Mitchell Hayes

1.2k total citations
29 papers, 925 citations indexed

About

Mitchell Hayes is a scholar working on Oncology, Immunology and Epidemiology. According to data from OpenAlex, Mitchell Hayes has authored 29 papers receiving a total of 925 indexed citations (citations by other indexed papers that have themselves been cited), including 20 papers in Oncology, 11 papers in Immunology and 10 papers in Epidemiology. Recurrent topics in Mitchell Hayes's work include Viral-associated cancers and disorders (17 papers), Immune Cell Function and Interaction (8 papers) and Cytomegalovirus and herpesvirus research (7 papers). Mitchell Hayes is often cited by papers focused on Viral-associated cancers and disorders (17 papers), Immune Cell Function and Interaction (8 papers) and Cytomegalovirus and herpesvirus research (7 papers). Mitchell Hayes collaborates with scholars based in United States, Germany and United Kingdom. Mitchell Hayes's co-authors include Bill Sugden, Wolfgang Hammerschmidt, Takanobu Tagawa, Eric Johannsen, Dominik Lutter, Jonathan Hoser, Mickaël Bouvet, Maximilian Hastreiter, Andreas Moosmann and Manuel Albanese and has published in prestigious journals such as Proceedings of the National Academy of Sciences, The Journal of Experimental Medicine and Journal of Clinical Oncology.

In The Last Decade

Mitchell Hayes

28 papers receiving 904 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Mitchell Hayes United States 16 614 258 242 242 231 29 925
Katharina Bernhardt Germany 13 621 1.0× 249 1.0× 278 1.1× 285 1.2× 175 0.8× 17 959
Juan Carlos Ramos United States 13 650 1.1× 141 0.5× 209 0.9× 164 0.7× 159 0.7× 32 908
Gianna Ballon United States 12 462 0.8× 318 1.2× 327 1.4× 215 0.9× 82 0.4× 19 954
F. A. Grässer Germany 11 703 1.1× 220 0.9× 314 1.3× 210 0.9× 239 1.0× 15 1.0k
Dhananjay M. Nawandar United States 11 424 0.7× 156 0.6× 143 0.6× 186 0.8× 70 0.3× 13 595
Takuro Watanabe Japan 16 232 0.4× 185 0.7× 172 0.7× 258 1.1× 93 0.4× 43 858
Fabiana Rizzo Italy 16 224 0.4× 498 1.9× 249 1.0× 148 0.6× 170 0.7× 29 955
Sandra A.W.M. Verkuijlen Netherlands 16 496 0.8× 81 0.3× 168 0.7× 180 0.7× 142 0.6× 27 803
Eleanore Lambley Australia 8 357 0.6× 314 1.2× 149 0.6× 107 0.4× 60 0.3× 10 683

Countries citing papers authored by Mitchell Hayes

Since Specialization
Citations

This map shows the geographic impact of Mitchell Hayes's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Mitchell Hayes with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Mitchell Hayes more than expected).

Fields of papers citing papers by Mitchell Hayes

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Mitchell Hayes. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Mitchell Hayes. The network helps show where Mitchell Hayes may publish in the future.

Co-authorship network of co-authors of Mitchell Hayes

This figure shows the co-authorship network connecting the top 25 collaborators of Mitchell Hayes. A scholar is included among the top collaborators of Mitchell Hayes based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Mitchell Hayes. Mitchell Hayes is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhang, Liyan, et al.. (2025). MmuPV1 E7 promotes phenotypes associated with “high-risk” HPV infection in mouse keratinocytes. Journal of Virology. 99(11). e0109725–e0109725. 1 indexed citations
2.
3.
Bristol, Jillian A., Scott E. Nelson, Makoto Ohashi, et al.. (2024). Latent Epstein-Barr virus infection collaborates with Myc over-expression in normal human B cells to induce Burkitt-like Lymphomas in mice. PLoS Pathogens. 20(4). e1012132–e1012132. 4 indexed citations
4.
Wang, Wei, Denis Lee, Mitchell Hayes, et al.. (2022). Stress Keratin 17 Expression in Head and Neck Cancer Contributes to Immune Evasion and Resistance to Immune-Checkpoint Blockade. Clinical Cancer Research. 28(13). 2953–2968. 22 indexed citations
5.
Bristol, Jillian A., Joshua Brand, Makoto Ohashi, et al.. (2022). Reduced IRF4 expression promotes lytic phenotype in Type 2 EBV-infected B cells. PLoS Pathogens. 18(4). e1010453–e1010453. 15 indexed citations
6.
Singh, Deo R., Scott E. Nelson, Jillian A. Bristol, et al.. (2022). Type 1 and Type 2 Epstein-Barr viruses induce proliferation, and inhibit differentiation, in infected telomerase-immortalized normal oral keratinocytes. PLoS Pathogens. 18(10). e1010868–e1010868. 5 indexed citations
7.
8.
Sugden, Arthur U., Mitchell Hayes, & Bill Sugden. (2021). How Epstein–Barr Virus and Kaposi’s Sarcoma-Associated Herpesvirus Are Maintained Together to Transform the Same B-Cell. Viruses. 13(8). 1478–1478. 5 indexed citations
9.
Ding, Lin, Qian Li, Jayati Chakrabarti, et al.. (2020). MiR130b from Schlafen4 + MDSCs stimulates epithelial proliferation and correlates with preneoplastic changes prior to gastric cancer. Gut. 69(10). 1750–1761. 49 indexed citations
10.
Wang, Wei, Aayushi Uberoi, Megan E. Spurgeon, et al.. (2020). Stress keratin 17 enhances papillomavirus infection-induced disease by downregulating T cell recruitment. PLoS Pathogens. 16(1). e1008206–e1008206. 32 indexed citations
11.
Li, Chunrong, James C. Romero-Masters, Shane M. Huebner, et al.. (2020). EBNA2-deleted Epstein-Barr virus (EBV) isolate, P3HR1, causes Hodgkin-like lymphomas and diffuse large B cell lymphomas with type II and Wp-restricted latency types in humanized mice. PLoS Pathogens. 16(6). e1008590–e1008590. 21 indexed citations
12.
Hayes, Mitchell, David Schlundt, Kemberlee Bonnet, et al.. (2019). Tales from the Trips: A Qualitative Study of Timely Recognition, Treatment, and Transfer of Emergency Department Patients with Acute Ischemic Stroke. Journal of Stroke and Cerebrovascular Diseases. 28(5). 1219–1228. 10 indexed citations
13.
Hayes, Mitchell, et al.. (2018). CAGE-seq analysis of Epstein-Barr virus lytic gene transcription: 3 kinetic classes from 2 mechanisms. PLoS Pathogens. 14(6). e1007114–e1007114. 38 indexed citations
14.
Bristol, Jillian A., Carrie B. Coleman, Makoto Ohashi, et al.. (2018). A cancer-associated Epstein-Barr virus BZLF1 promoter variant enhances lytic infection. PLoS Pathogens. 14(7). e1007179–e1007179. 76 indexed citations
15.
Albanese, Manuel, Takanobu Tagawa, Mickaël Bouvet, et al.. (2016). Epstein–Barr virus microRNAs reduce immune surveillance by virus-specific CD8 + T cells. Proceedings of the National Academy of Sciences. 113(42). E6467–E6475. 127 indexed citations
16.
Tagawa, Takanobu, Manuel Albanese, Mickaël Bouvet, et al.. (2016). Epstein-Barr viral miRNAs inhibit antiviral CD4+ T cell responses targeting IL-12 and peptide processing. The Journal of Experimental Medicine. 213(10). 2065–2080. 105 indexed citations
17.
Hayes, Mitchell, et al.. (2014). Multiple functions are mediated by the miRNAs of Epstein-Barr virus. Current Opinion in Virology. 7. 61–65. 34 indexed citations
18.
Alvarado, Michael, et al.. (2013). Patient preferences regarding intraoperative versus external beam radiotherapy following breast-conserving surgery. Breast Cancer Research and Treatment. 143(1). 135–140. 40 indexed citations
19.
Vereide, David, Eri Seto, Mitchell Hayes, et al.. (2013). Epstein–Barr virus maintains lymphomas via its miRNAs. Oncogene. 33(10). 1258–1264. 140 indexed citations
20.
Burton, R C, et al.. (1983). Effects of age, gender, and cigarette smoking on human immunoregulatory T-cell subsets: establishment of normal ranges and comparison with patients with colorectal cancer and multiple sclerosis.. PubMed. 1(3). 216–23. 38 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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