Matthew D. Linnik

4.5k total citations · 2 hit papers
61 papers, 3.6k citations indexed

About

Matthew D. Linnik is a scholar working on Rheumatology, Radiology, Nuclear Medicine and Imaging and Molecular Biology. According to data from OpenAlex, Matthew D. Linnik has authored 61 papers receiving a total of 3.6k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Rheumatology, 19 papers in Radiology, Nuclear Medicine and Imaging and 18 papers in Molecular Biology. Recurrent topics in Matthew D. Linnik's work include Systemic Lupus Erythematosus Research (23 papers), Monoclonal and Polyclonal Antibodies Research (19 papers) and T-cell and B-cell Immunology (9 papers). Matthew D. Linnik is often cited by papers focused on Systemic Lupus Erythematosus Research (23 papers), Monoclonal and Polyclonal Antibodies Research (19 papers) and T-cell and B-cell Immunology (9 papers). Matthew D. Linnik collaborates with scholars based in United States, United Kingdom and Australia. Matthew D. Linnik's co-authors include R. Howard Zobrist, John P. MacManus, Richard Furie, Howard J. Federoff, Michael D. Geschwind, Peter Zahos, Mario H. Cardiel, G. Michael Iverson, Keith A. Cockerill and Vibeke Strand and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Lancet and Journal of Biological Chemistry.

In The Last Decade

Matthew D. Linnik

59 papers receiving 3.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Evidence supporting a role for programmed cell death in focal cerebral ischemia in rats.

1993 619 citations Matthew D. Linnik et al. Stroke profile →
Baricitinib for systemic lupus erythematosus: a double-blind, randomised, placebo-controlled, phase 2 trial

2018 356 citations Daniel J. Wallace, Richard Furie et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Matthew D. Linnik United States	26	1.4k	1.2k	764	706	572	61	3.6k
Yair Anikster Israel	36	1.9k 1.4×	231 0.2×	378 0.5×	247 0.3×	170 0.3×	132	4.2k
Yasuyuki Kihara Japan	30	1.9k 1.4×	417 0.3×	438 0.6×	247 0.3×	49 0.1×	89	3.6k
Ryuichi Hattori Japan	29	838 0.6×	105 0.1×	751 1.0×	492 0.7×	482 0.8×	83	3.8k
Christopher M. Hovens Australia	38	1.8k 1.3×	210 0.2×	281 0.4×	484 0.7×	224 0.4×	121	3.9k
Kiyoshi Suzuma Japan	42	2.8k 2.1×	103 0.1×	403 0.5×	304 0.4×	1.9k 3.3×	123	6.8k
Kousuke Noda Japan	41	2.2k 1.6×	101 0.1×	456 0.6×	192 0.3×	1.3k 2.3×	168	5.2k
Alex Agrotis Australia	35	1.2k 0.9×	166 0.1×	1.3k 1.7×	121 0.2×	182 0.3×	62	3.3k
Seijiro Mori Japan	31	2.5k 1.8×	292 0.2×	380 0.5×	189 0.3×	131 0.2×	137	4.5k
Linda S. Higgins United States	34	2.1k 1.6×	104 0.1×	395 0.5×	347 0.5×	145 0.3×	66	4.0k
Ikuko Kondo Japan	33	1.7k 1.3×	212 0.2×	218 0.3×	499 0.7×	109 0.2×	128	3.8k

Countries citing papers authored by Matthew D. Linnik

Since Specialization

Citations

This map shows the geographic impact of Matthew D. Linnik's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Matthew D. Linnik with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Matthew D. Linnik more than expected).

Fields of papers citing papers by Matthew D. Linnik

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Matthew D. Linnik. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Matthew D. Linnik. The network helps show where Matthew D. Linnik may publish in the future.

Co-authorship network of co-authors of Matthew D. Linnik

This figure shows the co-authorship network connecting the top 25 collaborators of Matthew D. Linnik. A scholar is included among the top collaborators of Matthew D. Linnik based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Matthew D. Linnik. Matthew D. Linnik is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Jaroszewski, Lukasz, et al.. (2024). B‐ and T‐Lymphocyte Attenuator in Systemic Lupus Erythematosus Disease Pathogenesis. Clinical Pharmacology & Therapeutics. 116(1). 247–256. 4 indexed citations

Espen, Benjamin Van, Andrew Wilson, Sho Sendo, et al.. (2024). Laser Capture Microscopy RNA Sequencing for Topological Mapping of Synovial Pathology During Rheumatoid Arthritis. Arthritis & Rheumatology. 76(8). 1243–1251.

Hoffman, Robert W., Joan T. Merrill, Michelle Petri, et al.. (2016). Gene Expression and Pharmacodynamic Changes in 1,760 Systemic Lupus Erythematosus Patients From Two Phase III Trials of BAFF Blockade With Tabalumab. Arthritis & Rheumatology. 69(3). 643–654. 57 indexed citations

Cardiel, Mario H., James A. Tumlin, Richard Furie, et al.. (2008). Abetimus sodium for renal flare in systemic lupus erythematosus: Results of a randomized, controlled phase III trial. Arthritis & Rheumatism. 58(8). 2470–2480. 107 indexed citations

O’Rourke, Anne M., A. L. Miller, Christina Keßler, et al.. (2007). Anti-Inflammatory Effects of LJP 1586 [Z-3-Fluoro-2-(4-methoxybenzyl)allylamine Hydrochloride], an Amine-Based Inhibitor of Semicarbazide-Sensitive Amine Oxidase Activity. Journal of Pharmacology and Experimental Therapeutics. 324(2). 867–875. 48 indexed citations

Salter–Cid, Luisa, et al.. (2006). Vascular Adhesion Protein-1 Plays an Important Role in Postischemic Inflammation and Neuropathology in Diabetic, Estrogen-Treated Ovariectomized Female Rats Subjected to Transient Forebrain Ischemia. Journal of Pharmacology and Experimental Therapeutics. 317(1). 19–29. 44 indexed citations

Shi, Tong, Bill Giannakopoulos, Xiaokai Yan, et al.. (2006). Anti–β₂‐glycoprotein I antibodies in complex with β₂‐glycoprotein I can activate platelets in a dysregulated manner via glycoprotein Ib‐IX‐V. Arthritis & Rheumatism. 54(8). 2558–2567. 133 indexed citations

Salter–Cid, Luisa, Eric W. Wang, Anne M. O’Rourke, et al.. (2005). Anti-Inflammatory Effects of Inhibiting the Amine Oxidase Activity of Semicarbazide-Sensitive Amine Oxidase. Journal of Pharmacology and Experimental Therapeutics. 315(2). 553–562. 61 indexed citations

Linnik, Matthew D., et al.. (2005). Relationship between anti–double‐stranded DNA antibodies and exacerbation of renal disease in patients with systemic lupus erythematosus. Arthritis & Rheumatism. 52(4). 1129–1137. 162 indexed citations

10.

Cockerill, Keith A., Matthew D. Linnik, & G. Michael Iverson. (2004). Detection and characterization of B cell epitopes on β2-glycoprotein I. Clinical Immunology. 112(2). 129–135. 5 indexed citations

11.

Alarcón-Segovia, D, James A. Tumlin, Richard Furie, et al.. (2003). LJP 394 for the prevention of renal flare in patients with systemic lupus erythematosus: Results from a randomized, double‐blind, placebo‐controlled study. Arthritis & Rheumatism. 48(2). 442–454. 183 indexed citations

12.

Linnik, Matthew D.. (2001). A novel therapeutic approach for treating antiphospholipid syndrome based on tolerizing anti-β2-GPI B cells. Stroke. 32. 330–330. 1 indexed citations

13.

Victor, Kimberly D., et al.. (2001). Identification of a peptide that protects the human acetylcholine receptor against antigenic modulation. Journal of Immunological Methods. 251(1-2). 177–186. 6 indexed citations

14.

Sem, Daniel S., et al.. (1999). Antibody Affinities and Relative Titers in Polyclonal Populations: Surface Plasmon Resonance Analysis of anti-DNA Antibodies. Archives of Biochemistry and Biophysics. 372(1). 62–68. 17 indexed citations

15.

Peet, Norton P., Hwa-Ok Kim, Michael R. Angelastro, et al.. (1999). Hydroxyoxazolidines as α-aminoacetaldehyde equivalents: Novel inhibitors of calpain. Bioorganic & Medicinal Chemistry Letters. 9(16). 2365–2370. 6 indexed citations

16.

Linnik, Matthew D., et al.. (1995). Apoptotic DNA fragmentation in the rat cerebral cortex induced by permanent middle cerebral artery occlusion. Molecular Brain Research. 32(1). 116–124. 132 indexed citations

17.

Linnik, Matthew D., et al.. (1993). Induction of programmed cell death in a dorsal root ganglia × neuroblastoma cell line. Journal of Neurobiology. 24(4). 433–446. 14 indexed citations

18.

Linnik, Matthew D. & Tony Jer‐Fu Lee. (1993). Effect of Hemoglobin on Sympathetic Neurovascular Transmission in the Porcine Cerebral Circulation. Journal of Cardiovascular Pharmacology. 21(4). 615–620. 6 indexed citations

19.

Borcherding, David R., Andrew T. McPhail, Donna M. Radzik, et al.. (1992). The resolution, isolation, and pharmacological characterization of the enantiomers of a benzamide containing a chiral sulfoxide. Chirality. 4(3). 155–162. 9 indexed citations

20.

Linnik, Matthew D., et al.. (1991). Analysis of serotonergic mechanisms underlying benzamide-induced gastroprokinesis.. Journal of Pharmacology and Experimental Therapeutics. 259(2). 501–507. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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