David Pei‐Cheng Lin
- Molecular Biology top 0.5%
- Oncology top 0.2%
- Cancer Research top 1%
- Biotechnology top 0.2%
- Cell Biology top 2%
- Co-authors
- W E MercerDaniel B. LevyBert VogelsteinRamon ParsonsJ.M. TrentK W KinzlerVictor E. VelculescuTakashi Tokino
- Topics
- Ocular Surface and Contact Lens (9 papers)Cancer-related Molecular Pathways (7 papers)Epigenetics and DNA Methylation (7 papers)
- Cited by
- OncologyBiotechnologyCancer Research
- Partner nations
- TaiwanUnited StatesJapan
In The Last Decade
David Pei‐Cheng Lin
55 papers receiving 9.4k citations
Hit Papers
Peers
Comparison fields: 5 of 124
- Molecular Biology 6.7k
- Oncology 6.2k
- Cancer Research 1.6k
- Biotechnology 1.3k
- Cell Biology 739
Countries citing papers authored by David Pei‐Cheng Lin
This map shows the geographic impact of David Pei‐Cheng Lin's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by David Pei‐Cheng Lin with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites David Pei‐Cheng Lin more than expected).
Fields of papers citing papers by David Pei‐Cheng Lin
This network shows the impact of papers produced by David Pei‐Cheng Lin. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by David Pei‐Cheng Lin. The network helps show where David Pei‐Cheng Lin may publish in the future.
Co-authorship network of co-authors of David Pei‐Cheng Lin
This figure shows the co-authorship network connecting the top 25 collaborators of David Pei‐Cheng Lin. A scholar is included among the top collaborators of David Pei‐Cheng Lin based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with David Pei‐Cheng Lin. David Pei‐Cheng Lin is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 0 | |
| 2 | 3 | |
| 3 | 0 | |
| 4 | 1 | |
| 5 | 3 | |
| 6 | 2 | |
| 7 | 3 | |
| 8 | 1 | |
| 9 | 4 | |
| 10 | 11 | |
| 11 | 72 | |
| 12 | 177 | |
| 13 | 17 | |
| 14 | 1 | |
| 15 | 2 | |
| 16 | 7 | |
| 17 | 15 | |
| 18 | 5 | |
| 19 | 46 | |
| 20 | WAF1, a potential mediator of p53 tumor suppressionbreakdown → | 7234 |
About David Pei‐Cheng Lin
David Pei‐Cheng Lin is a scholar working on Nephrology, General Dentistry and Public Health, Environmental and Occupational Health, having authored 59 papers that have together received 9.5k indexed citations. Recurring topics across this work include Ocular Surface and Contact Lens (9 papers), Cancer-related Molecular Pathways (7 papers) and Epigenetics and DNA Methylation (7 papers). The work is most often cited by research in Oncology (6.2k citations), Biotechnology (1.3k citations) and Cancer Research (1.6k citations). David Pei‐Cheng Lin has collaborated with scholars based in Taiwan, United States and Japan. Frequent co-authors include W E Mercer, Daniel B. Levy, Bert Vogelstein, Ramon Parsons, J.M. Trent, K W Kinzler, Victor E. Velculescu, Takashi Tokino, Wafik S. El‐Deiry and Márcio F. Chedid. Their work appears in journals such as Cell, Proceedings of the National Academy of Sciences and Journal of Clinical Oncology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.