Simon N. Willis

9.4k total citations · 5 hit papers
36 papers, 7.2k citations indexed

About

Simon N. Willis is a scholar working on Immunology, Molecular Biology and Oncology. According to data from OpenAlex, Simon N. Willis has authored 36 papers receiving a total of 7.2k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Immunology, 14 papers in Molecular Biology and 9 papers in Oncology. Recurrent topics in Simon N. Willis's work include Immune Cell Function and Interaction (12 papers), T-cell and B-cell Immunology (12 papers) and Immunotherapy and Immune Responses (10 papers). Simon N. Willis is often cited by papers focused on Immune Cell Function and Interaction (12 papers), T-cell and B-cell Immunology (12 papers) and Immunotherapy and Immune Responses (10 papers). Simon N. Willis collaborates with scholars based in Australia, United States and New Zealand. Simon N. Willis's co-authors include Jerry M. Adams, David C.S. Huang, Andrew H. Wei, Jamie I. Fletcher, Catherine L. Day, Lin Chen, Mark G. Hinds, Peter M. Colman, Mark F. van Delft and Peter E. Czabotar and has published in prestigious journals such as Science, Nucleic Acids Research and Nature Communications.

In The Last Decade

Simon N. Willis

36 papers receiving 7.1k citations

Hit Papers

5 papers align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Differential Targeting of Prosurvival Bcl-2 Proteins by Their BH3-Only Ligands Allows Complementary Apoptotic Function

2005 1.5k citations Simon N. Willis, Andrew H. Wei et al. profile →
Proapoptotic Bak is sequestered by Mcl-1 and Bcl-x _L , but not Bcl-2, until displaced by BH3-only proteins

2005 1.0k citations Simon N. Willis, Chen Lin et al. Genes & Development profile →
The BH3 mimetic ABT-737 targets selective Bcl-2 proteins and efficiently induces apoptosis via Bak/Bax if Mcl-1 is neutralized

2006 1.0k citations Mark F. van Delft, Andrew H. Wei et al. Cancer Cell profile →
Apoptosis Initiated When BH3 Ligands Engage Multiple Bcl-2 Homologs, Not Bax or Bak

2007 883 citations Simon N. Willis, Jamie I. Fletcher et al. profile →
Life in the balance: how BH3-only proteins induce apoptosis

2005 612 citations Simon N. Willis, Jerry M. Adams profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Simon N. Willis Australia	24	4.9k	2.0k	1.7k	922	690	36	7.2k
Grazia Ambrosini United States	27	4.8k 1.0×	1.2k 0.6×	2.2k 1.3×	414 0.4×	496 0.7×	48	6.8k
Domenico Delia Italy	43	4.2k 0.9×	950 0.5×	2.5k 1.5×	673 0.7×	379 0.5×	125	6.9k
Elaine M. Hurt United States	35	3.1k 0.6×	2.0k 1.0×	2.4k 1.4×	457 0.5×	410 0.6×	59	6.3k
Christine M. Eischen United States	44	5.0k 1.0×	1.1k 0.5×	3.1k 1.8×	643 0.7×	356 0.5×	99	6.8k
David T. Weaver United States	49	6.5k 1.3×	1.4k 0.7×	2.6k 1.5×	462 0.5×	384 0.6×	150	9.0k
Jason D. Weber United States	39	6.2k 1.3×	1.6k 0.8×	3.9k 2.2×	397 0.4×	623 0.9×	78	8.9k
Nils Holler Switzerland	20	5.0k 1.0×	3.9k 1.9×	1.1k 0.7×	632 0.7×	917 1.3×	20	8.3k
Arthur L. Shaffer United States	36	3.2k 0.6×	4.0k 2.0×	1.9k 1.1×	1.6k 1.8×	615 0.9×	55	7.9k
Andreas Villunger Austria	55	7.5k 1.5×	2.8k 1.4×	3.1k 1.8×	595 0.6×	1.0k 1.5×	183	10.7k
Dirk Eick Germany	57	8.9k 1.8×	968 0.5×	2.6k 1.5×	689 0.7×	500 0.7×	131	10.8k

Countries citing papers authored by Simon N. Willis

Since Specialization

Citations

This map shows the geographic impact of Simon N. Willis's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Simon N. Willis with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Simon N. Willis more than expected).

Fields of papers citing papers by Simon N. Willis

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Simon N. Willis. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Simon N. Willis. The network helps show where Simon N. Willis may publish in the future.

Co-authorship network of co-authors of Simon N. Willis

This figure shows the co-authorship network connecting the top 25 collaborators of Simon N. Willis. A scholar is included among the top collaborators of Simon N. Willis based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Simon N. Willis. Simon N. Willis is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Bergamasco, Maria, Waruni Abeysekera, Connie S.N. Li Wai Suen, et al.. (2024). The histone acetyltransferase KAT6B is required for hematopoietic stem cell development and function. Stem Cell Reports. 19(4). 469–485. 8 indexed citations

Kong, Isabella Y., et al.. (2023). An arrayed CRISPR screen of primary B cells reveals the essential elements of the antibody secretion pathway. Frontiers in Immunology. 14. 1089243–1089243. 6 indexed citations

Fedele, Pasquale L., Yang Liao, Jianan Gong, et al.. (2020). The transcription factor IRF4 represses proapoptotic BMF and BIM to licence multiple myeloma survival. Leukemia. 35(7). 2114–2118. 21 indexed citations

Willis, Simon N. & Stephen L. Nutt. (2019). New players in the gene regulatory network controlling late B cell differentiation. Current Opinion in Immunology. 58. 68–74. 17 indexed citations

Willis, Simon N., Julie Tellier, Yang Liao, et al.. (2017). Environmental sensing by mature B cells is controlled by the transcription factors PU.1 and SpiB. Nature Communications. 8(1). 1426–1426. 71 indexed citations

Narayan, Nisha, Belinda Phipson, Simon N. Willis, et al.. (2016). Functionally distinct roles for different miR-155 expression levels through contrasting effects on gene expression, in acute myeloid leukaemia. Leukemia. 31(4). 808–820. 44 indexed citations

Willis, Simon N., et al.. (2015). Investigating the Antigen Specificity of Multiple Sclerosis Central Nervous System-Derived Immunoglobulins. Frontiers in Immunology. 6. 600–600. 33 indexed citations

Grigoriadis, George, Lorraine A. O’Reilly, Simon N. Willis, et al.. (2014). 65. Cytokine. 70(1). 43–43. 1 indexed citations

Amato, Anthony A., Elizabeth M. Bradshaw, Kevin J. Felice, et al.. (2012). Autoantibodies Produced at the Site of Tissue Damage Provide Evidence of Humoral Autoimmunity in Inclusion Body Myositis. PLoS ONE. 7(10). e46709–e46709. 19 indexed citations

10.

Obermeier, Birgit, Laura Lovato, Reinhard Mentele, et al.. (2011). Related B cell clones that populate the CSF and CNS of patients with multiple sclerosis produce CSF immunoglobulin. Journal of Neuroimmunology. 233(1-2). 245–248. 107 indexed citations

11.

Lovato, Laura, Simon N. Willis, Scott J. Rodig, et al.. (2011). Related B cell clones populate the meninges and parenchyma of patients with multiple sclerosis. Brain. 134(2). 534–541. 167 indexed citations

12.

Ligocki, Ann J., Laura Lovato, Richard H. Scheuermann, et al.. (2010). A unique antibody gene signature is prevalent in the central nervous system of patients with multiple sclerosis. Journal of Neuroimmunology. 226(1-2). 192–193. 15 indexed citations

13.

Kvansakul, Marc, Andrew H. Wei, Jamie I. Fletcher, et al.. (2010). Structural Basis for Apoptosis Inhibition by Epstein-Barr Virus BHRF1. PLoS Pathogens. 6(12). e1001236–e1001236. 93 indexed citations

14.

Willis, Simon N., Scott Mallozzi, Scott J. Rodig, et al.. (2009). The Microenvironment of Germ Cell Tumors Harbors a Prominent Antigen-Driven Humoral Response. The Journal of Immunology. 182(5). 3310–3317. 53 indexed citations

15.

Huntington, Nicholas D., Hamsa Puthalakath, Edwina Naik, et al.. (2007). Interleukin 15–mediated survival of natural killer cells is determined by interactions among Bim, Noxa and Mcl-1. Nature Immunology. 8(8). 856–863. 216 indexed citations

16.

Delft, Mark F. van, Andrew H. Wei, Kylie D. Mason, et al.. (2006). The BH3 mimetic ABT-737 targets selective Bcl-2 proteins and efficiently induces apoptosis via Bak/Bax if Mcl-1 is neutralized. Cancer Cell. 10(5). 389–399. 1008 indexed citations breakdown →

17.

Willis, Simon N., Chen Lin, Grant Dewson, et al.. (2005). Proapoptotic Bak is sequestered by Mcl-1 and Bcl-x _L , but not Bcl-2, until displaced by BH3-only proteins. Genes & Development. 19(11). 1294–1305. 1024 indexed citations breakdown →

18.

Huntington, Nicholas D., Yuekang Xu, Hamsa Puthalakath, et al.. (2005). CD45 links the B cell receptor with cell survival and is required for the persistence of germinal centers. Nature Immunology. 7(2). 190–198. 65 indexed citations

19.

Willis, Simon N., Anne‐Marie Hutchins, Fleur Hammet, et al.. (2003). Detailed gene copy number and RNA expression analysis of the 17q12–23 region in primary breast cancers. Genes Chromosomes and Cancer. 36(4). 382–392. 22 indexed citations

20.

Willis, Simon N.. (1996). Pituitary-specific chromatin structure of the rat prolactin distal enhancer element. Nucleic Acids Research. 24(6). 1065–1072. 12 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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