Sarah V. Williams

875 total citations
15 papers, 664 citations indexed

About

Sarah V. Williams is a scholar working on Molecular Biology, Surgery and Genetics. According to data from OpenAlex, Sarah V. Williams has authored 15 papers receiving a total of 664 indexed citations (citations by other indexed papers that have themselves been cited), including 11 papers in Molecular Biology, 4 papers in Surgery and 3 papers in Genetics. Recurrent topics in Sarah V. Williams's work include Fibroblast Growth Factor Research (5 papers), Epigenetics and DNA Methylation (5 papers) and Bladder and Urothelial Cancer Treatments (4 papers). Sarah V. Williams is often cited by papers focused on Fibroblast Growth Factor Research (5 papers), Epigenetics and DNA Methylation (5 papers) and Bladder and Urothelial Cancer Treatments (4 papers). Sarah V. Williams collaborates with scholars based in United Kingdom, Italy and Germany. Sarah V. Williams's co-authors include Margaret A. Knowles, Carolyn D. Hurst, Darren C. Tomlinson, Paul Ashford, Matilda Katan, Nethaji Thiyagarajan, Tom D. Bunney, Erica di Martino, Christine Orengo and Ludovico Sutto and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Nature Communications and Journal of Clinical Oncology.

In The Last Decade

Sarah V. Williams

15 papers receiving 654 citations

Peers

Countries citing papers authored by Sarah V. Williams

Since Specialization

Citations

This map shows the geographic impact of Sarah V. Williams's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sarah V. Williams with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sarah V. Williams more than expected).

Fields of papers citing papers by Sarah V. Williams

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sarah V. Williams. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sarah V. Williams. The network helps show where Sarah V. Williams may publish in the future.

Co-authorship network of co-authors of Sarah V. Williams

This figure shows the co-authorship network connecting the top 25 collaborators of Sarah V. Williams. A scholar is included among the top collaborators of Sarah V. Williams based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sarah V. Williams. Sarah V. Williams is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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15 of 15 papers shown

#	Work	Indexed citations
1	Small HSPs at the crossroad between protein aggregation, autophagy and unconventional secretion: clinical implications and potential therapeutic opportunities in the context of neurodegenerative diseases 2025 ·Frontiers in Cell and Developmental Biology ·(unknown), (unknown), (unknown), Sarah V. Williams, (unknown), Angeleen Fleming, Maurizio Renna	1
2	A method for the analysis of the oligomerization profile of the Huntington’s disease-associated, aggregation-prone mutant huntingtin protein by isopycnic ultracentrifugation 2024 ·Frontiers in Molecular Biosciences ·(unknown), Rosaria Di Martino, (unknown), (unknown), (unknown), (unknown), (unknown), Silvia Nuzzo, Marco Salvatore, Sarah V. Williams, (unknown), (unknown), M. D’Agostino, Francesca M. Pisani, Angeleen Fleming, Maurizio Renna	3
3	The HSPB1-p62/SQSTM1 functional complex regulates the unconventional secretion and transcellular spreading of the HD-associated mutant huntingtin protein 2023 ·Human Molecular Genetics ·(unknown), (unknown), Rosaria Di Martino, Silvia Nuzzo, Elena Polishchuk, (unknown), Sarah V. Williams, (unknown), (unknown), Roman Polishchuk, M. D’Agostino, Angeleen Fleming, Maurizio Renna	12
4	Chemokine receptor trafficking coordinates neutrophil clustering and dispersal at wounds in zebrafish 2019 ·Nature Communications ·(unknown), (unknown), (unknown), (unknown), Nicole Merten, (unknown), Elisabeth M. Busch‐Nentwich, Sarah V. Williams, (unknown), Evi Kostenis, Milka Sarris	49
5	Unique signalling connectivity of FGFR3-TACC3 oncoprotein revealed by quantitative phosphoproteomics and differential network analysis 2017 ·Oncotarget ·(unknown), Paul Ashford, Aurelio A. Moya‐García, (unknown), Mark Crawford, Sarah V. Williams, Margaret A. Knowles, Matilda Katan, Christine Orengo, Jasminka Godovac‐Zimmermann	11
6	A Place for Precision Medicine in Bladder Cancer: Targeting the Fgfrs 2016 ·Future Oncology ·Erica di Martino, Darren C. Tomlinson, Sarah V. Williams, Margaret A. Knowles	38
7	Landscape of activating cancer mutations in FGFR kinases and their differential responses to inhibitors in clinical use 2016 ·Oncotarget ·(unknown), Tom D. Bunney, Nethaji Thiyagarajan, Richard A. Norman, D. Ogg, J. Breed, Paul Ashford, (unknown), Mina Edwards, Sarah V. Williams, (unknown), Camilla Pang, Margaret A. Knowles, Alexander L. Breeze, Christine Orengo, Chris Phillips, Matilda Katan	91
8	The Effect of Mutations on Drug Sensitivity and Kinase Activity of Fibroblast Growth Factor Receptors: A Combined Experimental and Theoretical Study 2015 ·EBioMedicine ·Tom D. Bunney, Shunzhou Wan, Nethaji Thiyagarajan, Ludovico Sutto, Sarah V. Williams, Paul Ashford, Hans Koss, Margaret A. Knowles, Francesco Luigi Gervasio, Peter V. Coveney, Matilda Katan	59
9	Is uterine serous carcinoma a part of hereditary breast cancer syndrome? 2013 ·Journal of Clinical Oncology ·Saeed Rafii, (unknown), Sarah V. Williams, Jennifer Pascoe, James Nevin, Sudha Sundar	1
10	Oncogenic FGFR3 gene fusions in bladder cancer 2012 ·Human Molecular Genetics ·Sarah V. Williams, Carolyn D. Hurst, Margaret A. Knowles	282
11	Inactivation of the Rb pathway and overexpression of both isoforms of E2F3 are obligate events in bladder tumours with 6p22 amplification 2007 ·Oncogene ·(unknown), Darren C. Tomlinson, Sarah V. Williams, (unknown), Margaret A. Knowles	70
12	Assessment by M‐FISH of karyotypic complexity and cytogenetic evolution in bladder cancer in vitro 2005 ·Genes Chromosomes and Cancer ·Sarah V. Williams, Jacqui Adams, (unknown), Brenda Summersgill, Janet Shipley, Margaret A. Knowles	15
13	Endocrine therapy for early breast cancer 2004 ·Expert Review of Anticancer Therapy ·Syed A. Hussain, Sarah V. Williams, Andrea Stevens, Daniel Rea	7
14	Fine Mapping of Probes in the Adenomatous Polyposis Coli Region of Chromosome 5 by In Situ Hybridization 1991 ·Genes Chromosomes and Cancer ·Sarah V. Williams, T. Alwyn Jones, Sally Cottrell, Günther Zehetner, Liliana Varesco, (unknown), Huw Thomas, Paul A. Lawson, Ellen Solomon, Walter F. Bodmer, (unknown), Denise Sheer	16
15	Gene amplification accompanied by the loss of a chromosome containing the native allele and the appearance of the amplified DNA at a new chromosomal location. 1991 ·Proceedings of the National Academy of Sciences ·Édith Heard, Sarah V. Williams, Denise Sheer, Michael G. Fried	9

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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