Soichiro Ikeda
- Molecular Biology
- Pulmonary and Respiratory Medicine top 10%
- Cardiology and Cardiovascular Medicine top 10%
- Cancer Research top 10%
- Oncology
- Co-authors
- Hiroyuki TsutsuiKosuke OkabeMasataka IkedaAkihito IshikitaShouji MatsushimaTomomi IdeTomonori TadokoroHiroko Deguchi
- Topics
- Mitochondrial Function and Pathology (3 papers)Cardiac Fibrosis and Remodeling (3 papers)Chemotherapy-induced cardiotoxicity and mitigation (3 papers)
- Partner nations
- JapanUnited StatesHong Kong
In The Last Decade
Soichiro Ikeda
24 papers receiving 1.0k citations
Hit Papers
Peers
Comparison fields: 5 of 92
- Molecular Biology 660
- Pulmonary and Respiratory Medicine 322
- Cardiology and Cardiovascular Medicine 255
- Cancer Research 214
- Oncology 187
Countries citing papers authored by Soichiro Ikeda
This map shows the geographic impact of Soichiro Ikeda's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Soichiro Ikeda with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Soichiro Ikeda more than expected).
Fields of papers citing papers by Soichiro Ikeda
This network shows the impact of papers produced by Soichiro Ikeda. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Soichiro Ikeda. The network helps show where Soichiro Ikeda may publish in the future.
Co-authorship network of co-authors of Soichiro Ikeda
This figure shows the co-authorship network connecting the top 25 collaborators of Soichiro Ikeda. A scholar is included among the top collaborators of Soichiro Ikeda based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Soichiro Ikeda. Soichiro Ikeda is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 1 | |
| 2 | 0 | |
| 3 | 0 | |
| 4 | 61 | |
| 5 | 3 | |
| 6 | 14 | |
| 7 | 26 | |
| 8 | 18 | |
| 9 | 13 | |
| 10 | Mitochondria-dependent ferroptosis plays a pivotal role in doxorubicin cardiotoxicitybreakdown → | 487 |
| 11 | 43 | |
| 12 | 0 | |
| 13 | 11 | |
| 14 | 22 | |
| 15 | 2 | |
| 16 | 11 | |
| 17 | 9 | |
| 18 | 62 | |
| 19 | 28 | |
| 20 | 153 |
About Soichiro Ikeda
Soichiro Ikeda is a scholar working on Aging, Cardiology and Cardiovascular Medicine and Physiology, having authored 27 papers that have together received 1.1k indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (3 papers), Cardiac Fibrosis and Remodeling (3 papers) and Chemotherapy-induced cardiotoxicity and mitigation (3 papers). The work is most often cited by research in Cancer Research (214 citations), Cardiology and Cardiovascular Medicine (255 citations) and Pulmonary and Respiratory Medicine (322 citations). Soichiro Ikeda has collaborated with scholars based in Japan, United States and Hong Kong. Frequent co-authors include Hiroyuki Tsutsui, Kosuke Okabe, Masataka Ikeda, Akihito Ishikita, Shouji Matsushima, Tomomi Ide, Tomonori Tadokoro, Hiroko Deguchi, Hirotaka Imai and Ken‐ichi Yamada. Their work appears in journals such as Journal of Biological Chemistry, Circulation Research and Scientific Reports.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.