Satoko Kumada
- Neurology top 5%
- Neurological disorders and treatments 13
- Autoimmune Neurological Disorders and Treatments 7
-
- Genetic Neurodegenerative Diseases 9
- Genetics top 5%
- Genetics and Neurodevelopmental Disorders 16
- Genetics top 5%
- Genetics and Neurodevelopmental Disorders 16
- Psychiatry and Mental health top 5%
- Epilepsy research and treatment 9
-
- Glycogen Storage Diseases and Myoclonus 12
-
- Lysosomal Storage Disorders Research 9
-
- Metabolism and Genetic Disorders 8
- Co-authors
- Masaharu HayashiHirotomo SaitsuNaomichi MatsumotoMitsuhiro KatoTakeshi MizuguchiKazuhiro OgataHitoshi OsakaAtsuo Fukuda
- Partner nations
- JapanUnited KingdomUnited States
In The Last Decade
Satoko Kumada
82 papers receiving 1.4k citations
Peers
Comparison fields: 5 of 74
- Neurology 260
- Cellular and Molecular Neuroscience 289
- Genetics 423
- Genetics 160
- Psychiatry and Mental health 202
Countries citing papers authored by Satoko Kumada
This map shows the geographic impact of Satoko Kumada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Satoko Kumada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Satoko Kumada more than expected).
Fields of papers citing papers by Satoko Kumada
This network shows the impact of papers produced by Satoko Kumada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Satoko Kumada. The network helps show where Satoko Kumada may publish in the future.
Co-authorship network
The 25 scholars most cited alongside Satoko Kumada, linked wherever they have co-authored with each other. Click a name or a connecting line to browse the papers they share.
All Works
| # | Work | ||
|---|---|---|---|
| 1 | 2025 | 0 | |
| 2 | 2024 | 1 | |
| 3 | 2024 | 1 | |
| 4 | 2024 | 1 | |
| 5 | 2024 | 1 | |
| 6 | 2024 | 0 | |
| 7 | 2024 | 5 | |
| 8 | 2024 | 1 | |
| 9 | 2023 | 1 | |
| 10 | 2023 | 1 | |
| 11 | 2022 | 1 | |
| 12 | 2022 | 28 | |
| 13 | 2021 | 2 | |
| 14 | 2021 | 16 | |
| 15 | 2020 | 13 | |
| 16 | 2019 | 3 | |
| 17 | 2002 | 33 | |
| 18 | 2001 | 46 | |
| 19 | 2000 | 26 | |
| 20 | 2000 | 26 |
About Satoko Kumada
Satoko Kumada is a scholar working on Neurology, Clinical Biochemistry and Rheumatology, having authored 86 papers that have together received 1.4k indexed citations. Recurring topics across this work include Genetics and Neurodevelopmental Disorders (16 papers), Neurological disorders and treatments (13 papers), Glycogen Storage Diseases and Myoclonus (12 papers), Lysosomal Storage Disorders Research (9 papers), Genetic Neurodegenerative Diseases (9 papers), Epilepsy research and treatment (9 papers), Metabolism and Genetic Disorders (8 papers) and Autoimmune Neurological Disorders and Treatments (7 papers). The work is most often cited by research in Neurology (260 citations), Cellular and Molecular Neuroscience (289 citations) and Genetics (423 citations). Satoko Kumada has collaborated with scholars based in Japan, United Kingdom and United States. Frequent co-authors include Masaharu Hayashi, Hirotomo Saitsu, Naomichi Matsumoto, Mitsuhiro Kato, Takeshi Mizuguchi, Kazuhiro Ogata, Hitoshi Osaka, Atsuo Fukuda, Tatsuro Kumada and Kiyomi Nishiyama. Their work appears in journals such as Nature Genetics, Neurology and Annals of Neurology.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.