Sho Sendo

713 total citations
37 papers, 515 citations indexed

About

Sho Sendo is a scholar working on Rheumatology, Immunology and Physiology. According to data from OpenAlex, Sho Sendo has authored 37 papers receiving a total of 515 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Rheumatology, 12 papers in Immunology and 9 papers in Physiology. Recurrent topics in Sho Sendo's work include Sarcoidosis and Beryllium Toxicity Research (6 papers), Immune cells in cancer (5 papers) and Vasculitis and related conditions (5 papers). Sho Sendo is often cited by papers focused on Sarcoidosis and Beryllium Toxicity Research (6 papers), Immune cells in cancer (5 papers) and Vasculitis and related conditions (5 papers). Sho Sendo collaborates with scholars based in Japan and United States. Sho Sendo's co-authors include Akio Morinobu, Jun Saegusa, S. Takahashi, T. Okano, Keisuke Nishimura, Yasuhiro Irino, Kengo Akashi, Yo Ueda, Goichi Kageyama and Shinwa Tanaka and has published in prestigious journals such as Scientific Reports, Frontiers in Immunology and Annals of the Rheumatic Diseases.

In The Last Decade

Sho Sendo

31 papers receiving 507 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sho Sendo Japan 11 167 164 133 101 85 37 515
S. Takahashi Japan 11 135 0.8× 164 1.0× 106 0.8× 76 0.8× 34 0.4× 33 455
Jason Hwang United States 12 92 0.6× 255 1.6× 100 0.8× 80 0.8× 73 0.9× 25 675
Junya Yamahana Japan 10 152 0.9× 172 1.0× 142 1.1× 30 0.3× 93 1.1× 14 570
Hermann-Josef GroCombining Diaeresisne Germany 8 316 1.9× 151 0.9× 114 0.9× 91 0.9× 63 0.7× 8 740
Ewa Gińdzieńska‐Sieśkiewicz Poland 10 122 0.7× 168 1.0× 110 0.8× 47 0.5× 25 0.3× 34 427
Bihua Xu China 11 219 1.3× 237 1.4× 113 0.8× 32 0.3× 34 0.4× 14 588
Keiichi Yoshimoto Japan 10 249 1.5× 190 1.2× 194 1.5× 44 0.4× 86 1.0× 14 869
Yasunori Matsuki Japan 15 221 1.3× 122 0.7× 230 1.7× 61 0.6× 75 0.9× 40 662
Hongyan Wen China 16 109 0.7× 159 1.0× 104 0.8× 28 0.3× 47 0.6× 45 527
Lin Tang China 17 74 0.4× 273 1.7× 83 0.6× 52 0.5× 103 1.2× 68 675

Countries citing papers authored by Sho Sendo

Since Specialization
Citations

This map shows the geographic impact of Sho Sendo's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sho Sendo with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sho Sendo more than expected).

Fields of papers citing papers by Sho Sendo

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sho Sendo. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sho Sendo. The network helps show where Sho Sendo may publish in the future.

Co-authorship network of co-authors of Sho Sendo

This figure shows the co-authorship network connecting the top 25 collaborators of Sho Sendo. A scholar is included among the top collaborators of Sho Sendo based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sho Sendo. Sho Sendo is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
2.
Sendo, Sho, et al.. (2024). Musculoskeletal ultrasound findings in adult-onset Still's disease: A case series. Joint Bone Spine. 91(6). 105771–105771.
3.
Espen, Benjamin Van, Andrew Wilson, Sho Sendo, et al.. (2024). Laser Capture Microscopy RNA Sequencing for Topological Mapping of Synovial Pathology During Rheumatoid Arthritis. Arthritis & Rheumatology. 76(8). 1243–1251.
4.
Sendo, Sho, David L. Boyle, Robert J. Benschop, et al.. (2024). Dysregulated NUB1 and Neddylation Enhances Rheumatoid Arthritis Fibroblast‐Like Synoviocyte Inflammatory Responses. Arthritis & Rheumatology. 76(8). 1252–1262. 6 indexed citations
5.
Sendo, Sho, William B. Kiosses, Shen K. Yang, et al.. (2023). Clustering of phosphatase RPTPα promotes Src signaling and the arthritogenic action of synovial fibroblasts. Science Signaling. 16(792). eabn8668–eabn8668. 2 indexed citations
6.
Mizutani, Satoshi, Junko Nishio, Kanoh Kondo, et al.. (2021). Treatment with an Anti-CX3CL1 Antibody Suppresses M1 Macrophage Infiltration in Interstitial Lung Disease in SKG Mice. Pharmaceuticals. 14(5). 474–474. 11 indexed citations
7.
Yamamoto, Yuzuru, T. Okano, Kengo Akashi, et al.. (2021). Soluble guanylate cyclase stimulator reduced the gastrointestinal fibrosis in bleomycin-induced mouse model of systemic sclerosis. Arthritis Research & Therapy. 23(1). 133–133. 4 indexed citations
8.
Saegusa, Jun, Sho Sendo, Yo Ueda, et al.. (2021). Effect of resolvin D5 on T cell differentiation and osteoclastogenesis analyzed by lipid mediator profiling in the experimental arthritis. Scientific Reports. 11(1). 17312–17312. 21 indexed citations
9.
Kageyama, Goichi, Akira Ōnishi, Yo Ueda, et al.. (2019). Subjective well‐being among rheumatoid arthritis patients. International Journal of Rheumatic Diseases. 22(10). 1863–1870. 6 indexed citations
10.
Sendo, Sho, et al.. (2019). Tofacitinib facilitates the expansion of myeloid-derived suppressor cells and ameliorates interstitial lung disease in SKG mice. Arthritis Research & Therapy. 21(1). 184–184. 46 indexed citations
11.
Ueda, Yo, Jun Saegusa, T. Okano, et al.. (2019). Additive effects of inhibiting both mTOR and glutamine metabolism on the arthritis in SKG mice. Scientific Reports. 9(1). 6374–6374. 18 indexed citations
12.
Sendo, Sho, Jun Saegusa, & Akio Morinobu. (2018). Myeloid-derived suppressor cells in non-neoplastic inflamed organs. Inflammation and Regeneration. 38(1). 19–19. 32 indexed citations
13.
Takahashi, S., Jun Saegusa, Sho Sendo, et al.. (2017). Glutaminase 1 plays a key role in the cell growth of fibroblast-like synoviocytes in rheumatoid arthritis. Arthritis Research & Therapy. 19(1). 76–76. 96 indexed citations
14.
Akashi, Kengo, Jun Saegusa, Sho Sendo, et al.. (2016). Knockout of endothelin type B receptor signaling attenuates bleomycin-induced skin sclerosis in mice. Arthritis Research & Therapy. 18(1). 113–113. 10 indexed citations
15.
Kumagai, Shunichi, et al.. (2016). AB0240 MMP-3 as A Biomarker of Disease Activity of Rheumatoid Arthritis. Annals of the Rheumatic Diseases. 75. 980–980. 2 indexed citations
16.
Sendo, Sho, Jun Saegusa, Yukiko Morinaga, et al.. (2015). IgG4-related disease manifesting as pericarditis with elevated adenosine deaminase and IL-10 levels in pericardial fluid. Modern Rheumatology. 27(5). 894–897. 5 indexed citations
18.
Morinobu, Akio, Goh Tsuji, Shimpei Kasagi, et al.. (2011). Role of imaging studies in the diagnosis and evaluation of giant cell arteritis in Japanese: report of eight cases. Modern Rheumatology. 21(4). 391–396. 8 indexed citations
19.
Kasagi, Shimpei, Jun Saegusa, Goh Tsuji, et al.. (2011). Epidural spinal tumor and periaortitis as rare complications of Wegener’s granulomatosis. Modern Rheumatology. 21(6). 678–683. 7 indexed citations
20.
Yamashita, Yumi, Sho Sendo, Toshiyuki Hosokawa, et al.. (1998). Exogenous thrombospondin stimulates the proliferation of non-thrombospondin-producing cells.. International Journal of Oncology. 13(2). 355–9. 7 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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