Satoko Shimbara
- Molecular Biology top 10%
- Pharmacology top 2%
- Genetics top 10%
- Immunology top 10%
- Physiology top 10%
- Co-authors
- Ichiro KudoMakoto MurakamiTerumi KambeHiroshi KuwataJay A. TischfieldMichelle V. WinsteadKumiko YoshiharaSachiko Oh‐ishi
- Topics
- Protein Kinase Regulation and GTPase Signaling (13 papers)Cell Adhesion Molecules Research (8 papers)Inflammatory mediators and NSAID effects (7 papers)
- Journals
- Journal of Biological ChemistryThe Journal of ImmunologyBiochemical and Biophysical Research Communications
- Partner nations
- JapanUnited StatesFrance
In The Last Decade
Satoko Shimbara
21 papers receiving 1.9k citations
Peers
Comparison fields: 5 of 105
- Molecular Biology 1.2k
- Pharmacology 498
- Genetics 326
- Immunology 289
- Physiology 254
Countries citing papers authored by Satoko Shimbara
This map shows the geographic impact of Satoko Shimbara's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Satoko Shimbara with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Satoko Shimbara more than expected).
Fields of papers citing papers by Satoko Shimbara
This network shows the impact of papers produced by Satoko Shimbara. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Satoko Shimbara. The network helps show where Satoko Shimbara may publish in the future.
Co-authorship network of co-authors of Satoko Shimbara
This figure shows the co-authorship network connecting the top 25 collaborators of Satoko Shimbara. A scholar is included among the top collaborators of Satoko Shimbara based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Satoko Shimbara. Satoko Shimbara is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 31 | |
| 2 | 63 | |
| 3 | 63 | |
| 4 | 71 | |
| 5 | 61 | |
| 6 | 47 | |
| 7 | 27 | |
| 8 | 155 | |
| 9 | 54 | |
| 10 | 149 | |
| 11 | 306 | |
| 12 | 145 | |
| 13 | 74 | |
| 14 | 326 | |
| 15 | 164 | |
| 16 | 17 | |
| 17 | 116 | |
| 18 | 17 | |
| 19 | 5 | |
| 20 | 4 |
About Satoko Shimbara
Satoko Shimbara is a scholar working on Immunology and Allergy, Pharmacology and Cell Biology, having authored 21 papers that have together received 1.9k indexed citations. Recurring topics across this work include Protein Kinase Regulation and GTPase Signaling (13 papers), Cell Adhesion Molecules Research (8 papers) and Inflammatory mediators and NSAID effects (7 papers). The work is most often cited by research in Biochemistry (216 citations), Pharmacology (498 citations) and Immunology and Allergy (161 citations). Satoko Shimbara has collaborated with scholars based in Japan, United States and France. Frequent co-authors include Ichiro Kudo, Makoto Murakami, Terumi Kambe, Hiroshi Kuwata, Jay A. Tischfield, Michelle V. Winstead, Kumiko Yoshihara, Sachiko Oh‐ishi, Michael H. Gelb and Gérard Lambeau. Their work appears in journals such as Journal of Biological Chemistry, The Journal of Immunology and Biochemical and Biophysical Research Communications.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.