Shi‐Young Park

1.4k total citations · 1 hit paper
16 papers, 911 citations indexed

About

Shi‐Young Park is a scholar working on Molecular Biology, Physiology and Epidemiology. According to data from OpenAlex, Shi‐Young Park has authored 16 papers receiving a total of 911 indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 7 papers in Physiology and 4 papers in Epidemiology. Recurrent topics in Shi‐Young Park's work include Adipose Tissue and Metabolism (6 papers), Metabolism, Diabetes, and Cancer (4 papers) and Muscle Physiology and Disorders (3 papers). Shi‐Young Park is often cited by papers focused on Adipose Tissue and Metabolism (6 papers), Metabolism, Diabetes, and Cancer (4 papers) and Muscle Physiology and Disorders (3 papers). Shi‐Young Park collaborates with scholars based in South Korea, United States and Japan. Shi‐Young Park's co-authors include Cheol Soo Choi, Yung-Jin Kim, Won‐Jun Jang, Cheol Soo Choi, Eui-Yeun Yi, Ja Hyun Koo, Sang Geon Kim, Ting Wen, Katsutaro Morino and Natsuko Ohashi and has published in prestigious journals such as Journal of Clinical Investigation, Diabetes and Scientific Reports.

In The Last Decade

Shi‐Young Park

14 papers receiving 901 citations

Hit Papers

Insulin Resistance: From Mechanisms to Therapeutic Strate... 2021 2026 2022 2024 2021 100 200 300 400 500

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Shi‐Young Park South Korea 11 312 282 254 214 116 16 911
Tenagne D. Challa Switzerland 15 293 0.9× 278 1.0× 276 1.1× 127 0.6× 87 0.8× 18 743
Po‐Shiuan Hsieh Taiwan 19 298 1.0× 369 1.3× 318 1.3× 283 1.3× 169 1.5× 58 1.1k
Olena Oliyarnyk Czechia 18 367 1.2× 253 0.9× 177 0.7× 144 0.7× 146 1.3× 48 1.0k
Marta Chacińska Poland 7 308 1.0× 248 0.9× 201 0.8× 125 0.6× 98 0.8× 8 700
Francesca Iannantuoni Spain 15 497 1.6× 229 0.8× 185 0.7× 300 1.4× 191 1.6× 23 1.2k
Min Young Kim South Korea 12 353 1.1× 245 0.9× 262 1.0× 120 0.6× 116 1.0× 15 958
Sofie Desmet Belgium 8 460 1.5× 175 0.6× 190 0.7× 173 0.8× 100 0.9× 13 957
Iria Nieto-Vázquez Spain 10 280 0.9× 288 1.0× 313 1.2× 121 0.6× 84 0.7× 11 747
Kazuo Fujisawa Japan 10 474 1.5× 300 1.1× 153 0.6× 152 0.7× 93 0.8× 12 925
Katarina Lalić Serbia 16 284 0.9× 258 0.9× 196 0.8× 296 1.4× 168 1.4× 75 1.0k

Countries citing papers authored by Shi‐Young Park

Since Specialization
Citations

This map shows the geographic impact of Shi‐Young Park's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shi‐Young Park with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shi‐Young Park more than expected).

Fields of papers citing papers by Shi‐Young Park

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shi‐Young Park. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shi‐Young Park. The network helps show where Shi‐Young Park may publish in the future.

Co-authorship network of co-authors of Shi‐Young Park

This figure shows the co-authorship network connecting the top 25 collaborators of Shi‐Young Park. A scholar is included among the top collaborators of Shi‐Young Park based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shi‐Young Park. Shi‐Young Park is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

16 of 16 papers shown
1.
Park, Shi‐Young, Yeongmin Kim, Hyunhee Oh, et al.. (2025). Activin E is a new guardian protecting against hepatic steatosis via inhibiting lipolysis in white adipose tissue. Experimental & Molecular Medicine. 57(2). 466–477. 1 indexed citations
2.
Park, Shi‐Young, Miseon Lee, Seungyoon Nam, et al.. (2025). ARHGEF3 coordinates adipocyte hypertrophy and differentiation through dual YAP-RhoA and PPARγ activation. Journal of Advanced Research. 79. 445–460.
3.
Park, Shi‐Young, et al.. (2025). Skeletal Muscle‐Specific Deletion of E3 Ligase Asb2 Enhances Muscle Mass and Strength. Journal of Cachexia Sarcopenia and Muscle. 16(4). e70007–e70007.
4.
Song, Taejeong, Sanghee Park, HeeJoo Kim, et al.. (2024). Free essential amino acid feeding improves endurance during resistance training via DRP1‐dependent mitochondrial remodelling. Journal of Cachexia Sarcopenia and Muscle. 15(5). 1651–1663. 2 indexed citations
5.
Um, Jee‐Hyun, Shi‐Young Park, Hui‐Young Lee, et al.. (2022). Bone morphogenic protein 9 is a novel thermogenic hepatokine secreted in response to cold exposure. Metabolism. 129. 155139–155139. 14 indexed citations
6.
Kim, Jinyoung, Seong‐Hun Kim, Hyereen Kang, et al.. (2021). TFEB–GDF15 axis protects against obesity and insulin resistance as a lysosomal stress response. Nature Metabolism. 3(3). 410–427. 53 indexed citations
7.
Park, Shi‐Young, et al.. (2021). Insulin Resistance: From Mechanisms to Therapeutic Strategies. Diabetes & Metabolism Journal. 46(1). 15–37. 588 indexed citations breakdown →
8.
Kim, Ayoung, Ja Hyun Koo, Xing Jin, et al.. (2021). Ablation of USP21 in skeletal muscle promotes oxidative fibre phenotype, inhibiting obesity and type 2 diabetes. Journal of Cachexia Sarcopenia and Muscle. 12(6). 1669–1689. 24 indexed citations
9.
Park, Hyun‐Jun, et al.. (2020). The essential role of fructose-1,6-bisphosphatase 2 enzyme in thermal homeostasis upon cold stress. Experimental & Molecular Medicine. 52(3). 485–496. 20 indexed citations
10.
Lee, Eun Hui, Michal Itan, Perri Rozenberg, et al.. (2018). Eosinophils support adipocyte maturation and promote glucose tolerance in obesity. Scientific Reports. 8(1). 9894–9894. 66 indexed citations
11.
Koo, Ja Hyun, Tae Hyun Kim, Shi‐Young Park, et al.. (2017). Gα13 ablation reprograms myofibers to oxidative phenotype and enhances whole-body metabolism. Journal of Clinical Investigation. 127(10). 3845–3860. 24 indexed citations
12.
Murata, Koichiro, Katsutaro Morino, Shogo Ida, et al.. (2017). Lack of O-GlcNAcylation enhances exercise-dependent glucose utilization potentially through AMP-activated protein kinase activation in skeletal muscle. Biochemical and Biophysical Research Communications. 495(2). 2098–2104. 16 indexed citations
13.
Ohashi, Natsuko, Katsutaro Morino, Shogo Ida, et al.. (2017). Pivotal Role of O-GlcNAc Modification in Cold-Induced Thermogenesis by Brown Adipose Tissue Through Mitochondrial Biogenesis. Diabetes. 66(9). 2351–2362. 32 indexed citations
14.
Yi, Eui-Yeun, et al.. (2015). Mitochondrial dysfunction induces EMT through the TGF-β/Smad/Snail signaling pathway in Hep3B hepatocellular carcinoma cells. International Journal of Oncology. 47(5). 1845–1853. 40 indexed citations
15.
Kim, Su-Ryun, Soo‐Kyung Bae, Hyun-Joo Park, et al.. (2010). Thromboxane A2 increases endothelial permeability through upregulation of interleukin-8. Biochemical and Biophysical Research Communications. 397(3). 413–419. 27 indexed citations
16.
Park, Shi‐Young, et al.. (2008). KR-31831, benzopyran derivative, inhibits VEGF-induced angiogenesis of HUVECs through suppressing KDR expression. International Journal of Oncology. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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