Shih‐Kai Wang

1.3k total citations
42 papers, 1.0k citations indexed

About

Shih‐Kai Wang is a scholar working on Molecular Biology, Rheumatology and Oral Surgery. According to data from OpenAlex, Shih‐Kai Wang has authored 42 papers receiving a total of 1.0k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Molecular Biology, 27 papers in Rheumatology and 15 papers in Oral Surgery. Recurrent topics in Shih‐Kai Wang's work include Bone and Dental Protein Studies (27 papers), dental development and anomalies (26 papers) and Oral and Maxillofacial Pathology (15 papers). Shih‐Kai Wang is often cited by papers focused on Bone and Dental Protein Studies (27 papers), dental development and anomalies (26 papers) and Oral and Maxillofacial Pathology (15 papers). Shih‐Kai Wang collaborates with scholars based in United States, Taiwan and South Korea. Shih‐Kai Wang's co-authors include James P. Simmer, Jan C.‐C. Hu, Bryan M. Reid, Amelia S. Richardson, Yuanyuan Hu, Murim Choi, Jung‐Wook Kim, Figen Seymen, Jie Yang and Koray Gençay and has published in prestigious journals such as PLoS ONE, Scientific Reports and Biochemical and Biophysical Research Communications.

In The Last Decade

Shih‐Kai Wang

40 papers receiving 994 citations

Peers

Shih‐Kai Wang

RHEUM

UROLO

GENET

Gang Fu China

Wenjuan Yan China

Katsumasa Maeda Japan

Hitoshi Hotokezaka Japan

Sikder M. Asaduzzaman Bangladesh

Takashi Takaki Japan

Max M. Villa United States

Tapani E. Sorvari Finland

S Roy United Kingdom

Sreenatha Kirakodu United States

Gang Fu China

Shih‐Kai Wang

638 ×0.9

208 ×0.4

RHEUM

83 ×0.4

21 ×0.2

UROLO

279 ×2.3

GENET

Citations per year, relative to Shih‐Kai Wang Shih‐Kai Wang (= 1×) peers Gang Fu

Countries citing papers authored by Shih‐Kai Wang

Since Specialization

Citations

This map shows the geographic impact of Shih‐Kai Wang's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shih‐Kai Wang with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shih‐Kai Wang more than expected).

Fields of papers citing papers by Shih‐Kai Wang

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shih‐Kai Wang. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shih‐Kai Wang. The network helps show where Shih‐Kai Wang may publish in the future.

Co-authorship network of co-authors of Shih‐Kai Wang

This figure shows the co-authorship network connecting the top 25 collaborators of Shih‐Kai Wang. A scholar is included among the top collaborators of Shih‐Kai Wang based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shih‐Kai Wang. Shih‐Kai Wang is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Wang, Yin‐Lin, et al.. (2024). Orodental malformations associated with human MSX1 sequence variants. The Journal of the American Dental Association. 155(6). 484–495.e21. 1 indexed citations

Wang, Shih‐Kai, Yin‐Lin Wang, Figen Seymen, et al.. (2024). AMELX Mutations and Genotype–Phenotype Correlation in X-Linked Amelogenesis Imperfecta. International Journal of Molecular Sciences. 25(11). 6132–6132. 2 indexed citations

Wang, Yin‐Lin, Chia‐Hui Lin, Yi‐Jane Chen, et al.. (2023). PAX9 mutations and genetic synergism in familial tooth agenesis. Annals of the New York Academy of Sciences. 1524(1). 87–96. 9 indexed citations

Wang, Shih‐Kai, et al.. (2023). Orthodontically induced changes to the genetic profile in periodontal ligament tissue and cytokine release in gingival crevicular fluid – A pilot investigation. Journal of Dental Sciences. 19(1). 387–396.

Cheng, Feng-Chou, Guay‐Fen Huang, Yin‐Lin Wang, et al.. (2023). The implication of integrating pediatric education into a pediatric dentistry course for undergraduate dental students. Journal of Dental Sciences. 18(4). 1794–1803. 2 indexed citations

Wang, Shih‐Kai, et al.. (2023). Molecular signaling and mechanisms of low-level laser-induced gene expression in cells involved in orthodontic tooth movement. Journal of the Formosan Medical Association. 123(4). 442–451. 1 indexed citations

Wang, Shih‐Kai, Hong Zhang, Yin‐Lin Wang, et al.. (2022). Phenotypic variability in LAMA3 ‐associated amelogenesis imperfecta. Oral Diseases. 29(8). 3514–3524. 9 indexed citations

Liang, Tian, Shih‐Kai Wang, Charles E. Smith, et al.. (2022). Enamel defects in Acp4R110C/R110C mice and human ACP4 mutations. Scientific Reports. 12(1). 16477–16477. 6 indexed citations

Liang, Tian, Yuanyuan Hu, Hong Zhang, et al.. (2021). Mouse Dspp frameshift model of human dentinogenesis imperfecta. Scientific Reports. 11(1). 20653–20653. 13 indexed citations

10.

Simmer, James P., Jan C.‐C. Hu, Yuanyuan Hu, et al.. (2021). A genetic model for the secretory stage of dental enamel formation. Journal of Structural Biology. 213(4). 107805–107805. 34 indexed citations

11.

Zhang, Hong, Yuanyuan Hu, Figen Seymen, et al.. (2019). ENAM mutations and digenic inheritance. Molecular Genetics & Genomic Medicine. 7(10). e00928–e00928. 12 indexed citations

12.

Song, Zhigang, Xiyi Hu, Shih‐Kai Wang, et al.. (2018). Heat exposure affects jejunal tight junction remodeling independently of adenosine monophosphate-activated protein kinase in 9-day-old broiler chicks. Poultry Science. 97(10). 3681–3690. 40 indexed citations

13.

Kang, Xuewen, Juanjuan Hu, Shih‐Kai Wang, & Jinfeng Wang. (2015). Effectiveness of muscle basal lamina carrying neural stem cells and olfactory ensheathing cells in spinal cord repair. Genetics and Molecular Research. 14(4). 13437–13455. 9 indexed citations

14.

Vieira, Alexandre R., Moses Lee, Filippo Pinto e Vairo, et al.. (2015). Root anomalies and dentin dysplasia in autosomal recessive hyperphosphatemic familial tumoral calcinosis (HFTC). Oral Surgery Oral Medicine Oral Pathology and Oral Radiology. 120(6). e235–e239. 9 indexed citations

15.

Wang, Shih‐Kai, Murim Choi, Amelia S. Richardson, et al.. (2013). ITGB6 loss-of-function mutations cause autosomal recessive amelogenesis imperfecta. Human Molecular Genetics. 23(8). 2157–2163. 53 indexed citations

16.

Wang, Shih‐Kai, Yuanyuan Hu, James P. Simmer, et al.. (2013). FAM20A Mutations Can Cause Enamel-Renal Syndrome (ERS). PLoS Genetics. 9(2). e1003302–e1003302. 79 indexed citations

17.

Wang, Shih‐Kai, et al.. (2012). Novel PAX9 and COL1A2 Missense Mutations Causing Tooth Agenesis and OI/DGI without Skeletal Abnormalities. PLoS ONE. 7(12). e51533–e51533. 22 indexed citations

18.

Wang, Shih‐Kai, Yoshihiro Komatsu, & Yuji Mishina. (2011). Potential contribution of neural crest cells to dental enamel formation. Biochemical and Biophysical Research Communications. 415(1). 114–119. 28 indexed citations

19.

Wang, Shih‐Kai, et al.. (2011). Enamel malformations associated with a defined dentin sialophosphoprotein mutation in two families. European Journal Of Oral Sciences. 119(s1). 158–167. 24 indexed citations

20.

Chan, Hsun‐Liang, et al.. (2010). Altered Enamelin Phosphorylation Site Causes Amelogenesis Imperfecta. Journal of Dental Research. 89(7). 695–699. 45 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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