Zelda Plener

553 total citations · 1 hit paper
7 papers, 410 citations indexed

About

Zelda Plener is a scholar working on Rheumatology, Molecular Biology and Immunology and Allergy. According to data from OpenAlex, Zelda Plener has authored 7 papers receiving a total of 410 indexed citations (citations by other indexed papers that have themselves been cited), including 6 papers in Rheumatology, 3 papers in Molecular Biology and 3 papers in Immunology and Allergy. Recurrent topics in Zelda Plener's work include Osteoarthritis Treatment and Mechanisms (6 papers), Cell Adhesion Molecules Research (3 papers) and Inflammatory mediators and NSAID effects (2 papers). Zelda Plener is often cited by papers focused on Osteoarthritis Treatment and Mechanisms (6 papers), Cell Adhesion Molecules Research (3 papers) and Inflammatory mediators and NSAID effects (2 papers). Zelda Plener collaborates with scholars based in Belgium and France. Zelda Plener's co-authors include Dominique de Seny, Federica Ciregia, Michel Malaise, Céline Deroyer, Olivier Malaise, Sophie Neuville, Edith Charlier, Gaël Cobraiville, Olivier Malaise and Philippe Gillet and has published in prestigious journals such as Biochemical Journal, Cellular and Molecular Life Sciences and Biochemical Pharmacology.

In The Last Decade

Zelda Plener

7 papers receiving 407 citations

Hit Papers

Chondrocyte dedifferentiation and osteoarthritis (OA) 2019 2026 2021 2023 2019 100 200 300

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Zelda Plener Belgium 5 282 181 115 61 48 7 410
Brian Wu Canada 7 231 0.8× 207 1.1× 93 0.8× 77 1.3× 63 1.3× 9 495
Nobuho Tanaka Japan 10 215 0.8× 126 0.7× 90 0.8× 49 0.8× 71 1.5× 15 371
Youngnim Shin South Korea 8 225 0.8× 140 0.8× 81 0.7× 70 1.1× 40 0.8× 10 385
Kirsty L. Culley United States 14 271 1.0× 242 1.3× 129 1.1× 88 1.4× 55 1.1× 20 489
Shanxing Zhang China 12 181 0.6× 174 1.0× 59 0.5× 56 0.9× 119 2.5× 22 465
E. Blaney Davidson Netherlands 7 244 0.9× 169 0.9× 39 0.3× 62 1.0× 54 1.1× 17 335
Helen Quasnichka United Kingdom 7 228 0.8× 155 0.9× 44 0.4× 71 1.2× 88 1.8× 11 412
Yoko Hosaka Japan 9 342 1.2× 221 1.2× 147 1.3× 125 2.0× 114 2.4× 15 549

Countries citing papers authored by Zelda Plener

Since Specialization
Citations

This map shows the geographic impact of Zelda Plener's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Zelda Plener with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Zelda Plener more than expected).

Fields of papers citing papers by Zelda Plener

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Zelda Plener. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Zelda Plener. The network helps show where Zelda Plener may publish in the future.

Co-authorship network of co-authors of Zelda Plener

This figure shows the co-authorship network connecting the top 25 collaborators of Zelda Plener. A scholar is included among the top collaborators of Zelda Plener based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Zelda Plener. Zelda Plener is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

7 of 7 papers shown
1.
Paulissen, Geneviève, Olivier Malaise, Céline Deroyer, et al.. (2025). JAK inhibitors alleviate metabolic dysregulation, inflammation and fibrosis in osteoarthritis: insights from human joint cells and synovium. Lara D. Veeken. 64(10). 5539–5550. 1 indexed citations
2.
Deroyer, Céline, Christophe Poulet, Geneviève Paulissen, et al.. (2022). CEMIP (KIAA1199) regulates inflammation, hyperplasia and fibrosis in osteoarthritis synovial membrane. Cellular and Molecular Life Sciences. 79(5). 260–260. 24 indexed citations
3.
Ciregia, Federica, Céline Deroyer, Gaël Cobraiville, et al.. (2021). Modulation of αVβ6 integrin in osteoarthritis-related synovitis and the interaction with VTN(381–397 a.a.) competing for TGF-β1 activation. Experimental & Molecular Medicine. 53(2). 210–222. 31 indexed citations
4.
Relić, Biserka, Céline Deroyer, Olivier Malaise, et al.. (2021). TFEB phosphorylation on Serine 211 is induced by autophagy in human synovial fibroblasts and by p62/SQSTM1 overexpression in HEK293 cells. Biochemical Journal. 478(16). 3145–3155. 2 indexed citations
5.
Malaise, Olivier, Geneviève Paulissen, Céline Deroyer, et al.. (2021). Influence of Glucocorticoids on Cellular Senescence Hallmarks in Osteoarthritic Fibroblast-like Synoviocytes. Journal of Clinical Medicine. 10(22). 5331–5331. 6 indexed citations
6.
Charlier, Edith, Céline Deroyer, Sophie Neuville, et al.. (2020). Toward diagnostic relevance of the αVβ5, αVβ3, and αVβ6 integrins in OA: expression within human cartilage and spinal osteophytes. Bone Research. 8(1). 35–35. 6 indexed citations
7.
Charlier, Edith, Céline Deroyer, Federica Ciregia, et al.. (2019). Chondrocyte dedifferentiation and osteoarthritis (OA). Biochemical Pharmacology. 165. 49–65. 340 indexed citations breakdown →

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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