Shenhav Cohen

3.3k total citations · 2 hit papers
23 papers, 2.5k citations indexed

About

Shenhav Cohen is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Cardiology and Cardiovascular Medicine. According to data from OpenAlex, Shenhav Cohen has authored 23 papers receiving a total of 2.5k indexed citations (citations by other indexed papers that have themselves been cited), including 19 papers in Molecular Biology, 8 papers in Cellular and Molecular Neuroscience and 6 papers in Cardiology and Cardiovascular Medicine. Recurrent topics in Shenhav Cohen's work include Muscle Physiology and Disorders (14 papers), Genetic Neurodegenerative Diseases (8 papers) and Autophagy in Disease and Therapy (5 papers). Shenhav Cohen is often cited by papers focused on Muscle Physiology and Disorders (14 papers), Genetic Neurodegenerative Diseases (8 papers) and Autophagy in Disease and Therapy (5 papers). Shenhav Cohen collaborates with scholars based in Israel, United States and Germany. Shenhav Cohen's co-authors include Alfred L. Goldberg, James A. Nathan, Steven P. Gygi, David J. Glass, Esther Latres, Jeffrey J. Brault, David M. Valenzuela, Carlos A. Gartner, Bo Zhai and Kfir Baruch Umansky and has published in prestigious journals such as Nature, Proceedings of the National Academy of Sciences and Nature Communications.

In The Last Decade

Shenhav Cohen

23 papers receiving 2.5k citations

Hit Papers

align trajectories sort by overperformance

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Muscle wasting in disease: molecular mechanisms and promising therapies

2014 832 citations Shenhav Cohen, James A. Nathan et al. Nature Reviews Drug Discovery profile →
The extracellular matrix protein agrin promotes heart regeneration in mice

2017 478 citations Elad Bassat, Ilya Y. Shadrin et al. Nature profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Shenhav Cohen Israel	16	1.8k	693	623	327	278	23	2.5k
Roberta Sartori Italy	19	1.6k 0.9×	899 1.3×	428 0.7×	129 0.4×	147 0.5×	30	2.2k
Gilles Carnac France	35	2.8k 1.5×	590 0.9×	353 0.6×	263 0.8×	252 0.9×	70	3.3k
Katherine J. Ladner United States	22	2.5k 1.3×	1.1k 1.6×	227 0.4×	171 0.5×	135 0.5×	33	3.7k
Michelle Wehling‐Henricks United States	23	1.8k 1.0×	755 1.1×	250 0.4×	203 0.6×	370 1.3×	32	2.5k
Susanne Baumhueter United States	9	3.1k 1.7×	1.0k 1.5×	900 1.4×	302 0.9×	344 1.2×	11	4.0k
Michael P. Czubryt Canada	28	1.8k 1.0×	500 0.7×	239 0.4×	902 2.8×	415 1.5×	74	3.0k
María Gabriela Morales Chile	26	1.1k 0.6×	407 0.6×	252 0.4×	270 0.8×	286 1.0×	42	1.8k
Anjali K. Nath United States	20	1.4k 0.8×	702 1.0×	224 0.4×	298 0.9×	302 1.1×	38	3.2k
Mercè Jardı́ Spain	17	1.9k 1.0×	888 1.3×	290 0.5×	86 0.3×	318 1.1×	24	2.8k
Marina Bouché Italy	27	1.3k 0.7×	285 0.4×	204 0.3×	200 0.6×	199 0.7×	64	1.7k

Countries citing papers authored by Shenhav Cohen

Since Specialization

Citations

This map shows the geographic impact of Shenhav Cohen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shenhav Cohen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shenhav Cohen more than expected).

Fields of papers citing papers by Shenhav Cohen

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shenhav Cohen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shenhav Cohen. The network helps show where Shenhav Cohen may publish in the future.

Co-authorship network of co-authors of Shenhav Cohen

This figure shows the co-authorship network connecting the top 25 collaborators of Shenhav Cohen. A scholar is included among the top collaborators of Shenhav Cohen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shenhav Cohen. Shenhav Cohen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Cohen, Shenhav, et al.. (2022). The AAA‐ATPase ATAD1 and its partners promote degradation of desmin intermediate filaments in muscle. EMBO Reports. 23(12). e55175–e55175. 9 indexed citations

Cohen, Shenhav, et al.. (2021). Breakdown of Filamentous Myofibrils by the UPS–Step by Step. Biomolecules. 11(1). 110–110. 14 indexed citations

Agnetti, Giulio, Harald Herrmann, & Shenhav Cohen. (2021). New roles for desmin in the maintenance of muscle homeostasis. FEBS Journal. 289(10). 2755–2770. 70 indexed citations

Gilda, Jennifer E., et al.. (2021). A semiautomated measurement of muscle fiber size using the Imaris software. American Journal of Physiology-Cell Physiology. 321(3). C615–C631. 15 indexed citations

Gilda, Jennifer E., et al.. (2020). USP 1 deubiquitinates Akt to inhibit PI 3K‐Akt‐FoxO signaling in muscle during prolonged starvation. EMBO Reports. 21(4). e48791–e48791. 85 indexed citations

Dahan, Nitsan, et al.. (2020). A signaling hub of insulin receptor, dystrophin glycoprotein complex and plakoglobin regulates muscle size. Nature Communications. 11(1). 1381–1381. 33 indexed citations

Cohen, Shenhav. (2020). Role of calpains in promoting desmin filaments depolymerization and muscle atrophy. Biochimica et Biophysica Acta (BBA) - Molecular Cell Research. 1867(10). 118788–118788. 26 indexed citations

Yatsenko, Andriy S., Mariya M. Kucherenko, Yuanbin Xie, et al.. (2020). Profiling of the muscle-specific dystroglycan interactome reveals the role of Hippo signaling in muscular dystrophy and age-dependent muscle atrophy. BMC Medicine. 18(1). 8–8. 19 indexed citations

Shiloh, Ruth, Yaara Ber, Miriam Eisenstein, et al.. (2018). Non-canonical activation of DAPK2 by AMPK constitutes a new pathway linking metabolic stress to autophagy. Nature Communications. 9(1). 1759–1759. 31 indexed citations

10.

Cohen, Shenhav, et al.. (2018). GSK3-β promotes calpain-1–mediated desmin filament depolymerization and myofibril loss in atrophy. The Journal of Cell Biology. 217(10). 3698–3714. 52 indexed citations

11.

Bassat, Elad, Ilya Y. Shadrin, Kfir Baruch Umansky, et al.. (2017). The extracellular matrix protein agrin promotes heart regeneration in mice. Nature. 547(7662). 179–184. 478 indexed citations breakdown →

12.

Braunstein, Ilana, et al.. (2014). VWA domain of S5a restricts the ability to bind ubiquitin and Ubl to the 26S proteasome. Molecular Biology of the Cell. 25(25). 3988–3998. 12 indexed citations

13.

Cohen, Shenhav, James A. Nathan, & Alfred L. Goldberg. (2014). Muscle wasting in disease: molecular mechanisms and promising therapies. Nature Reviews Drug Discovery. 14(1). 58–74. 832 indexed citations breakdown →

14.

Cohen, Shenhav, Donghoon Lee, Bo Zhai, Steven P. Gygi, & Alfred L. Goldberg. (2014). Trim32 reduces PI3K–Akt–FoxO signaling in muscle atrophy by promoting plakoglobin–PI3K dissociation. The Journal of Cell Biology. 204(5). 747–758. 73 indexed citations

15.

Cohen, Shenhav, Bo Zhai, Steven P. Gygi, & Alfred L. Goldberg. (2012). Ubiquitylation by Trim32 causes coupled loss of desmin, Z-bands, and thin filaments in muscle atrophy. The Journal of Cell Biology. 198(4). 575–589. 154 indexed citations

16.

Junghans, Richard P., Ritesh Rathore, Q.Y. Ma, et al.. (2011). Phase I trial of anti-PSMA designer T cells in advanced prostate cancer.. Journal of Clinical Oncology. 29(7_suppl). 130–130. 1 indexed citations

17.

Cohen, Shenhav, Jeffrey J. Brault, Steven P. Gygi, et al.. (2009). During muscle atrophy, thick, but not thin, filament components are degraded by MuRF1-dependent ubiquitylation. The Journal of Cell Biology. 185(6). 1083–1095. 472 indexed citations

18.

Cohen, Shenhav, Jeffrey J. Brault, Steven P. Gygi, et al.. (2009). During muscle atrophy, thick, but not thin, filament components are degraded by MuRF1-dependent ubiquitylation. The Journal of Experimental Medicine. 206(7). i13–i13. 8 indexed citations

19.

Cohen, Shenhav, Sara Dovrat, Ronit Sarid, Eliezer Huberman, & Samuel Salzberg. (2005). JAK–STAT signaling involved in phorbol 12-myristate 13-acetate- and dimethyl sulfoxide-induced 2′-5′ oligoadenylate synthetase expression in human HL-60 leukemia cells. Leukemia Research. 29(8). 923–931. 13 indexed citations

20.

Salzberg, Samuel, et al.. (2000). Expression of a PKR Dominant-Negative Mutant in Myogenic Cells Interferes with the Myogenic Process. Experimental Cell Research. 254(1). 45–54. 22 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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