Sarah Svirsky

831 total citations
19 papers, 417 citations indexed

About

Sarah Svirsky is a scholar working on Neurology, Physiology and Molecular Biology. According to data from OpenAlex, Sarah Svirsky has authored 19 papers receiving a total of 417 indexed citations (citations by other indexed papers that have themselves been cited), including 8 papers in Neurology, 7 papers in Physiology and 5 papers in Molecular Biology. Recurrent topics in Sarah Svirsky's work include Traumatic Brain Injury and Neurovascular Disturbances (8 papers), Alzheimer's disease research and treatments (6 papers) and Traumatic Brain Injury Research (5 papers). Sarah Svirsky is often cited by papers focused on Traumatic Brain Injury and Neurovascular Disturbances (8 papers), Alzheimer's disease research and treatments (6 papers) and Traumatic Brain Injury Research (5 papers). Sarah Svirsky collaborates with scholars based in United States, Australia and Germany. Sarah Svirsky's co-authors include Oksana Berezovska, Bradley T. Hyman, Kathryn Post, Muriel Arimon, Shuko Takeda, Jonathan D. Cherry, Russell D. Romeo, Thor D. Stein, Victor E. Alvarez and Ann C. McKee and has published in prestigious journals such as The Journal of Comparative Neurology, Scientific Reports and Neuroscience.

In The Last Decade

Sarah Svirsky

18 papers receiving 414 citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sarah Svirsky United States 11 163 109 95 67 57 19 417
Irena Majkutewicz Poland 9 119 0.7× 112 1.0× 76 0.8× 46 0.7× 79 1.4× 24 354
Zhongya Gu China 8 104 0.6× 143 1.3× 97 1.0× 48 0.7× 96 1.7× 12 408
Hualong Wang China 8 116 0.7× 135 1.2× 91 1.0× 43 0.6× 89 1.6× 15 358
Yunkai Xie China 10 80 0.5× 124 1.1× 56 0.6× 54 0.8× 80 1.4× 13 420
Jie Xia China 13 231 1.4× 147 1.3× 56 0.6× 72 1.1× 113 2.0× 24 525
Xin‐shang Wang China 18 168 1.0× 197 1.8× 140 1.5× 79 1.2× 84 1.5× 34 619
Colleen Pappas United States 15 202 1.2× 115 1.1× 125 1.3× 30 0.4× 63 1.1× 30 521
Helena Passarelli Giroud Joaquim Brazil 14 191 1.2× 150 1.4× 66 0.7× 27 0.4× 76 1.3× 36 476
Imane Lejri Switzerland 11 149 0.9× 144 1.3× 77 0.8× 34 0.5× 29 0.5× 17 403
Rafaella A. Gonçalves Brazil 11 254 1.6× 145 1.3× 101 1.1× 23 0.3× 42 0.7× 13 477

Countries citing papers authored by Sarah Svirsky

Since Specialization
Citations

This map shows the geographic impact of Sarah Svirsky's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sarah Svirsky with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sarah Svirsky more than expected).

Fields of papers citing papers by Sarah Svirsky

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sarah Svirsky. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sarah Svirsky. The network helps show where Sarah Svirsky may publish in the future.

Co-authorship network of co-authors of Sarah Svirsky

This figure shows the co-authorship network connecting the top 25 collaborators of Sarah Svirsky. A scholar is included among the top collaborators of Sarah Svirsky based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sarah Svirsky. Sarah Svirsky is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

19 of 19 papers shown
1.
Eagle, Shawn R., Sarah Svirsky, Ava M. Puccio, et al.. (2024). Predictive Blood Biomarkers of Targeted Intervention for Chronic Mental Health Symptoms following Traumatic Brain Injury. Journal of Neurotrauma. 42(5-6). e454–e460. 2 indexed citations
2.
Svirsky, Sarah, Jeremy Henchir, Ting Zhang, et al.. (2024). Viral-mediated increased hippocampal neurogranin modulate synapses at one month in a rat model of controlled cortical impact. Scientific Reports. 14(1). 28998–28998.
3.
Svirsky, Sarah, Jeremy Henchir, Youming Li, Shaun W. Carlson, & C. Edward Dixon. (2024). Temporal-Specific Sex and Injury-Dependent Changes on Neurogranin-Associated Synaptic Signaling After Controlled Cortical Impact in Rats. Molecular Neurobiology. 61(9). 7256–7268. 2 indexed citations
4.
Svirsky, Sarah, Youming Li, Jeremy Henchir, et al.. (2023). Experimental traumatic brain injury increases epichaperome formation. Neurobiology of Disease. 188. 106331–106331. 5 indexed citations
5.
6.
Roberts, Andrea L., et al.. (2022). Assessment of behavioral, neuroinflammatory, and histological responses in a model of rat repetitive mild fluid percussion injury at 2 weeks post-injury. Frontiers in Neurology. 13. 945735–945735. 4 indexed citations
7.
Kaufman, Sarah K., Sarah Svirsky, Jonathan D. Cherry, Ann C. McKee, & Marc I. Diamond. (2021). Tau seeding in chronic traumatic encephalopathy parallels disease severity. Acta Neuropathologica. 142(6). 951–960. 11 indexed citations
8.
Cherry, Jonathan D., Gaoyuan Meng, Sarah Daley, et al.. (2020). CCL2 is associated with microglia and macrophage recruitment in chronic traumatic encephalopathy. Journal of Neuroinflammation. 17(1). 370–370. 50 indexed citations
9.
Friedberg, Jacob, Nurgül Aytan, Jonathan D. Cherry, et al.. (2020). Associations between brain inflammatory profiles and human neuropathology are altered based on apolipoprotein E ε4 genotype. Scientific Reports. 10(1). 2924–2924. 47 indexed citations
10.
Shin, Hyun‐Soo, Sarah Svirsky, Cynthia M. Schumann, et al.. (2020). Putative dendritic correlates of chronic traumatic encephalopathy: A preliminary quantitative Golgi exploration. The Journal of Comparative Neurology. 529(7). 1308–1326. 3 indexed citations
11.
Svirsky, Sarah, et al.. (2019). Presenilin 1 increases association with synaptotagmin 1 during normal aging. Neurobiology of Aging. 86. 156–161. 5 indexed citations
12.
Svirsky, Sarah, Jeremy Henchir, Youming Li, et al.. (2019). Neurogranin Protein Expression Is Reduced after Controlled Cortical Impact in Rats. Journal of Neurotrauma. 37(7). 939–949. 14 indexed citations
13.
Morrison, Filomene G., Mark W. Miller, Erika J. Wolf, et al.. (2018). Reduced interleukin 1A gene expression in the dorsolateral prefrontal cortex of individuals with PTSD and depression. Neuroscience Letters. 692. 204–209. 30 indexed citations
14.
Maesako, Masato, Katarzyna Marta Zoltowska, Ksenia V. Kastanenka, et al.. (2017). Pathogenic PS1 phosphorylation at Ser367. eLife. 6. 19 indexed citations
15.
Kuzuya, Akira, Katarzyna Marta Zoltowska, Kathryn Post, et al.. (2016). Identification of the novel activity-driven interaction between synaptotagmin 1 and presenilin 1 links calcium, synapse, and amyloid beta. BMC Biology. 14(1). 25–25. 32 indexed citations
16.
Arimon, Muriel, Shuko Takeda, Kathryn Post, et al.. (2015). Oxidative stress and lipid peroxidation are upstream of amyloid pathology. Neurobiology of Disease. 84. 109–119. 120 indexed citations
17.
Romeo, Russell D., et al.. (2014). Pubertal shifts in adrenal responsiveness to stress and adrenocorticotropic hormone in male rats. Psychoneuroendocrinology. 42. 146–152. 34 indexed citations
18.
19.
Svirsky, Sarah, et al.. (1974). Neonatal hyperbilirubinemia and glucose-6-phosphate dehydrogenase deficiency.. PubMed. 9(11). 1547–52. 16 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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