Satoru Nakata
- Dermatology top 0.5%
- Cell Biology top 5%
- Molecular Biology
- Epidemiology
- Genetics top 5%
- Co-authors
- Hirohiko AkamatsuKayoko MatsunagaSeiji HasegawaHiroshi MizutaniTakaaki YamadaHiroshi TanakaYu InoueNaoki Yamamoto
- Topics
- Skin Protection and Aging (12 papers)Dermatology and Skin Diseases (10 papers)melanin and skin pigmentation (10 papers)
- Cited by
- DermatologyCell BiologyGenetics
- Partner nations
- JapanUnited KingdomMexico
In The Last Decade
Satoru Nakata
57 papers receiving 1.3k citations
Peers
Comparison fields: 5 of 107
- Dermatology 581
- Cell Biology 383
- Molecular Biology 374
- Epidemiology 205
- Genetics 195
Countries citing papers authored by Satoru Nakata
This map shows the geographic impact of Satoru Nakata's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Satoru Nakata with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Satoru Nakata more than expected).
Fields of papers citing papers by Satoru Nakata
This network shows the impact of papers produced by Satoru Nakata. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Satoru Nakata. The network helps show where Satoru Nakata may publish in the future.
Co-authorship network of co-authors of Satoru Nakata
This figure shows the co-authorship network connecting the top 25 collaborators of Satoru Nakata. A scholar is included among the top collaborators of Satoru Nakata based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Satoru Nakata. Satoru Nakata is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 52 | |
| 2 | 18 | |
| 3 | 10 | |
| 4 | 26 | |
| 5 | 13 | |
| 6 | 17 | |
| 7 | 5 | |
| 8 | Stimulation of Noggin Signaling in Keratinocytes by an Extract of Mulberry (Morus alba L) | 2 |
| 9 | 1 | |
| 10 | 120 | |
| 11 | 20 | |
| 12 | 8 | |
| 13 | 16 | |
| 14 | 30 | |
| 15 | 31 | |
| 16 | 43 | |
| 17 | 16 | |
| 18 | 34 | |
| 19 | 44 | |
| 20 | Prediction of Human Skin Irritancy Using a Cultured Human Skin Model : Comparison of Chemical Application Procedures and Development of a Novel Chemical Application Procedure Using the Vitrolife-Skin^ Model | 1 |
About Satoru Nakata
Satoru Nakata is a scholar working on Dermatology, Aging and Urology, having authored 59 papers that have together received 1.4k indexed citations. Recurring topics across this work include Skin Protection and Aging (12 papers), Dermatology and Skin Diseases (10 papers) and melanin and skin pigmentation (10 papers). The work is most often cited by research in Dermatology (581 citations), Cell Biology (383 citations) and Genetics (195 citations). Satoru Nakata has collaborated with scholars based in Japan, United Kingdom and Mexico. Frequent co-authors include Hirohiko Akamatsu, Kayoko Matsunaga, Seiji Hasegawa, Hiroshi Mizutani, Takaaki Yamada, Hiroshi Tanaka, Yu Inoue, Naoki Yamamoto, Yasushi Date and Narifumi Akaza. Their work appears in journals such as Journal of Biological Chemistry, PLoS ONE and Macromolecules.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.