Seda Koyuncu

941 total citations
20 papers, 617 citations indexed

About

Seda Koyuncu is a scholar working on Molecular Biology, Aging and Cellular and Molecular Neuroscience. According to data from OpenAlex, Seda Koyuncu has authored 20 papers receiving a total of 617 indexed citations (citations by other indexed papers that have themselves been cited), including 14 papers in Molecular Biology, 7 papers in Aging and 6 papers in Cellular and Molecular Neuroscience. Recurrent topics in Seda Koyuncu's work include Genetics, Aging, and Longevity in Model Organisms (7 papers), Endoplasmic Reticulum Stress and Disease (5 papers) and Genetic Neurodegenerative Diseases (5 papers). Seda Koyuncu is often cited by papers focused on Genetics, Aging, and Longevity in Model Organisms (7 papers), Endoplasmic Reticulum Stress and Disease (5 papers) and Genetic Neurodegenerative Diseases (5 papers). Seda Koyuncu collaborates with scholars based in Germany, Hungary and Spain. Seda Koyuncu's co-authors include David Vı́lchez, Ricardo Gutiérrez-García, Azra Fatima, Isabel Sáez, Hyun Ju Lee, Christoph Dieterich, Hyun Ju Lee, Hyun Ju Lee, Rute Loureiro and Marcus Krüeger and has published in prestigious journals such as Nature, Nature Communications and SHILAP Revista de lepidopterología.

In The Last Decade

Seda Koyuncu

19 papers receiving 611 citations

Peers

Seda Koyuncu

AGING

CMN

PHYSI

Girish Harinath United States

Laure Granger France

Ilija Melentijevic United States

Anthony S. Castanza United States

Yon Ju Ji United States

David B. Rhee United States

Ricardo Gutiérrez-García Germany

J. Gavin Daigle United States

James Jiayuan Tong United States

Siôn L. Williams United States

Girish Harinath United States

Seda Koyuncu

487 ×1.1

174 ×1.3

AGING

49 ×0.4

49 ×0.5

CMN

116 ×1.1

PHYSI

Citations per year, relative to Seda Koyuncu Seda Koyuncu (= 1×) peers Girish Harinath

Countries citing papers authored by Seda Koyuncu

Since Specialization

Citations

This map shows the geographic impact of Seda Koyuncu's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Seda Koyuncu with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Seda Koyuncu more than expected).

Fields of papers citing papers by Seda Koyuncu

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Seda Koyuncu. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Seda Koyuncu. The network helps show where Seda Koyuncu may publish in the future.

Co-authorship network of co-authors of Seda Koyuncu

This figure shows the co-authorship network connecting the top 25 collaborators of Seda Koyuncu. A scholar is included among the top collaborators of Seda Koyuncu based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Seda Koyuncu. Seda Koyuncu is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Koyuncu, Seda, et al.. (2025). The aging factor EPS8 induces disease-related protein aggregation through RAC signaling hyperactivation. Nature Aging. 5(9). 1750–1770. 2 indexed citations

Zhang, William, et al.. (2025). C9orf72 ALS ‐causing mutations lead to mislocalization and aggregation of nucleoporin Nup107 into stress granules. FEBS Letters. 599(21). 3047–3065. 1 indexed citations

Li, Xuexin, Ivó H. Hernández, Seda Koyuncu, et al.. (2024). The anti-leprosy drug clofazimine reduces polyQ toxicity through activation of PPARγ. EBioMedicine. 103. 105124–105124.

Loureiro, Rute, et al.. (2024). ALS-FUS mutations cause abnormal PARylation and histone H1.2 interaction, leading to pathological changes. Cell Reports. 43(8). 114626–114626. 8 indexed citations

Gutiérrez-García, Ricardo, et al.. (2023). G3BP1-dependent mechanism suppressing protein aggregation in Huntington’s models and its demise upon stress granule assembly. Human Molecular Genetics. 32(10). 1607–1621. 15 indexed citations

Koyuncu, Seda, et al.. (2023). Neuroprotective effects of hepatoma-derived growth factor in models of Huntington’s disease. Life Science Alliance. 6(11). e202302018–e202302018. 2 indexed citations

Lee, Hyun Ju, et al.. (2023). Cold temperature extends longevity and prevents disease-related protein aggregation through PA28γ-induced proteasomes. Nature Aging. 3(5). 546–566. 35 indexed citations

Zhang, William, Seda Koyuncu, & David Vı́lchez. (2022). Insights Into the Links Between Proteostasis and Aging From C. elegans. SHILAP Revista de lepidopterología. 3. 854157–854157. 14 indexed citations

Koyuncu, Seda, Rute Loureiro, Hyun Ju Lee, et al.. (2021). Rewiring of the ubiquitinated proteome determines ageing in C. elegans. Nature. 596(7871). 285–290. 70 indexed citations

10.

Koyuncu, Seda, Isabel Sáez, Ricardo Gutiérrez-García, et al.. (2020). Author Correction: The ubiquitin ligase UBR5 suppresses proteostasis collapse in pluripotent stem cells from Huntington’s disease patients. Nature Communications. 11(1). 985–985. 2 indexed citations

11.

Mattos, Eduardo Preusser de, Jeanette F. Brunsting, Rob Bakels, et al.. (2020). DNAJB6, a Key Factor in Neuronal Sensitivity to Amyloidogenesis. Molecular Cell. 78(2). 346–358.e9. 55 indexed citations

12.

Lee, Hyun Ju, Seda Koyuncu, Milos Simic, et al.. (2019). Prostaglandin signals from adult germline stem cells delay somatic ageing of Caenorhabditis elegans. Nature Metabolism. 1(8). 790–810. 35 indexed citations

13.

Sáez, Isabel, Jennifer V. Gerbracht, Seda Koyuncu, et al.. (2019). The E3 ubiquitin ligase UBR5 interacts with the H/ACA ribonucleoprotein complex and regulates ribosomal RNA biogenesis in embryonic stem cells. FEBS Letters. 594(1). 175–188. 24 indexed citations

14.

Koyuncu, Seda, Isabel Sáez, Hyun Ju Lee, et al.. (2018). The ubiquitin ligase UBR5 suppresses proteostasis collapse in pluripotent stem cells from Huntington’s disease patients. Nature Communications. 9(1). 2886–2886. 80 indexed citations

15.

Sáez, Isabel, Seda Koyuncu, Ricardo Gutiérrez-García, Christoph Dieterich, & David Vı́lchez. (2018). Insights into the ubiquitin-proteasome system of human embryonic stem cells. Scientific Reports. 8(1). 4092–4092. 36 indexed citations

16.

Gutiérrez-García, Ricardo, et al.. (2017). Mechanisms of protein homeostasis (proteostasis) maintain stem cell identity in mammalian pluripotent stem cells. Cellular and Molecular Life Sciences. 75(2). 275–290. 40 indexed citations

17.

Koyuncu, Seda, Azra Fatima, Ricardo Gutiérrez-García, & David Vı́lchez. (2017). Proteostasis of Huntingtin in Health and Disease. International Journal of Molecular Sciences. 18(7). 1568–1568. 39 indexed citations

18.

Gutiérrez-García, Ricardo, Hyun Ju Lee, Seda Koyuncu, et al.. (2016). Somatic increase of CCT8 mimics proteostasis of human pluripotent stem cells and extends C. elegans lifespan. Nature Communications. 7(1). 13649–13649. 72 indexed citations

19.

Çimen, İsmail, Begüm Kocatürk, Seda Koyuncu, et al.. (2016). Prevention of atherosclerosis by bioactive palmitoleate through suppression of organelle stress and inflammasome activation. Science Translational Medicine. 8(358). 358ra126–358ra126. 83 indexed citations

20.

Koyuncu, Seda, et al.. (2015). Defining the General Principles of Stem Cell Aging: Lessons from Organismal Models. Current Stem Cell Reports. 1(3). 162–169. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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