nociceptive Sensory Nerves via the Direct Activation of TRPA1. Molecular Pharmacology. 75(4). 820–829. 145 indexed citations
11.
Ghatta, Srinivas, Irina M. Lozinskaya, Zuojun Lin, et al.. (2007). Acetic acid opens large-conductance Ca2+-activated K+ channels in guinea pig detrusor smooth muscle cells. European Journal of Pharmacology. 563(1-3). 203–208. 10 indexed citations
12.
Taylor‐Clark, Thomas E., Bradley J. Undem, Donald W. MacGlashan, et al.. (2007). Prostaglandin-Induced Activation of Nociceptive Neurons via Direct Interaction with Transient Receptor Potential A1 (TRPA1). Molecular Pharmacology. 73(2). 274–281. 241 indexed citations
13.
O’Rourke, Stephen T., et al.. (2007). Altered coronary vasodilator mechanisms in fetal sheep during maternal nutrient restriction. Journal of Molecular and Cellular Cardiology. 42(6). S228–S229. 1 indexed citations
14.
Ghatta, Srinivas, et al.. (2006). Role of endogenous hydrogen peroxide in the development of nitrate tolerance. Vascular Pharmacology. 46(4). 247–252. 5 indexed citations
15.
Ghatta, Srinivas, et al.. (2006). Sirolimus Causes Relaxation of Human Vascular Smooth Muscle: A Novel Action of Sirolimus Mediated via ATP-Sensitive Potassium Channels. Journal of Pharmacology and Experimental Therapeutics. 320(3). 1204–1208. 7 indexed citations
16.
Ghatta, Srinivas, et al.. (2006). Nitroglycerin-Induced Release of Calcitonin Gene-Related Peptide From Sensory Nerves Attenuates the Development of Nitrate Tolerance. Journal of Cardiovascular Pharmacology. 47(2). 175–181. 12 indexed citations
17.
Ghatta, Srinivas, et al.. (2005). Large-conductance, calcium-activated potassium channels: Structural and functional implications. Pharmacology & Therapeutics. 110(1). 103–116. 275 indexed citations
18.
Ghatta, Srinivas, et al.. (2005). A study on alpha-adrenoceptor mediated contractile responses of high fat diet fed rat thoracic aorta.. PubMed. 60(2). 142–6. 4 indexed citations
19.
Ghatta, Srinivas, et al.. (2004). CALCITONIN GENE-RELATED PEPTIDE: UNDERSTANDING ITS ROLE. Indian Journal of Pharmacology. 36(5). 277–283. 19 indexed citations
20.
Ghatta, Srinivas & Poduri Ramarao. (2004). Increased contractile responses to 5-hydroxytryptamine and Angiotensin II in high fat diet fed rat thoracic aorta. Lipids in Health and Disease. 3(1). 19–19. 19 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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