James C. Romero-Masters

550 total citations
17 papers, 449 citations indexed

About

James C. Romero-Masters is a scholar working on Oncology, Immunology and Pathology and Forensic Medicine. According to data from OpenAlex, James C. Romero-Masters has authored 17 papers receiving a total of 449 indexed citations (citations by other indexed papers that have themselves been cited), including 12 papers in Oncology, 10 papers in Immunology and 8 papers in Pathology and Forensic Medicine. Recurrent topics in James C. Romero-Masters's work include Viral-associated cancers and disorders (10 papers), Immune Cell Function and Interaction (8 papers) and Lymphoma Diagnosis and Treatment (8 papers). James C. Romero-Masters is often cited by papers focused on Viral-associated cancers and disorders (10 papers), Immune Cell Function and Interaction (8 papers) and Lymphoma Diagnosis and Treatment (8 papers). James C. Romero-Masters collaborates with scholars based in United States, Germany and United Kingdom. James C. Romero-Masters's co-authors include Shannon C. Kenney, Jillian A. Bristol, Shidong Ma, Eric Johannsen, Jenny E. Gumperz, Elizabeth A. Barlow, Makoto Ohashi, Mitchell Hayes, Erik A. Ranheim and Paul F. Lambert and has published in prestigious journals such as The Journal of Immunology, Journal of Virology and PLoS Pathogens.

In The Last Decade

James C. Romero-Masters

17 papers receiving 444 citations

Peers

James C. Romero-Masters
Jillian A. Bristol United States
Shidong Ma United States
Ana Raykova Switzerland
Fathima Zumla Cader United Kingdom
Gregory K. Hong United States
Leah Fitzsimmons United Kingdom
Bruce F. Israel United States
Anja M. Mehl United Kingdom
Nadine Meru Germany
Jillian A. Bristol United States
James C. Romero-Masters
Citations per year, relative to James C. Romero-Masters James C. Romero-Masters (= 1×) peers Jillian A. Bristol

Countries citing papers authored by James C. Romero-Masters

Since Specialization
Citations

This map shows the geographic impact of James C. Romero-Masters's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by James C. Romero-Masters with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites James C. Romero-Masters more than expected).

Fields of papers citing papers by James C. Romero-Masters

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by James C. Romero-Masters. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by James C. Romero-Masters. The network helps show where James C. Romero-Masters may publish in the future.

Co-authorship network of co-authors of James C. Romero-Masters

This figure shows the co-authorship network connecting the top 25 collaborators of James C. Romero-Masters. A scholar is included among the top collaborators of James C. Romero-Masters based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with James C. Romero-Masters. James C. Romero-Masters is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

17 of 17 papers shown
1.
Zhang, Liyan, et al.. (2025). MmuPV1 E7 promotes phenotypes associated with “high-risk” HPV infection in mouse keratinocytes. Journal of Virology. 99(11). e0109725–e0109725. 1 indexed citations
2.
Romero-Masters, James C., Miranda Grace, Denis Lee, et al.. (2023). MmuPV1 E7’s interaction with PTPN14 delays Epithelial differentiation and contributes to virus-induced skin disease. PLoS Pathogens. 19(4). e1011215–e1011215. 6 indexed citations
3.
Romero-Masters, James C., Laura K. Muehlbauer, Mitchell Hayes, et al.. (2023). MmuPV1 E6 induces cell proliferation and other hallmarks of cancer. mBio. 14(6). e0245823–e0245823. 3 indexed citations
4.
Bristol, Jillian A., Joshua Brand, Makoto Ohashi, et al.. (2022). Reduced IRF4 expression promotes lytic phenotype in Type 2 EBV-infected B cells. PLoS Pathogens. 18(4). e1010453–e1010453. 15 indexed citations
5.
Romero-Masters, James C., Paul F. Lambert, & Karl Münger. (2022). Molecular Mechanisms of MmuPV1 E6 and E7 and Implications for Human Disease. Viruses. 14(10). 2138–2138. 16 indexed citations
6.
Wei, Tao, Miranda Grace, Aayushi Uberoi, et al.. (2021). The Mus musculus Papillomavirus Type 1 E7 Protein Binds to the Retinoblastoma Tumor Suppressor: Implications for Viral Pathogenesis. mBio. 12(4). e0227721–e0227721. 9 indexed citations
7.
Romero-Masters, James C., Shane M. Huebner, Makoto Ohashi, et al.. (2020). B cells infected with Type 2 Epstein-Barr virus (EBV) have increased NFATc1/NFATc2 activity and enhanced lytic gene expression in comparison to Type 1 EBV infection. PLoS Pathogens. 16(2). e1008365–e1008365. 31 indexed citations
8.
Li, Chunrong, James C. Romero-Masters, Shane M. Huebner, et al.. (2020). EBNA2-deleted Epstein-Barr virus (EBV) isolate, P3HR1, causes Hodgkin-like lymphomas and diffuse large B cell lymphomas with type II and Wp-restricted latency types in humanized mice. PLoS Pathogens. 16(6). e1008590–e1008590. 21 indexed citations
9.
Romero-Masters, James C., Makoto Ohashi, Mitchell Hayes, et al.. (2020). An EBNA3A-Mutated Epstein-Barr Virus Retains the Capacity for Lymphomagenesis in a Cord Blood-Humanized Mouse Model. Journal of Virology. 94(10). 9 indexed citations
10.
Tikhmyanova, Nadezhda, James C. Romero-Masters, Xin Feng, et al.. (2019). Development of a novel inducer for EBV lytic therapy. Bioorganic & Medicinal Chemistry Letters. 29(16). 2259–2264. 7 indexed citations
11.
Bristol, Jillian A., Carrie B. Coleman, Makoto Ohashi, et al.. (2018). A cancer-associated Epstein-Barr virus BZLF1 promoter variant enhances lytic infection. PLoS Pathogens. 14(7). e1007179–e1007179. 76 indexed citations
12.
Romero-Masters, James C., Makoto Ohashi, Mitchell Hayes, et al.. (2018). An EBNA3C-deleted Epstein-Barr virus (EBV) mutant causes B-cell lymphomas with delayed onset in a cord blood-humanized mouse model. PLoS Pathogens. 14(8). e1007221–e1007221. 24 indexed citations
13.
Kraus, Richard J., Xianming Yu, Tawin Iempridee, et al.. (2017). Hypoxia-inducible factor-1α plays roles in Epstein-Barr virus’s natural life cycle and tumorigenesis by inducing lytic infection through direct binding to the immediate-early BZLF1 gene promoter. PLoS Pathogens. 13(6). e1006404–e1006404. 57 indexed citations
14.
Zumwalde, Nicholas A., Akshat Sharma, Shidong Ma, et al.. (2017). Adoptively transferred Vγ9Vδ2 T cells show potent antitumor effects in a preclinical B cell lymphomagenesis model. JCI Insight. 2(13). 59 indexed citations
15.
Tsai, Ming‐Han, James C. Romero-Masters, Erik A. Ranheim, et al.. (2017). Latent Membrane Protein 1 (LMP1) and LMP2A Collaborate To Promote Epstein-Barr Virus-Induced B Cell Lymphomas in a Cord Blood-Humanized Mouse Model but Are Not Essential. Journal of Virology. 91(7). 39 indexed citations
16.
Bilger, Andrea, Shidong Ma, Dhananjay M. Nawandar, et al.. (2017). Leflunomide/teriflunomide inhibit Epstein-Barr virus (EBV)-induced lymphoproliferative disease and lytic viral replication. Oncotarget. 8(27). 44266–44280. 61 indexed citations
17.
Sharma, Akshat, et al.. (2016). Human Invariant NKT Cells Induce IL-1β Secretion by Peripheral Blood Monocytes via a P2X7-Independent Pathway. The Journal of Immunology. 197(6). 2455–2464. 15 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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