Satyan Chintawar
- Molecular Biology
- Cellular and Molecular Neuroscience top 5%
- Neurology top 2%
- Developmental Neuroscience top 5%
- Immunology
- Co-authors
- M. Zameel CaderSally A. CowleyMassimo PandolfoJack P. AntelSarah E. NeweyStephen N. SansomChristian SchnellWalther Haenseler
- Topics
- Mitochondrial Function and Pathology (3 papers)Genetic Neurodegenerative Diseases (3 papers)Barrier Structure and Function Studies (3 papers)
- Journals
- Proceedings of the National Academy of SciencesJournal of Biological ChemistryJournal of Neuroscience
- Partner nations
- BelgiumUnited KingdomCanada
In The Last Decade
Satyan Chintawar
13 papers receiving 949 citations
Peers
Comparison fields: 5 of 77
- Molecular Biology 542
- Cellular and Molecular Neuroscience 327
- Neurology 306
- Developmental Neuroscience 175
- Immunology 148
Countries citing papers authored by Satyan Chintawar
This map shows the geographic impact of Satyan Chintawar's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Satyan Chintawar with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Satyan Chintawar more than expected).
Fields of papers citing papers by Satyan Chintawar
This network shows the impact of papers produced by Satyan Chintawar. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Satyan Chintawar. The network helps show where Satyan Chintawar may publish in the future.
Co-authorship network of co-authors of Satyan Chintawar
This figure shows the co-authorship network connecting the top 25 collaborators of Satyan Chintawar. A scholar is included among the top collaborators of Satyan Chintawar based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Satyan Chintawar. Satyan Chintawar is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 31 | |
| 2 | 49 | |
| 3 | 22 | |
| 4 | 366 | |
| 5 | 76 | |
| 6 | 54 | |
| 7 | 120 | |
| 8 | 2 | |
| 9 | 113 | |
| 10 | 57 | |
| 11 | 31 | |
| 12 | 7 | |
| 13 | 48 |
About Satyan Chintawar
Satyan Chintawar is a scholar working on Developmental Neuroscience, Neurology and Cellular and Molecular Neuroscience, having authored 13 papers that have together received 976 indexed citations. Recurring topics across this work include Mitochondrial Function and Pathology (3 papers), Genetic Neurodegenerative Diseases (3 papers) and Barrier Structure and Function Studies (3 papers). The work is most often cited by research in Developmental Neuroscience (175 citations), Neurology (306 citations) and Cellular and Molecular Neuroscience (327 citations). Satyan Chintawar has collaborated with scholars based in Belgium, United Kingdom and Canada. Frequent co-authors include M. Zameel Cader, Sally A. Cowley, Massimo Pandolfo, Jack P. Antel, Sarah E. Newey, Stephen N. Sansom, Christian Schnell, Walther Haenseler, Julian Buchrieser and Nicholas D. Allen. Their work appears in journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Neuroscience.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.