Sascha Hagemann

532 citations
15 papers · 431 indexed · h-index 11
Topics
Protease and Inhibitor Mechanisms (4 papers)Glycosylation and Glycoproteins Research (2 papers)Cancer, Hypoxia, and Metabolism (2 papers)

In The Last Decade

Sascha Hagemann

14 papers receiving 425 citations

Peers

Sascha Hagemann
Comparison fields: 5 of 69
  • Molecular Biology 207
  • Cancer Research 141
  • Oncology 120
  • Immunology 91
  • Cell Biology 69
Replace Ana Schweiger with:
Ana Schweiger Slovenia
Mehdi Tnani United States
Eun Gene Sun South Korea
Qingdong Deng United States
Paul W. Hollenbach United States
Jean C. Tien United States
Annemieke Walker United Kingdom
Hong‐Yong Cui China
Meritxell Reverter Australia
Erin E. Witalison United States
Sascha Hagemann relative to Ana Schweiger Slovenia Ana Schweiger's profile →
Citations per field
00.5×4.2×
Ana Schweiger · 1×
Citations per year

Countries citing papers authored by Sascha Hagemann

Since Specialization
Citations

This map shows the geographic impact of Sascha Hagemann's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sascha Hagemann with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sascha Hagemann more than expected).

Fields of papers citing papers by Sascha Hagemann

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sascha Hagemann. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sascha Hagemann. The network helps show where Sascha Hagemann may publish in the future.

Co-authorship network of co-authors of Sascha Hagemann

This figure shows the co-authorship network connecting the top 25 collaborators of Sascha Hagemann. A scholar is included among the top collaborators of Sascha Hagemann based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sascha Hagemann. Sascha Hagemann is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

15 of 15 papers shown
#WorkIndexed citations
1 32
2 24
3 16
4 4
5 21
6 42
7 45
8 1
9 25
10 48
11 89
12 41
13 41
14 1
15 1

About Sascha Hagemann

Sascha Hagemann is a scholar working on Cancer Research, Cell Biology and Oncology, having authored 15 papers that have together received 431 indexed citations. Recurring topics across this work include Protease and Inhibitor Mechanisms (4 papers), Glycosylation and Glycoproteins Research (2 papers) and Cancer, Hypoxia, and Metabolism (2 papers). The work is most often cited by research in Cancer Research (141 citations), Immunology and Allergy (35 citations) and Oncology (120 citations). Sascha Hagemann has collaborated with scholars based in Germany, Netherlands and United Kingdom. Frequent co-authors include Christoph Peters, Thomas Reinheckel, Desmond J. Tobin, Nicole Maas‐Szabowski, Roland Schüle, Thomas Günther, Barbara Sitek, Hideo A. Baba, Wolfram von Bernstorff and Nico van Rooijen. Their work appears in journals such as Journal of Biological Chemistry, Journal of Cell Science and Cell Death and Differentiation.

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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