Shanwei Shen

427 total citations
21 papers, 352 citations indexed

About

Shanwei Shen is a scholar working on Cellular and Molecular Neuroscience, Physiology and Molecular Biology. According to data from OpenAlex, Shanwei Shen has authored 21 papers receiving a total of 352 indexed citations (citations by other indexed papers that have themselves been cited), including 7 papers in Cellular and Molecular Neuroscience, 7 papers in Physiology and 6 papers in Molecular Biology. Recurrent topics in Shanwei Shen's work include Gastrointestinal motility and disorders (6 papers), Neuropeptides and Animal Physiology (6 papers) and Urinary Bladder and Prostate Research (5 papers). Shanwei Shen is often cited by papers focused on Gastrointestinal motility and disorders (6 papers), Neuropeptides and Animal Physiology (6 papers) and Urinary Bladder and Prostate Research (5 papers). Shanwei Shen collaborates with scholars based in United States, China and South Korea. Shanwei Shen's co-authors include Li‐Ya Qiao, Chunmei Xia, Miao Liu, John R. Grider, Phillip B. Hylemon, Shunlin Ren, Douglas M. Heuman, Min Liu, Yongjie Ma and Daniel Rodrı́guez-Agudo and has published in prestigious journals such as Nature Communications, Gastroenterology and PLoS ONE.

In The Last Decade

Shanwei Shen

19 papers receiving 350 citations

Peers

Shanwei Shen

PHYSI

CMN

GASTR

UROLO

Jiuan-Miaw Liao Taiwan

Silke Wiegand Germany

Pierangelo Geppetti Italy

Rachel Price United States

Irina Zabbarova United States

Carlo Alberto Maggi Italy

Roddy J. Large Ireland

Kirti Tewari United States

Jun‐Ge Yu United States

Paz Recio Spain

Jiuan-Miaw Liao Taiwan

Shanwei Shen

116 ×0.9

PHYSI

45 ×0.5

CMN

121 ×1.6

23 ×0.4

GASTR

89 ×1.6

UROLO

Citations per year, relative to Shanwei Shen Shanwei Shen (= 1×) peers Jiuan-Miaw Liao

Countries citing papers authored by Shanwei Shen

Since Specialization

Citations

This map shows the geographic impact of Shanwei Shen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shanwei Shen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shanwei Shen more than expected).

Fields of papers citing papers by Shanwei Shen

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shanwei Shen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shanwei Shen. The network helps show where Shanwei Shen may publish in the future.

Co-authorship network of co-authors of Shanwei Shen

This figure shows the co-authorship network connecting the top 25 collaborators of Shanwei Shen. A scholar is included among the top collaborators of Shanwei Shen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shanwei Shen. Shanwei Shen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Zhang, Ran, Y. Q. Qi, Songwei Lu, et al.. (2025). Evaluating Chelerythrine Chloride as a Potential Treatment for Multidrug-Resistant Gonorrhea. Infection and Drug Resistance. Volume 18. 5109–5114.

Shen, Shanwei, Senthil K. Radhakrishnan, J. Chuck Harrell, et al.. (2024). The Human Intermediate Prolactin Receptor I-tail Contributes Breast Oncogenesis by Targeting Ras/MAPK Pathway. Endocrinology. 165(6). 3 indexed citations

Sharma, Divya, et al.. (2024). Plp1-expresssing perineuronal DRG cells facilitate colonic and somatic chronic mechanical pain involving Piezo2 upregulation in DRG neurons. Cell Reports. 43(5). 114230–114230. 2 indexed citations

Sharma, Divya, et al.. (2023). Piezo2 regulates colonic mechanical sensitivity in a sex specific manner in mice. Nature Communications. 14(1). 2158–2158. 12 indexed citations

Sierra, Salvador, Karan H. Muchhala, David L. Stevens, et al.. (2022). Sex-specific role for serotonin 5-HT2A receptor in modulation of opioid-induced antinociception and reward in mice. Neuropharmacology. 209. 108988–108988. 16 indexed citations

Shen, Shanwei, et al.. (2021). Beta 2-adrenergic receptor mediates noradrenergic action to induce cyclic adenosine monophosphate response element-binding protein phosphorylation in satellite glial cells of dorsal root ganglia to regulate visceral hypersensitivity. Pain. 163(1). 180–192. 20 indexed citations

Qiao, Li‐Ya, Chunmei Xia, Shanwei Shen, et al.. (2018). Urinary bladder organ hypertrophy is partially regulated by Akt1-mediated protein synthesis pathway. Life Sciences. 201. 63–71. 9 indexed citations

Shen, Shanwei, et al.. (2017). Regulation of transient receptor potential cation channel subfamily V1 protein synthesis by the phosphoinositide 3-kinase/Akt pathway in colonic hypersensitivity. Experimental Neurology. 295. 104–115. 23 indexed citations

Qiao, Li‐Ya, et al.. (2016). Inflammation and activity augment brain-derived neurotrophic factor peripheral release. Neuroscience. 318. 114–121. 23 indexed citations

10.

Shen, Shanwei, et al.. (2016). Phospholipase C gamma mediates endogenous brain-derived neurotrophic factor-regulated calcitonin gene-related peptide expression in colitis-induced visceral pain. Molecular Pain. 12. 17 indexed citations

11.

Liu, Miao, et al.. (2015). Endogenous BDNF augments NMDA receptor phosphorylation in the spinal cord via PLCγ, PKC, and PI3K/Akt pathways during colitis. Journal of Neuroinflammation. 12(1). 151–151. 44 indexed citations

12.

Xia, Chunmei, et al.. (2015). Colitis-induced bladder afferent neuronal activation is regulated by BDNF through PLCγ pathway. Experimental Neurology. 285(Pt B). 126–135. 16 indexed citations

13.

Shen, Shanwei, Derek M. Kendig, Sunila Mahavadi, et al.. (2015). Inhibition of NMDAR Reduces Bladder Hypertrophy and Improves Bladder Function in Cyclophosphamide Induced Cystitis. The Journal of Urology. 193(5). 1676–1683. 22 indexed citations

14.

Xia, Chunmei, Shanwei Shen, Frank Corwin, et al.. (2014). Suppression of the PI3K Pathway In Vivo Reduces Cystitis-Induced Bladder Hypertrophy and Restores Bladder Capacity Examined by Magnetic Resonance Imaging. PLoS ONE. 9(12). e114536–e114536. 20 indexed citations

15.

Shen, Shanwei, Chunmei Xia, & Li‐Ya Qiao. (2014). The urinary bladder of spontaneously hypertensive rat demonstrates bladder hypertrophy, inflammation, and fibrosis but not hyperplasia. Life Sciences. 121. 22–27. 13 indexed citations

16.

Qiao, Li‐Ya & Shanwei Shen. (2014). Mo2032 Brain-Derived Neurotrophic Factor Regulates Bladder Afferent Activity via PLCγ/Calcium Pathway During Colitis. Gastroenterology. 146(5). S–722.

17.

Xia, Chunmei, et al.. (2013). Endogenous PI3K/Akt and NMDAR act independently in the regulation of CREB activity in lumbosacral spinal cord in cystitis. Experimental Neurology. 250. 366–375. 39 indexed citations

18.

Grider, John R., et al.. (2012). Up-regulation of brain-derived neurotrophic factor is regulated by extracellular signal-regulated protein kinase 5 and by nerve growth factor retrograde signaling in colonic afferent neurons in colitis. Experimental Neurology. 238(2). 209–217. 29 indexed citations

19.

Shen, Shanwei & Houyan Song. (2009). Progesterone promotes propagation and viability of mouse embryonic stem cells.. PubMed. 61(5). 431–8. 2 indexed citations

20.

Loder, Merewyn K., Shanwei Shen, Paul Wren, et al.. (2000). The production and analysis of transgenic mice expressing the human serotonin transporter.. European Journal of Neuroscience. 12. 173–173. 2 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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