S.H. Madsen

469 total citations
11 papers, 388 citations indexed

About

S.H. Madsen is a scholar working on Rheumatology, Cancer Research and Pharmacology. According to data from OpenAlex, S.H. Madsen has authored 11 papers receiving a total of 388 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Rheumatology, 6 papers in Cancer Research and 2 papers in Pharmacology. Recurrent topics in S.H. Madsen's work include Protease and Inhibitor Mechanisms (5 papers), Osteoarthritis Treatment and Mechanisms (5 papers) and Inflammatory mediators and NSAID effects (2 papers). S.H. Madsen is often cited by papers focused on Protease and Inhibitor Mechanisms (5 papers), Osteoarthritis Treatment and Mechanisms (5 papers) and Inflammatory mediators and NSAID effects (2 papers). S.H. Madsen collaborates with scholars based in Denmark, Australia and United States. S.H. Madsen's co-authors include M.A. Karsdal, Claus Christiansen, Amanda Fosang, Kim Henriksen, Anne‐Christine Bay‐Jensen, E.U. Sumer, Per Qvist, Christian E. Pedersen, A.V. Neutzsky-Wulff and Sarah Juel Paulsen and has published in prestigious journals such as Scientific Reports, Osteoarthritis and Cartilage and Arthritis Research & Therapy.

In The Last Decade

S.H. Madsen

11 papers receiving 384 citations

Peers

Countries citing papers authored by S.H. Madsen

Since Specialization

Citations

This map shows the geographic impact of S.H. Madsen's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by S.H. Madsen with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites S.H. Madsen more than expected).

Fields of papers citing papers by S.H. Madsen

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by S.H. Madsen. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by S.H. Madsen. The network helps show where S.H. Madsen may publish in the future.

Co-authorship network of co-authors of S.H. Madsen

This figure shows the co-authorship network connecting the top 25 collaborators of S.H. Madsen. A scholar is included among the top collaborators of S.H. Madsen based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with S.H. Madsen. S.H. Madsen is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

Sort: Min cites: Since: Top N: Style:

11 of 11 papers shown

#	Title	Journal	Authors	Indexed citations
1	Inner histopathologic changes and disproportionate zone volumes in foetal growth plates following gestational hypoglycaemia in rats	Scientific Reports	Ingrid Brück Bøgh, Jette Nowak et al.	1
2	Interleukin‐15‐activated natural killer cells kill autologous osteoclasts via LFA‐1, DNAM‐1 and TRAIL, and inhibit osteoclast‐mediated bone erosion in vitro	Immunology	Shan Feng, S.H. Madsen et al.	38
3	Glucocorticoids exert context-dependent effects on cells of the joint in vitro	Steroids	S.H. Madsen, Kim Vietz Andreassen et al.	7
4	Investigation of the direct effects of salmon calcitonin on human osteoarthritic chondrocytes	BMC Musculoskeletal Disorders	S.H. Madsen, Toni Segovia‐Silvestre et al.	41
5	Characterization of an Ex vivo Femoral Head Model Assessed by Markers of Bone and Cartilage Turnover	Cartilage	S.H. Madsen, Søren T. Christensen et al.	16
6	Aggrecanase- and matrix metalloproteinase-mediated aggrecan degradation is associated with different molecular characteristics of aggrecan and separated in timeex vivo	Biomarkers	S.H. Madsen, E.U. Sumer et al.	25
7	Suppression of MMP activity in bovine cartilage explants cultures has little if any effect on the release of aggrecanase-derived aggrecan fragments	BMC Research Notes	Pingping Chen, Anne‐Christine Bay‐Jensen et al.	29
8	268 MAPKS ARE ESSENTIAL UPSTREAM SIGNALING PATHWAYS IN PROTEOLYTIC DEGRADATION - DIVERGENCE IN PATHWAYS LEADING TO AGGRECANASE AND MMP MEDIATED ARTICULAR CARTILAGE DEGRADATION	Osteoarthritis and Cartilage	B.C. Søndergaard, S.H. Madsen et al.	4
9	Cartilage degradation is fully reversible in the presence of aggrecanase but not matrix metalloproteinase activity	Arthritis Research & Therapy	M.A. Karsdal, S.H. Madsen et al.	150
10	Patients with rheumatoid arthritis have an altered circulatory aggrecan profile	BMC Musculoskeletal Disorders	E.U. Sumer, Gert Hein et al.	18
11	Induction of increased cAMP levels in articular chondrocytes blocks matrix metalloproteinase–mediated cartilage degradation, but not aggrecanase‐mediated cartilage degradation	Arthritis & Rheumatism	M.A. Karsdal, E.U. Sumer et al.	59

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact