W. Douglas Cress

8.3k total citations · 2 hit papers
86 papers, 6.8k citations indexed

About

W. Douglas Cress is a scholar working on Oncology, Molecular Biology and Cancer Research. According to data from OpenAlex, W. Douglas Cress has authored 86 papers receiving a total of 6.8k indexed citations (citations by other indexed papers that have themselves been cited), including 59 papers in Oncology, 57 papers in Molecular Biology and 19 papers in Cancer Research. Recurrent topics in W. Douglas Cress's work include Cancer-related Molecular Pathways (39 papers), RNA modifications and cancer (18 papers) and Lung Cancer Treatments and Mutations (14 papers). W. Douglas Cress is often cited by papers focused on Cancer-related Molecular Pathways (39 papers), RNA modifications and cancer (18 papers) and Lung Cancer Treatments and Mutations (14 papers). W. Douglas Cress collaborates with scholars based in United States, Puerto Rico and Taiwan. W. Douglas Cress's co-authors include Joseph R. Nevins, Steven J. Triezenberg, D. Gale Johnson, Edward Seto, Yihong Ma, James K. Schwarz, Eric B. Haura, Matthew B. Schabath, Domenico Coppola and Jiandong Chen and has published in prestigious journals such as Nature, Science and Cell.

In The Last Decade

W. Douglas Cress

85 papers receiving 6.8k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Expression of transcription factor E2F1 induces quiescent cells to enter S phase

1993 845 citations W. Douglas Cress, Joseph R. Nevins et al. profile →
Histone deacetylases, transcriptional control, and cancer

2000 525 citations W. Douglas Cress et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
W. Douglas Cress United States	38	4.8k	2.8k	1.2k	825	775	86	6.8k
Peter M. Chumakov Russia	38	4.6k 1.0×	3.0k 1.1×	1.3k 1.1×	591 0.7×	887 1.1×	175	6.7k
Steven B. McMahon United States	42	6.5k 1.4×	2.2k 0.8×	1.9k 1.6×	711 0.9×	533 0.7×	77	8.1k
Gerardo Ferbeyre Canada	43	5.5k 1.2×	2.0k 0.7×	1.3k 1.1×	1.1k 1.3×	420 0.5×	122	7.8k
Akihiro Kurimasa Japan	36	4.8k 1.0×	1.6k 0.6×	1.2k 1.0×	399 0.5×	468 0.6×	90	6.3k
Jason D. Weber United States	39	6.2k 1.3×	3.9k 1.4×	1.4k 1.2×	1.6k 2.0×	566 0.7×	78	8.9k
Zhenkun Lou United States	53	6.8k 1.4×	2.8k 1.0×	1.4k 1.2×	989 1.2×	576 0.7×	134	9.0k
Asha S. Multani United States	40	5.0k 1.0×	3.3k 1.2×	1.9k 1.6×	803 1.0×	691 0.9×	126	8.2k
Katsutoshi Oda Japan	35	2.6k 0.5×	1.6k 0.6×	786 0.7×	703 0.9×	332 0.4×	195	4.9k
Srikumar Chellappan United States	46	5.5k 1.2×	3.4k 1.2×	1.1k 0.9×	643 0.8×	952 1.2×	108	7.9k
Samuel Benchimol Canada	46	5.7k 1.2×	4.6k 1.7×	1.4k 1.1×	671 0.8×	971 1.3×	94	8.8k

Countries citing papers authored by W. Douglas Cress

Since Specialization

Citations

This map shows the geographic impact of W. Douglas Cress's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by W. Douglas Cress with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites W. Douglas Cress more than expected).

Fields of papers citing papers by W. Douglas Cress

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by W. Douglas Cress. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by W. Douglas Cress. The network helps show where W. Douglas Cress may publish in the future.

Co-authorship network of co-authors of W. Douglas Cress

This figure shows the co-authorship network connecting the top 25 collaborators of W. Douglas Cress. A scholar is included among the top collaborators of W. Douglas Cress based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with W. Douglas Cress. W. Douglas Cress is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Gordián, Edna R., Marilin Rosa, Joseph Johnson, et al.. (2025). Creation and Characterization of a Breast Cancer Tissue Microarray Including Black and White Patients from Florida and Hispanic Patients from Puerto Rico and Florida. Cancer Research Communications. 5(5). 804–813.

Cress, W. Douglas, et al.. (2025). Profiling Sociodemographic Risk Factors and Clinical Outcomes of Women with Endometrial Cancer in Puerto Rico: The Central Role of Obesity and Obstetric Features. Journal of Racial and Ethnic Health Disparities. 13(1). 552–561. 1 indexed citations

Li, Jiannong, Edna R. Gordián, Steven A. Eschrich, et al.. (2024). Worse Clinical and Survival Outcomes in Breast Cancer Patients Living in Puerto Rico Compared to Hispanics, Non-Hispanic Blacks, and Non-Hispanic Whites from Florida. Journal of Racial and Ethnic Health Disparities. 13(1). 130–140. 1 indexed citations

Gordián, Edna R., Eric A. Welsh, Erin M. Siegel, et al.. (2019). Transforming growth factor β-induced epithelial-to-mesenchymal signature predicts metastasis-free survival in non-small cell lung cancer. Oncotarget. 10(8). 810–824. 27 indexed citations

Woods, Nicholas T., Gabriëla Wright, Lily L. Remsing Rix, et al.. (2016). PAXIP1 Potentiates the Combination of WEE1 Inhibitor AZD1775 and Platinum Agents in Lung Cancer. Molecular Cancer Therapeutics. 15(7). 1669–1681. 21 indexed citations

Chen, Dung‐Tsa, Po‐Yu Huang, Hui‐Yi Lin, et al.. (2016). Strategies for power calculations in predictive biomarker studies in survival data. Oncotarget. 7(49). 80373–80381. 2 indexed citations

Engel, Brienne E., et al.. (2015). The Retinoblastoma Tumor Suppressor Transcriptionally Represses Pak1 in Osteoblasts. PLoS ONE. 10(11). e0142406–e0142406. 4 indexed citations

Engel, Brienne E., et al.. (2013). Expression of integrin alpha 10 is transcriptionally activated by pRb in mouse osteoblasts and is downregulated in multiple solid tumors. Cell Death and Disease. 4(11). e938–e938. 17 indexed citations

Chen, Lu, Dung‐Tsa Chen, Bhupendra Rawal, et al.. (2012). Tripartite Motif Containing 28 (Trim28) Can Regulate Cell Proliferation by Bridging HDAC1/E2F Interactions. Journal of Biological Chemistry. 287(48). 40106–40118. 88 indexed citations

10.

Corsino, Patrick, Nicole A. Horenstein, David A. Ostrov, et al.. (2009). A Novel Class of Cyclin-dependent Kinase Inhibitors Identified by Molecular Docking Act through a Unique Mechanism. Journal of Biological Chemistry. 284(43). 29945–29955. 15 indexed citations

11.

Ma, Yihong, Sandhya Boyapalle, Shen‐Shu Sung, et al.. (2008). A Small-Molecule E2F Inhibitor Blocks Growth in a Melanoma Culture Model. Cancer Research. 68(15). 6292–6299. 80 indexed citations

12.

Niu, Guilian, Jiehui Deng, Yihong Ma, et al.. (2008). Signal Transducer and Activator of Transcription 3 Is Required for Hypoxia-Inducible Factor-1α RNA Expression in Both Tumor Cells and Tumor-Associated Myeloid Cells. Molecular Cancer Research. 6(7). 1099–1105. 151 indexed citations

13.

Wang, Chuangui, Xinghua Hou, Subhra Mohapatra, et al.. (2005). Activation of p27Kip1 Expression by E2F1. Journal of Biological Chemistry. 280(13). 12339–12343. 67 indexed citations

14.

Song, Lanxi, et al.. (2005). Mcl-1 regulates survival and sensitivity to diverse apoptotic stimuli in human non-small cell lung cancer cells. Cancer Biology & Therapy. 4(3). 267–276. 181 indexed citations

15.

Ma, Yihong, W. Douglas Cress, & Eric B. Haura. (2003). Flavopiridol-induced apoptosis is mediated through up-regulation of E2F1 and repression of Mcl-1.. PubMed. 2(1). 73–81. 91 indexed citations

16.

Ma, Yihong, et al.. (2002). Identification of Novel E2F1-Regulated Genes by Microarray. Archives of Biochemistry and Biophysics. 399(2). 212–224. 91 indexed citations

17.

Cuenca, Alex G., et al.. (2001). Calorie Restriction Influences Cell Cycle Protein Expression and DNA Synthesis during Liver Regeneration. Experimental Biology and Medicine. 226(11). 1061–1067. 15 indexed citations

18.

Tao, Yunxia, et al.. (1997). Subunit Composition Determines E2F DNA-Binding Site Specificity. Molecular and Cellular Biology. 17(12). 6994–7007. 116 indexed citations

19.

Cress, W. Douglas & Joseph R. Nevins. (1996). A Role for a Bent DNA Structure in E2F-Mediated Transcription Activation. Molecular and Cellular Biology. 16(5). 2119–2127. 58 indexed citations

20.

Foster, Joyce G., W. Douglas Cress, Sara F. Wright, & John L. Hess. (1987). Intracellular Localization of the Neurotoxin 2,4-Diaminobutyric Acid in Lathyrus sylvestris L. Leaf Tissue. PLANT PHYSIOLOGY. 83(4). 900–904. 13 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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