Serena Grossi

1.7k total citations
33 papers, 1.2k citations indexed

About

Serena Grossi is a scholar working on Molecular Biology, Physiology and Organic Chemistry. According to data from OpenAlex, Serena Grossi has authored 33 papers receiving a total of 1.2k indexed citations (citations by other indexed papers that have themselves been cited), including 23 papers in Molecular Biology, 15 papers in Physiology and 7 papers in Organic Chemistry. Recurrent topics in Serena Grossi's work include Lysosomal Storage Disorders Research (13 papers), Inflammasome and immune disorders (8 papers) and Carbohydrate Chemistry and Synthesis (7 papers). Serena Grossi is often cited by papers focused on Lysosomal Storage Disorders Research (13 papers), Inflammasome and immune disorders (8 papers) and Carbohydrate Chemistry and Synthesis (7 papers). Serena Grossi collaborates with scholars based in Italy, Switzerland and United States. Serena Grossi's co-authors include Hans‐Dietmar Beer, Lars E. French, Mirella Filocamo, Michela Di Filippo, Paulina Hennig, Gabriele Fenini, Stefano Regis, Sabine Werner, Roberta Biancheri and Gerhard E. Strittmatter and has published in prestigious journals such as SHILAP Revista de lepidopterología, PLoS ONE and The Plant Journal.

In The Last Decade

Serena Grossi

33 papers receiving 1.2k citations

Peers

Serena Grossi

PHYSI

IMMUN

EPIDE

Hyun Tae Kang South Korea

Albin Pourtier France

Won Hee Jang South Korea

Dorit Avni Israel

Xufeng Qi China

Mingliang Chen China

Michelle Lindström United States

Cheryl L. Clauson United States

Ali Saleh Canada

Achim H.‐P. Krauss United States

Hyun Tae Kang South Korea

Serena Grossi

872 ×1.2

532 ×1.6

PHYSI

148 ×0.7

IMMUN

121 ×0.7

447 ×3.0

EPIDE

Citations per year, relative to Serena Grossi Serena Grossi (= 1×) peers Hyun Tae Kang

Countries citing papers authored by Serena Grossi

Since Specialization

Citations

This map shows the geographic impact of Serena Grossi's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Serena Grossi with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Serena Grossi more than expected).

Fields of papers citing papers by Serena Grossi

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Serena Grossi. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Serena Grossi. The network helps show where Serena Grossi may publish in the future.

Co-authorship network of co-authors of Serena Grossi

This figure shows the co-authorship network connecting the top 25 collaborators of Serena Grossi. A scholar is included among the top collaborators of Serena Grossi based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Serena Grossi. Serena Grossi is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Sand, Jennifer, Gabriele Fenini, Serena Grossi, et al.. (2019). The NLRP1 Inflammasome Pathway Is Silenced in Cutaneous Squamous Cell Carcinoma. Journal of Investigative Dermatology. 139(8). 1788–1797.e6. 23 indexed citations

Grossi, Serena, Gabriele Fenini, Paulina Hennig, Michela Di Filippo, & Hans‐Dietmar Beer. (2019). Generation of Knockout Human Primary Keratinocytes by CRISPR/Cas9. Methods in molecular biology. 2109. 125–145. 8 indexed citations

Grossi, Serena, Gabriele Fenini, Tobias Kockmann, et al.. (2019). Inactivation of the Cytoprotective Major Vault Protein by Caspase-1 and -9 in Epithelial Cells during Apoptosis. Journal of Investigative Dermatology. 140(7). 1335–1345.e10. 23 indexed citations

Fenini, Gabriele, Serena Grossi, Emmanuel Contassot, et al.. (2018). Genome Editing of Human Primary Keratinocytes by CRISPR/Cas9 Reveals an Essential Role of the NLRP1 Inflammasome in UVB Sensing. Journal of Investigative Dermatology. 138(12). 2644–2652. 88 indexed citations

Hennig, Paulina, et al.. (2018). The Crosstalk between Nrf2 and Inflammasomes. International Journal of Molecular Sciences. 19(2). 562–562. 195 indexed citations

Frati, Giacomo, Vasco Meneghini, Silvia De Cicco, et al.. (2018). Human iPSC-based models highlight defective glial and neuronal differentiation from neural progenitor cells in metachromatic leukodystrophy. Cell Death and Disease. 9(6). 698–698. 34 indexed citations

Hiebert, Paul, Mateusz S. Wietecha, Michael Cangkrama, et al.. (2018). Nrf2-Mediated Fibroblast Reprogramming Drives Cellular Senescence by Targeting the Matrisome. Developmental Cell. 46(2). 145–161.e10. 136 indexed citations

Fenini, Gabriele, Serena Grossi, Samuel Gehrke, et al.. (2017). The p38 Mitogen-Activated Protein Kinase Critically Regulates Human Keratinocyte Inflammasome Activation. Journal of Investigative Dermatology. 138(6). 1380–1390. 30 indexed citations

Grossi, Serena, et al.. (2016). MLPA-based approach for initial and simultaneous detection of GBA deletions and recombinant alleles in patients affected by Gaucher Disease. Molecular Genetics and Metabolism. 119(4). 329–337. 4 indexed citations

10.

Sestito, Simona, Mirella Filocamo, Renato Cantaffa, et al.. (2016). Norrbottnian clinical variant of Gaucher disease in Southern Italy. Journal of Human Genetics. 62(4). 507–511. 4 indexed citations

11.

Sollberger, Gabriel, Gerhard E. Strittmatter, Serena Grossi, et al.. (2015). Caspase-1 Activity Is Required for UVB-Induced Apoptosis of Human Keratinocytes. Journal of Investigative Dermatology. 135(5). 1395–1404. 66 indexed citations

12.

Buoso, Erica, Fabrizio Biundo, Cristina Lanni, et al.. (2013). Modulation of Rack-1/PKCβII Signalling By Soluble AβPPα in SH-SY5Y Cells. Current Alzheimer Research. 10(7). 697–705. 18 indexed citations

13.

Lorioli, Laura, Martina Cesani, Stefano Regis, et al.. (2013). Critical issues for the proper diagnosis of Metachromatic Leukodystrophy. Gene. 537(2). 348–351. 16 indexed citations

14.

Grossi, Serena, Marta Deganuto, Camillo Rosano, et al.. (2013). Functional analysis of 11 novel GBA alleles. European Journal of Human Genetics. 22(4). 511–516. 41 indexed citations

15.

Regis, Stefano, Fabio Corsolini, Serena Grossi, et al.. (2013). Restoration of the Normal Splicing Pattern of the PLP1 Gene by Means of an Antisense Oligonucleotide Directed against an Exonic Mutation. PLoS ONE. 8(9). e73633–e73633. 12 indexed citations

16.

Biancheri, Roberta, Serena Grossi, Stefano Regis, et al.. (2013). Further genotype–phenotype correlation emerging from two families with PLP1 exon 4 skipping. Clinical Genetics. 85(3). 267–272. 5 indexed citations

17.

Grossi, Serena, Stefano Regis, Roberta Biancheri, et al.. (2011). Molecular Genetic Analysis of the PLP1 Gene in 38 Families with PLP1-related disorders: Identification and Functional Characterization of 11 Novel PLP1 Mutations. Orphanet Journal of Rare Diseases. 6(1). 40–40. 32 indexed citations

18.

Regis, Stefano, Serena Grossi, Fabio Corsolini, Roberta Biancheri, & Mirella Filocamo. (2009). PLP1 gene duplication causes overexpression and alteration of the PLP/DM20 splicing balance in fibroblasts from Pelizaeus–Merzbacher disease patients. Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease. 1792(6). 548–554. 23 indexed citations

19.

Grossi, Serena, Stefano Regis, Camillo Rosano, et al.. (2008). Molecular analysis ofARSAandPSAPgenes in twenty-one Italian patients with metachromatic leukodystrophy: identification and functional characterization of 11 novelARSAalleles. Human Mutation. 29(11). E220–E230. 24 indexed citations

20.

Stroppiano, Marina, Fabio Corsolini, Maja Di Rocco, et al.. (2004). Screening of 25 Italian patients with Niemann-Pick a reveals fourteen new mutations, one common and thirteen private, inSMPD1. Human Mutation. 24(1). 105–105. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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