H. Iizuka

1.9k total citations
77 papers, 1.4k citations indexed

About

H. Iizuka is a scholar working on Molecular Biology, Dermatology and Cell Biology. According to data from OpenAlex, H. Iizuka has authored 77 papers receiving a total of 1.4k indexed citations (citations by other indexed papers that have themselves been cited), including 30 papers in Molecular Biology, 20 papers in Dermatology and 20 papers in Cell Biology. Recurrent topics in H. Iizuka's work include Skin and Cellular Biology Research (17 papers), Dermatology and Skin Diseases (11 papers) and Receptor Mechanisms and Signaling (6 papers). H. Iizuka is often cited by papers focused on Skin and Cellular Biology Research (17 papers), Dermatology and Skin Diseases (11 papers) and Receptor Mechanisms and Signaling (6 papers). H. Iizuka collaborates with scholars based in Japan, United States and Switzerland. H. Iizuka's co-authors include Akemi Ishida‐Yamamoto, Hidetoshi Takahashi, Yosuke Hashimoto, Hidekazu Tsuji, Hidenori Takahashi, Daniel Hohl, Robin A.J. Eady, H. Honda, T. Kobayashi and Naoyuki Taniguchi and has published in prestigious journals such as Journal of Investigative Dermatology, Journal of Histochemistry & Cytochemistry and British Journal of Dermatology.

In The Last Decade

H. Iizuka

76 papers receiving 1.3k citations

Peers

H. Iizuka

DERMA

IMMUN

PFM

Dagmar Födinger Austria

James F. Crish United States

Minao Furumura Japan

Takeshi Kono Japan

Francesca Mascia Italy

Armin Volz Germany

Toshifumi Nomura Japan

Annelii Ny Belgium

François Brégégère France

Peter Hevezi United States

Dagmar Födinger Austria

H. Iizuka

323 ×0.7

321 ×0.8

DERMA

135 ×0.4

359 ×1.0

IMMUN

132 ×0.6

PFM

Citations per year, relative to H. Iizuka H. Iizuka (= 1×) peers Dagmar Födinger

Countries citing papers authored by H. Iizuka

Since Specialization

Citations

This map shows the geographic impact of H. Iizuka's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by H. Iizuka with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites H. Iizuka more than expected).

Fields of papers citing papers by H. Iizuka

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by H. Iizuka. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by H. Iizuka. The network helps show where H. Iizuka may publish in the future.

Co-authorship network of co-authors of H. Iizuka

This figure shows the co-authorship network connecting the top 25 collaborators of H. Iizuka. A scholar is included among the top collaborators of H. Iizuka based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with H. Iizuka. H. Iizuka is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

Fujii, Mizue, Masaru Honma, Hiroyuki Takahashi, Akemi Ishida‐Yamamoto, & H. Iizuka. (2011). The nuclear factor kappa B p50 subunit and cortactin as markers to distinguish between keratoacanthoma and well-differentiated squamous cell carcinoma. Clinical and Experimental Dermatology. 36(7). 788–792. 8 indexed citations

Nakamura, Sachiko, Yoshio Makita, Akira Takagi, et al.. (2007). Hutchinson?Gilford progeria syndrome with severe skin calcinosis. Clinical and Experimental Dermatology. 32(5). 525–528. 2 indexed citations

Saeki, Hidehisa, H. Iizuka, Toshihide Akasaka, et al.. (2005). Prevalence of atopic dermatitis in Japanese elementary schoolchildren. British Journal of Dermatology. 152(1). 110–114. 61 indexed citations

Takahashi, Hidetoshi, Susumu Nakajima, Isao Sakata, Akemi Ishida‐Yamamoto, & H. Iizuka. (2005). ATX-S10(Na)-photodynamic therapy is less carcinogenic for mouse skin compared with ultraviolet B irradiation. British Journal of Dermatology. 153(6). 1182–1186. 10 indexed citations

Takahashi, Hidetoshi, Akemi Ishida‐Yamamoto, & H. Iizuka. (2004). Effects of bepotastine, cetirizine, fexofenadine, and olopatadine on histamine-induced wheal-and flare-response, sedation, and psychomotor performance. Clinical and Experimental Dermatology. 29(5). 526–532. 61 indexed citations

Wada, Takashi, Yuji Shirakata, Hidetoshi Takahashi, et al.. (2003). A Japanese case of segmental Darier's disease caused by mosaicism for the ATP2A2 mutation. British Journal of Dermatology. 149(1). 185–188. 28 indexed citations

Ishida‐Yamamoto, Akemi, et al.. (1997). Venous hemangioma. An immunohistochemical and ultrastructural study.. Acta Dermato Venereologica. 77(5). 382–384. 2 indexed citations

Ishida‐Yamamoto, Akemi, et al.. (1996). Immunoelectron microscopic analysis of cornified cell envelope formation in normal and psoriatic epidermis.. Journal of Histochemistry & Cytochemistry. 44(2). 167–175. 66 indexed citations

Ishida‐Yamamoto, Akemi, H. Iizuka, Motomu Manabe, et al.. (1995). Altered distribution of keratinization markers in epidermolytic hyperkeratosis. Archives of Dermatological Research. 287(8). 705–711. 21 indexed citations

10.

Takahashi, Hidetoshi, et al.. (1995). Repression of involucrin gene expression by transcriptional enhancer factor 1 (TEF-1). Archives of Dermatological Research. 287(8). 740–746. 12 indexed citations

11.

Ishida‐Yamamoto, Akemi, Daniel Hohl, Dennis R. Roop, H. Iizuka, & Robin A.J. Eady. (1993). Loricrin immunoreactivity in human skin: localization to specific granules (L-granules) in acrosyringia. Archives of Dermatological Research. 285(8). 491–498. 33 indexed citations

12.

Kobayashi, T., Mitsuhiro Matsumoto, H. Iizuka, Keiichiro Suzuki, & Naoyuki Taniguchi. (1991). Superoxide dismutase in psoriasis, squamous cell carcinoma and basal cell epithelioma: an immunohistochemical study. British Journal of Dermatology. 124(6). 555–559. 63 indexed citations

13.

Hashimoto, Yosuke, et al.. (1991). Reduced superoxide dismutase activity in UVB-induced hyperproliferative pig epidermis. Archives of Dermatological Research. 283(5). 317–320. 22 indexed citations

14.

Takahashi, Hidetoshi & H. Iizuka. (1991). Regulation of β2-adrenergic receptors in keratinocytes: glucocorticoids increase steady-state levels of receptor mRNA in foetal rat keratinizing epidermal cells (FRSK cells). British Journal of Dermatology. 124(4). 341–347. 13 indexed citations

15.

Iizuka, H., Hiroyuki Sakai, & Motoshi Kinouchi. (1990). Epidermal adenylate cyclase system is regulated by diacylglycerol-protein kinase C signal, but not by calcium signal. British Journal of Dermatology. 122(4). 459–467. 4 indexed citations

16.

Takahashi, Hidetoshi, H. Iizuka, & Makoto Katagiri. (1990). No evidence for the mutation of ras gene in psoriatic epidermis. Archives of Dermatological Research. 282(1). 8–11. 3 indexed citations

17.

Tsutsui, Masato, H. Iizuka, Akira Ohkawara, Kenji Adachi, & Tamotsu Kanzaki. (1987). Adenylate cyclase system of human trichilemmoma cell line. Archives of Dermatological Research. 279(8). 530–535. 3 indexed citations

18.

Iizuka, H., et al.. (1986). Topical application of potent glucocorticoids augments epidermal beta-adrenergic adenylate cyclase response in vivo. Acta Dermato Venereologica. 66(6). 491–496. 1 indexed citations

19.

Iizuka, H., et al.. (1981). Epinephrine-induced cyclic AMP accumulation in the psoriatic epidermis. Acta Dermato Venereologica. 61(5). 391–395. 23 indexed citations

20.

Iizuka, H.. (1977). Purification and properties of two forms of glucoamylase from Monascus Kaoliang nov. sp. F-1.. The Journal of General and Applied Microbiology. 23. 217–230. 4 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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