Joseph C. Koster

3.2k total citations
44 papers, 2.5k citations indexed

About

Joseph C. Koster is a scholar working on Molecular Biology, Surgery and Pathology and Forensic Medicine. According to data from OpenAlex, Joseph C. Koster has authored 44 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 25 papers in Molecular Biology, 18 papers in Surgery and 16 papers in Pathology and Forensic Medicine. Recurrent topics in Joseph C. Koster's work include Pancreatic function and diabetes (18 papers), Cardiac Ischemia and Reperfusion (15 papers) and Ion channel regulation and function (12 papers). Joseph C. Koster is often cited by papers focused on Pancreatic function and diabetes (18 papers), Cardiac Ischemia and Reperfusion (15 papers) and Ion channel regulation and function (12 papers). Joseph C. Koster collaborates with scholars based in United States, Italy and Germany. Joseph C. Koster's co-authors include Colin G. Nichols, Marı́a S. Remedi, Thomas P. Flagg, Decha Enkvetchakul, Bess A. Marshall, Qun Sha, John A. Corbett, Gustavo Blanco, Robert W. Mercer and M. Alan Permutt and has published in prestigious journals such as Cell, Proceedings of the National Academy of Sciences and Nucleic Acids Research.

In The Last Decade

Joseph C. Koster

44 papers receiving 2.4k citations

Peers

Joseph C. Koster

SURGE

PFM

EDM

CCM

Marı́a S. Remedi United States

Christophe Depré United States

B Ošťádal Czechia

Gary V. Désir United States

Bernice Marcus‐Samuels United States

W. Keith Jones United States

K Rakusan Canada

Peter Razeghi United States

P. Darwin Bell United States

Melissa E. Reichelt Australia

Marı́a S. Remedi United States

Joseph C. Koster

1.2k ×1.0

1.4k ×1.3

SURGE

375 ×0.5

PFM

813 ×1.3

EDM

215 ×0.5

CCM

Citations per year, relative to Joseph C. Koster Joseph C. Koster (= 1×) peers Marı́a S. Remedi

Countries citing papers authored by Joseph C. Koster

Since Specialization

Citations

This map shows the geographic impact of Joseph C. Koster's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Joseph C. Koster with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Joseph C. Koster more than expected).

Fields of papers citing papers by Joseph C. Koster

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Joseph C. Koster. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Joseph C. Koster. The network helps show where Joseph C. Koster may publish in the future.

Co-authorship network of co-authors of Joseph C. Koster

This figure shows the co-authorship network connecting the top 25 collaborators of Joseph C. Koster. A scholar is included among the top collaborators of Joseph C. Koster based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Joseph C. Koster. Joseph C. Koster is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Aw, Sherry, Joseph C. Koster, Wade L. Pearson, et al.. (2010). The ATP-sensitive K+-channel (KATP) controls early left–right patterning in Xenopus and chick embryos. Developmental Biology. 346(1). 39–53. 48 indexed citations

Vyas, Arpita, Joseph C. Koster, Anatoly Tzekov, & Paul W. Hruz. (2010). Effects of the HIV Protease Inhibitor Ritonavir on GLUT4 Knock-out Mice. Journal of Biological Chemistry. 285(47). 36395–36400. 50 indexed citations

Remedi, Marı́a S. & Joseph C. Koster. (2009). KATP channelopathies in the pancreas. Pflügers Archiv - European Journal of Physiology. 460(2). 307–320. 29 indexed citations

Remedi, Marı́a S., Harley T. Kurata, F. Thomas Wunderlich, et al.. (2009). Secondary Consequences of β Cell Inexcitability: Identification and Prevention in a Murine Model of KATP-Induced Neonatal Diabetes Mellitus. Cell Metabolism. 9(2). 140–151. 80 indexed citations

Nichols, Colin G., Joseph C. Koster, & Marı́a S. Remedi. (2007). β‐cell hyperexcitability: from hyperinsulinism to diabetes. Diabetes Obesity and Metabolism. 9(s2). 81–88. 30 indexed citations

Koster, Joseph C., Francesco Cadario, Cinzia Peruzzi, et al.. (2007). The G53D Mutation in Kir6.2 (KCNJ11) Is Associated with Neonatal Diabetes and Motor Dysfunction in Adulthood that Is Improved with Sulfonylurea Therapy. The Journal of Clinical Endocrinology & Metabolism. 93(3). 1054–1061. 81 indexed citations

Remedi, Marı́a S., Jonathan V. Rocheleau, Michael L. McDaniel, et al.. (2006). Hyperinsulinism in mice with heterozygous loss of KATP channels. Diabetologia. 49(10). 2368–2378. 49 indexed citations

Rocheleau, Jonathan V., Marı́a S. Remedi, W. Steven Head, et al.. (2006). Critical Role of Gap Junction Coupled KATP Channel Activity for Regulated Insulin Secretion. PLoS Biology. 4(2). e26–e26. 105 indexed citations

Flagg, Thomas P., Flavien Charpentier, Marı́a S. Remedi, et al.. (2004). Remodeling of excitation-contraction coupling in transgenic mice expressing ATP-insensitive sarcolemmal K_ATP channels. American Journal of Physiology-Heart and Circulatory Physiology. 286(4). H1361–H1369. 40 indexed citations

10.

Koster, Joseph C., et al.. (2000). Targeted Overactivity of β Cell KATP Channels Induces Profound Neonatal Diabetes. Cell. 100(6). 645–654. 235 indexed citations

11.

Koster, Joseph C., Qun Sha, Show‐Ling Shyng, & Colin G. Nichols. (1999). ATP inhibition of K_ATP channels: control of nucleotide sensitivity by the N‐terminal domain of the Kir6.2 subunit. The Journal of Physiology. 515(1). 19–30. 82 indexed citations

12.

Koster, Joseph C., et al.. (1998). Assembly of ROMK1 (Kir 1.1a) Inward Rectifier K+ Channel Subunits Involves Multiple Interaction Sites. Biophysical Journal. 74(4). 1821–1829. 33 indexed citations

13.

Blanco, Gustavo, et al.. (1997). Studies of Na, K‐ATPase Structure and Function Using Baculovirus. Annals of the New York Academy of Sciences. 834(1). 88–96. 5 indexed citations

14.

Koster, Joseph C., et al.. (1997). Characterization of Na,K‐ATPase α/α Oligomerization. Annals of the New York Academy of Sciences. 834(1). 135–138. 3 indexed citations

15.

Koster, Joseph C., W. H. L. Hackeng, & Hindrik Mulder. (1996). Diminished effect of etidronate in vitamin D deficient osteopenic postmenopausal women. European Journal of Clinical Pharmacology. 51(2). 145–147. 38 indexed citations

16.

Koster, Joseph C., et al.. (1995). Kinetic Properties of the .alpha.2.beta.1 and .alpha.2.beta.2 Isoenzymes of the Na,K-ATPase. Biochemistry. 34(1). 319–325. 72 indexed citations

17.

Koster, Joseph C., Gustavo Blanco, & Robert W. Mercer. (1995). A Cytoplasmic Region of the Na,K-ATPase α-Subunit Is Necessary for Specific α/α Association. Journal of Biological Chemistry. 270(24). 14332–14339. 40 indexed citations

18.

Ouwendijk, Rob J., et al.. (1994). Budd-Chiari syndrome in a young patient with anticardiolipin antibodies: need for prolonged anticoagulant treatment.. Gut. 35(7). 1004–1006. 20 indexed citations

19.

Koster, Joseph C., et al.. (1992). The human M creatine kinase gene enhancer contains multiple functional interacting domains. Nucleic Acids Research. 20(9). 2313–2320. 15 indexed citations

20.

Mulder, Hindrik, et al.. (1989). Influence of pharmacological doses of calcitonin on serum osteocalcin concentration in patients with Paget's disease of the bone. European Journal of Endocrinology. 120(6). 721–723. 3 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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