Shannon C. Kenney

10.4k total citations · 1 hit paper
145 papers, 8.5k citations indexed

About

Shannon C. Kenney is a scholar working on Oncology, Pathology and Forensic Medicine and Immunology. According to data from OpenAlex, Shannon C. Kenney has authored 145 papers receiving a total of 8.5k indexed citations (citations by other indexed papers that have themselves been cited), including 129 papers in Oncology, 57 papers in Pathology and Forensic Medicine and 44 papers in Immunology. Recurrent topics in Shannon C. Kenney's work include Viral-associated cancers and disorders (120 papers), Lymphoma Diagnosis and Treatment (57 papers) and Immune Cell Function and Interaction (28 papers). Shannon C. Kenney is often cited by papers focused on Viral-associated cancers and disorders (120 papers), Lymphoma Diagnosis and Treatment (57 papers) and Immune Cell Function and Interaction (28 papers). Shannon C. Kenney collaborates with scholars based in United States, Germany and France. Shannon C. Kenney's co-authors include Elizabeth Holley-Guthrie, Amy L. Adamson, David Gutsch, Joseph S. Pagano, Henri‐Jacques Delecluse, Janet E. Mertz, Amy Mauser, James Kamine, Nancy Raab‐Traub and Margaret L. Gulley and has published in prestigious journals such as Science, New England Journal of Medicine and Cell.

In The Last Decade

Shannon C. Kenney

139 papers receiving 8.4k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Epstein-Barr virus: Biology and clinical disease

2022 243 citations Shannon C. Kenney, Nancy Raab‐Traub et al. profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Shannon C. Kenney United States	56	6.6k	3.1k	2.3k	2.2k	1.6k	145	8.5k
Wolfgang Hammerschmidt Germany	59	6.5k 1.0×	3.7k 1.2×	2.6k 1.1×	2.4k 1.1×	2.1k 1.4×	136	9.5k
Kenzo Takada Japan	56	6.6k 1.0×	2.2k 0.7×	2.7k 1.2×	2.6k 1.2×	2.2k 1.4×	158	9.3k
Erle S. Robertson United States	57	6.4k 1.0×	4.0k 1.3×	1.4k 0.6×	2.1k 1.0×	2.8k 1.8×	226	9.3k
Rajiv Khanna Australia	57	5.5k 0.8×	4.7k 1.5×	5.5k 2.3×	2.0k 0.9×	1.5k 1.0×	222	11.1k
David A. Thorley‐Lawson United States	59	8.1k 1.2×	3.6k 1.2×	3.6k 1.5×	3.7k 1.7×	2.4k 1.5×	115	12.2k
Denis J. Moss Australia	55	5.6k 0.8×	2.9k 0.9×	4.8k 2.1×	2.0k 0.9×	1.1k 0.7×	141	8.9k
Riccardo Dolcetti Italy	48	4.8k 0.7×	1.4k 0.4×	2.1k 0.9×	3.2k 1.5×	1.6k 1.0×	255	8.3k
Blossom Damania United States	52	5.0k 0.8×	3.7k 1.2×	2.9k 1.2×	1.2k 0.6×	2.9k 1.9×	188	9.2k
Scott R. Burrows Australia	62	4.6k 0.7×	2.8k 0.9×	7.1k 3.0×	1.8k 0.8×	1.7k 1.1×	172	10.7k
Bala Chandran United States	54	5.0k 0.8×	4.7k 1.5×	2.3k 1.0×	791 0.4×	2.2k 1.4×	123	8.6k

Countries citing papers authored by Shannon C. Kenney

Since Specialization

Citations

This map shows the geographic impact of Shannon C. Kenney's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Shannon C. Kenney with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Shannon C. Kenney more than expected).

Fields of papers citing papers by Shannon C. Kenney

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Shannon C. Kenney. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Shannon C. Kenney. The network helps show where Shannon C. Kenney may publish in the future.

Co-authorship network of co-authors of Shannon C. Kenney

This figure shows the co-authorship network connecting the top 25 collaborators of Shannon C. Kenney. A scholar is included among the top collaborators of Shannon C. Kenney based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Shannon C. Kenney. Shannon C. Kenney is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

20 of 20 papers shown

Singh, Deo R., et al.. (2025). Inhibition of the Integrated stress response by Epstein-Barr virus oncoprotein LMP1 attenuates epithelial cell differentiation and lytic viral reactivation. PLoS Pathogens. 21(2). e1012934–e1012934. 3 indexed citations

Li, Xiaoyan, You Zhang, Jason C. Mixdorf, et al.. (2025). Development and Preclinical Evaluation of [⁶⁴Cu]Cu-NOTA-ABDB6: A CD70 and Albumin Dual-Binding Tracer with Improved Pharmacokinetics. Journal of Nuclear Medicine. 66(4). 552–558. 1 indexed citations

Bristol, Jillian A., Joshua Brand, Makoto Ohashi, et al.. (2022). Reduced IRF4 expression promotes lytic phenotype in Type 2 EBV-infected B cells. PLoS Pathogens. 18(4). e1010453–e1010453. 15 indexed citations

Ali, Ahmed, et al.. (2022). Rta is the principal activator of Epstein-Barr virus epithelial lytic transcription. PLoS Pathogens. 18(9). e1010886–e1010886. 13 indexed citations

Singh, Deo R., Scott E. Nelson, Jillian A. Bristol, et al.. (2022). Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes. PLoS Pathogens. 18(10). e1010868–e1010868. 5 indexed citations

Ohashi, Makoto, Dhananjay M. Nawandar, Denis Lee, et al.. (2021). ΔNp63α promotes Epstein-Barr virus latency in undifferentiated epithelial cells. PLoS Pathogens. 17(11). e1010045–e1010045. 12 indexed citations

Romero-Masters, James C., Shane M. Huebner, Makoto Ohashi, et al.. (2020). B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infection. PLoS Pathogens. 16(2). e1008365–e1008365. 31 indexed citations

Haverkos, Bradley M., Ludmila Katherine Martin, Mark Lustberg, et al.. (2018). Complete and Durable Responses in Primary Central Nervous System Posttransplant Lymphoproliferative Disorder with Zidovudine, Ganciclovir, Rituximab, and Dexamethasone. Clinical Cancer Research. 24(14). 3273–3281. 16 indexed citations

Tikhmyanova, Nadezhda, Kayla Martin, Fang Lü, et al.. (2017). Small molecule perturbation of the CAND1-Cullin1-ubiquitin cycle stabilizes p53 and triggers Epstein-Barr virus reactivation. PLoS Pathogens. 13(7). e1006517–e1006517. 9 indexed citations

10.

Jones, Richard J., Tawin Iempridee, Xiaobin Wang, et al.. (2016). Lenalidomide, Thalidomide, and Pomalidomide Reactivate the Epstein–Barr Virus Lytic Cycle through Phosphoinositide 3-Kinase Signaling and Ikaros Expression. Clinical Cancer Research. 22(19). 4901–4912. 39 indexed citations

11.

Ryan, Julie L., Richard J. Jones, Shannon C. Kenney, et al.. (2010). Epstein-Barr virus-specific methylation of human genes in gastric cancer cells. Infectious Agents and Cancer. 5(1). 27–27. 50 indexed citations

12.

Ryan, Julie L., Richard J. Jones, Sandra Elmore, et al.. (2009). Epstein-Barr Virus <i>WZhet</i> DNA Can Induce Lytic Replication in Epithelial Cells in vitro, although <i>WZhet</i> Is Not Detectable in Many Human Tissues in vivo. Intervirology. 52(1). 8–16. 5 indexed citations

13.

Jones, Richard J., William T. Seaman, Elizabeth A. Barlow, et al.. (2007). Roles of lytic viral infection and IL‐6 in early versus late passage lymphoblastoid cell lines and EBV‐associated lymphoproliferative disease. International Journal of Cancer. 121(6). 1274–1281. 50 indexed citations

14.

Israel, Bruce F., et al.. (2002). Chemotherapy induces lytic EBV replication and confers ganciclovir susceptibility to EBV-positive epithelial cell tumors.. PubMed. 62(6). 1920–6. 138 indexed citations

15.

Swenson, Jennifer J., Amy Mauser, William K. Kaufmann, & Shannon C. Kenney. (1999). The Epstein-Barr Virus Protein BRLF1 Activates S Phase Entry through E2F1 Induction. Journal of Virology. 73(8). 6540–6550. 52 indexed citations

16.

Adamson, Amy L. & Shannon C. Kenney. (1999). The Epstein-Barr Virus BZLF1 Protein Interacts Physically and Functionally with the Histone Acetylase CREB-Binding Protein. Journal of Virology. 73(8). 6551–6558. 97 indexed citations

17.

Kenney, Shannon C., et al.. (1998). Gene Therapy Strategies for Treating Epstein–Barr Virus-Associated Lymphomas: Comparison of Two Different Epstein–Barr Virus-Based Vectors. Human Gene Therapy. 9(8). 1131–1141. 28 indexed citations

18.

Zhang, Qisheng, Hong Yu, David I. Dorsky, et al.. (1996). Functional and physical interactions between the Epstein-Barr virus (EBV) proteins BZLF1 and BMRF1: Effects on EBV transcription and lytic replication. Journal of Virology. 70(8). 5131–5142. 80 indexed citations

19.

Furnari, Frank B., Valerie Zacny, E. Byrd Quinlivan, Shannon C. Kenney, & Joseph S. Pagano. (1994). RAZ, an Epstein-Barr virus transdominant repressor that modulates the viral reactivation mechanism. Journal of Virology. 68(3). 1827–1836. 46 indexed citations

20.

Kenney, Shannon C., Elizabeth Holley-Guthrie, E. Byrd Quinlivan, et al.. (1992). The Cellular Oncogene c- myb Can Interact Synergistically with the Epstein-Barr Virus BZLF1 Transactivator in Lymphoid Cells. Molecular and Cellular Biology. 12(1). 136–146. 24 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact