Libo Yu-Taeger

764 total citations
27 papers, 590 citations indexed

About

Libo Yu-Taeger is a scholar working on Cellular and Molecular Neuroscience, Molecular Biology and Neurology. According to data from OpenAlex, Libo Yu-Taeger has authored 27 papers receiving a total of 590 indexed citations (citations by other indexed papers that have themselves been cited), including 24 papers in Cellular and Molecular Neuroscience, 19 papers in Molecular Biology and 13 papers in Neurology. Recurrent topics in Libo Yu-Taeger's work include Genetic Neurodegenerative Diseases (22 papers), Mitochondrial Function and Pathology (15 papers) and Neurological disorders and treatments (11 papers). Libo Yu-Taeger is often cited by papers focused on Genetic Neurodegenerative Diseases (22 papers), Mitochondrial Function and Pathology (15 papers) and Neurological disorders and treatments (11 papers). Libo Yu-Taeger collaborates with scholars based in Germany, United States and Italy. Libo Yu-Taeger's co-authors include Huu Phuc Nguyen, Olaf Rieß, Arianna Novati, Laura E. Clemens, Bernd J. Pichler, Carsten Calaminus, Thierry Bordet, Rebecca M. Pruss, Elisabeth Petrasch‐Parwez and Antonio Pisani and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Neuroscience and PLoS ONE.

In The Last Decade

Libo Yu-Taeger

26 papers receiving 582 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Libo Yu-Taeger Germany 13 436 348 230 34 32 27 590
Lena E. Hjermind Denmark 18 403 0.9× 405 1.2× 388 1.7× 31 0.9× 119 3.7× 47 904
E. Mancinelli Italy 17 585 1.3× 651 1.9× 131 0.6× 55 1.6× 43 1.3× 28 807
Jesús F. Torres-Peraza Spain 13 543 1.2× 429 1.2× 189 0.8× 61 1.8× 44 1.4× 14 737
Georgia Kouroupi Greece 12 212 0.5× 358 1.0× 132 0.6× 48 1.4× 61 1.9× 16 613
Laetitia Francelle France 11 206 0.5× 291 0.8× 131 0.6× 26 0.8× 82 2.6× 14 545
Mary Y. Heng United States 10 439 1.0× 499 1.4× 165 0.7× 48 1.4× 42 1.3× 11 685
Patrizia Giallonardo Italy 6 263 0.6× 233 0.7× 122 0.5× 44 1.3× 23 0.7× 7 396
Jayalakshmi Srinidhi Mysore United States 13 594 1.4× 583 1.7× 227 1.0× 13 0.4× 24 0.8× 17 704
Alex Osmand United States 6 684 1.6× 669 1.9× 224 1.0× 72 2.1× 41 1.3× 7 792
Valerio Castoldi Italy 12 168 0.4× 332 1.0× 58 0.3× 41 1.2× 48 1.5× 26 586

Countries citing papers authored by Libo Yu-Taeger

Since Specialization
Citations

This map shows the geographic impact of Libo Yu-Taeger's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Libo Yu-Taeger with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Libo Yu-Taeger more than expected).

Fields of papers citing papers by Libo Yu-Taeger

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Libo Yu-Taeger. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Libo Yu-Taeger. The network helps show where Libo Yu-Taeger may publish in the future.

Co-authorship network of co-authors of Libo Yu-Taeger

This figure shows the co-authorship network connecting the top 25 collaborators of Libo Yu-Taeger. A scholar is included among the top collaborators of Libo Yu-Taeger based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Libo Yu-Taeger. Libo Yu-Taeger is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
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Cheng, Fubo, Chang Liu, Peter A. Barbuti, et al.. (2022). Intronic enhancers of the human SNCA gene predominantly regulate its expression in brain in vivo. Science Advances. 8(47). eabq6324–eabq6324. 6 indexed citations
4.
Yu-Taeger, Libo, Arianna Novati, Jonasz Jeremiasz Weber, et al.. (2022). Evidences for Mutant Huntingtin Inducing Musculoskeletal and Brain Growth Impairments via Disturbing Testosterone Biosynthesis in Male Huntington Disease Animals. Cells. 11(23). 3779–3779. 4 indexed citations
5.
Cheng, Fubo, Peter A. Barbuti, Paola Bonsi, et al.. (2022). DYT6 mutated THAP1 is a cell type dependent regulator of the SP1 family. Brain. 145(11). 3968–3984. 6 indexed citations
6.
Martella, Giuseppina, Paola Bonsi, Paola Imbriani, et al.. (2021). Rescue of striatal long-term depression by chronic mGlu5 receptor negative allosteric modulation in distinct dystonia models. Neuropharmacology. 192. 108608–108608. 12 indexed citations
7.
Cohen-Rosenzweig, Chen, Libo Yu-Taeger, Tamar Ziv, et al.. (2020). Site-specific ubiquitination of pathogenic huntingtin attenuates its deleterious effects. Proceedings of the National Academy of Sciences. 117(31). 18661–18669. 18 indexed citations
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Yu-Taeger, Libo, et al.. (2019). Olfactory bulb atrophy and caspase activation observed in the BACHD rat models of Huntington disease. Neurobiology of Disease. 125. 219–231. 10 indexed citations
10.
Yu-Taeger, Libo, Thomas Ott, Paola Bonsi, et al.. (2019). Impaired dopamine- and adenosine-mediated signaling and plasticity in a novel rodent model for DYT25 dystonia. Neurobiology of Disease. 134. 104634–104634. 21 indexed citations
11.
Novati, Arianna, et al.. (2018). Sexual behavior and testis morphology in the BACHD rat model. PLoS ONE. 13(6). e0198338–e0198338. 8 indexed citations
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Yu-Taeger, Libo, Nicolas Casadei, Michael Walter, et al.. (2018). Dynamic nuclear envelope phenotype in rats overexpressing mutated human torsinA protein. Biology Open. 7(7). 2 indexed citations
13.
Novati, Arianna, Thomas Hentrich, Zinah Wassouf, et al.. (2018). Environment-dependent striatal gene expression in the BACHD rat model for Huntington disease. Scientific Reports. 8(1). 5803–5803. 10 indexed citations
14.
Novati, Arianna, et al.. (2018). Genetic Rodent Models of Huntington Disease. Advances in experimental medicine and biology. 1049. 29–57. 20 indexed citations
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Massioui, Nicole El, Charlotte Lamirault, Alexis Maillard, et al.. (2016). Impaired Decision Making and Loss of Inhibitory-Control in a Rat Model of Huntington Disease. Frontiers in Behavioral Neuroscience. 10. 204–204. 16 indexed citations
17.
Yu-Taeger, Libo, Bruno Bozon, Olaf Rieß, et al.. (2015). Reduced impact of emotion on choice behavior in presymptomatic BACHD rats, a transgenic rodent model for Huntington Disease. Neurobiology of Learning and Memory. 125. 249–257. 12 indexed citations
18.
Clemens, Laura E., Schamim H. Eckert, Stephanie Hagl, et al.. (2014). Mitochondrial Membrane Fluidity is Consistently Increased in Different Models of Huntington Disease: Restorative Effects of Olesoxime. Molecular Neurobiology. 50(1). 107–118. 40 indexed citations
19.
Koeppen, Arnulf H., Elisabeth Petrasch‐Parwez, Carsten Calaminus, et al.. (2013). A Novel Transgenic Rat Model for Spinocerebellar Ataxia Type 17 Recapitulates Neuropathological Changes and SuppliesIn VivoImaging Biomarkers. Journal of Neuroscience. 33(21). 9068–9081. 35 indexed citations
20.
Yu-Taeger, Libo, Elisabeth Petrasch‐Parwez, Alexander P. Osmand, et al.. (2012). A Novel BACHD Transgenic Rat Exhibits Characteristic Neuropathological Features of Huntington Disease. Journal of Neuroscience. 32(44). 15426–15438. 78 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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