Silke Metzger

801 total citations
12 papers, 534 citations indexed

About

Silke Metzger is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Epidemiology. According to data from OpenAlex, Silke Metzger has authored 12 papers receiving a total of 534 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Cellular and Molecular Neuroscience, 6 papers in Molecular Biology and 6 papers in Epidemiology. Recurrent topics in Silke Metzger's work include Genetic Neurodegenerative Diseases (7 papers), Mitochondrial Function and Pathology (5 papers) and Autophagy in Disease and Therapy (3 papers). Silke Metzger is often cited by papers focused on Genetic Neurodegenerative Diseases (7 papers), Mitochondrial Function and Pathology (5 papers) and Autophagy in Disease and Therapy (3 papers). Silke Metzger collaborates with scholars based in Germany, United States and Netherlands. Silke Metzger's co-authors include Olaf Rieß, Huu Phuc Nguyen, Wilfried Schippert, Claus Garbe, Matthias Moehrle, G. Rassner, Helmut Breuninger, Laura E. Clemens, Liang Tong and Petra Fallier‐Becker and has published in prestigious journals such as Journal of Neuroscience, PLoS ONE and Neuropharmacology.

In The Last Decade

Silke Metzger

12 papers receiving 524 citations

Peers

Silke Metzger
Sara Matar United States
Kelly J. Conn United States
Michael Wooten United States
Yang Tian China
Xiaotong Cheng United Kingdom
Sara Matar United States
Silke Metzger
Citations per year, relative to Silke Metzger Silke Metzger (= 1×) peers Sara Matar

Countries citing papers authored by Silke Metzger

Since Specialization
Citations

This map shows the geographic impact of Silke Metzger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Silke Metzger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Silke Metzger more than expected).

Fields of papers citing papers by Silke Metzger

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Silke Metzger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Silke Metzger. The network helps show where Silke Metzger may publish in the future.

Co-authorship network of co-authors of Silke Metzger

This figure shows the co-authorship network connecting the top 25 collaborators of Silke Metzger. A scholar is included among the top collaborators of Silke Metzger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Silke Metzger. Silke Metzger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

12 of 12 papers shown
1.
Walter, Carolin, Laura E. Clemens, Amelie J. Müller, et al.. (2016). Activation of AMPK-induced autophagy ameliorates Huntington disease pathology in vitro. Neuropharmacology. 108. 24–38. 64 indexed citations
2.
Metzger, Silke, Olaf Rieß, Raymund A.C. Roos, et al.. (2013). The V471A Polymorphism in Autophagy-Related Gene ATG7 Modifies Age at Onset Specifically in Italian Huntington Disease Patients. PLoS ONE. 8(7). e68951–e68951. 41 indexed citations
3.
Yu-Taeger, Libo, Elisabeth Petrasch‐Parwez, Alexander P. Osmand, et al.. (2012). A Novel BACHD Transgenic Rat Exhibits Characteristic Neuropathological Features of Huntington Disease. Journal of Neuroscience. 32(44). 15426–15438. 78 indexed citations
4.
Tsai, Yu-Chun, Silke Metzger, Olaf Rieß, Anne S. Soehn, & Huu Phuc Nguyen. (2012). Genetic analysis of polymorphisms in the kalirin gene for association with age-at-onset in European Huntington disease patients. BMC Medical Genetics. 13(1). 48–48. 8 indexed citations
5.
Metzger, Silke, et al.. (2011). Localization of sequence variations in PGC-1α influence their modifying effect in Huntington disease. Molecular Neurodegeneration. 6(1). 1–1. 113 indexed citations
6.
Metzger, Silke, et al.. (2010). Age at onset in Huntington’s disease is modified by the autophagy pathway: implication of the V471A polymorphism in Atg7. Human Genetics. 128(4). 453–459. 76 indexed citations
7.
Nguyen, Huu Phuc, Silke Metzger, Carsten Holzmann, et al.. (2008). Age‐dependent gene expression profile and protein expression in a transgenic rat model of Huntington's disease. PROTEOMICS - CLINICAL APPLICATIONS. 2(12). 1638–1650. 13 indexed citations
8.
Moehrle, Matthias, Silke Metzger, Wilfried Schippert, et al.. (2003). "Functional" Surgery in Subungual Melanoma. Dermatologic Surgery. 29(4). 366–374. 108 indexed citations
9.
Moehrle, Matthias, Silke Metzger, Wilfried Schippert, et al.. (2003). “Functional” Surgery in Subungual Melanoma. Dermatologic Surgery. 29(4). 366–374. 9 indexed citations
10.
Willinger, Birgit, Elisabeth Engelmann, Heidelore Hofmann, et al.. (2002). Multicenter comparison of Fungitest® for susceptibility testing of Candida species. Diagnostic Microbiology and Infectious Disease. 44(3). 253–257. 5 indexed citations
11.
Metzger, Silke, et al.. (2001). Aleukemic Leukemia Cutis Presenting as Benign-appearing Exanthema. Acta Dermato Venereologica. 81(1). 45–47. 8 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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