Sarah L. Cole

5.9k total citations · 1 hit paper
14 papers, 4.5k citations indexed

About

Sarah L. Cole is a scholar working on Physiology, Pharmacology and Molecular Biology. According to data from OpenAlex, Sarah L. Cole has authored 14 papers receiving a total of 4.5k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Physiology, 6 papers in Pharmacology and 5 papers in Molecular Biology. Recurrent topics in Sarah L. Cole's work include Alzheimer's disease research and treatments (10 papers), Cholinesterase and Neurodegenerative Diseases (6 papers) and Computational Drug Discovery Methods (5 papers). Sarah L. Cole is often cited by papers focused on Alzheimer's disease research and treatments (10 papers), Cholinesterase and Neurodegenerative Diseases (6 papers) and Computational Drug Discovery Methods (5 papers). Sarah L. Cole collaborates with scholars based in United States, United Kingdom and Belgium. Sarah L. Cole's co-authors include Robert Vassar, R. Stephen Berry, John F. Disterhoft, Masuo Ohno, Erika Maus, Holly D. Oakley, Pei Shao, Linda Van Eldik, Angela Guillozet-Bongaarts and Sreemathi Logan and has published in prestigious journals such as Journal of Biological Chemistry, Neuron and Journal of Neuroscience.

In The Last Decade

Sarah L. Cole

14 papers receiving 4.5k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential Factors in Amyloid Plaque Formation

2006 2.5k citations Holly D. Oakley, Sarah L. Cole et al. Journal of Neuroscience profile →

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name	h						Papers	Cites
Sarah L. Cole United States	12	3.1k	1.6k	1.1k	1.1k	814	14	4.5k
Takeshi Kawarabayashi Japan	31	4.1k 1.3×	2.2k 1.4×	1.0k 1.0×	1.3k 1.2×	889 1.1×	129	5.7k
Yasuji Matsuoka United States	40	3.2k 1.0×	2.4k 1.5×	1.1k 1.0×	1.4k 1.4×	845 1.0×	80	5.9k
Nina E. Shepardson United States	7	4.3k 1.4×	1.9k 1.2×	980 0.9×	1.9k 1.7×	1.1k 1.4×	8	5.4k
Dick Terwel Belgium	36	2.6k 0.8×	1.6k 1.0×	1.3k 1.3×	1.1k 1.0×	531 0.7×	46	4.4k
Leigh A. Holcomb United States	16	2.5k 0.8×	1.3k 0.8×	623 0.6×	1.0k 0.9×	836 1.0×	20	3.7k
Holly D. Oakley United States	8	2.5k 0.8×	1.2k 0.7×	924 0.9×	933 0.9×	673 0.8×	10	3.6k
Mary Beth Finn United States	19	2.9k 0.9×	1.2k 0.7×	1.5k 1.4×	1.1k 1.1×	508 0.6×	23	5.1k
Laurent Pradier France	39	2.9k 0.9×	2.6k 1.6×	777 0.7×	1.8k 1.7×	815 1.0×	98	5.2k
Mark R. Emmerling United States	35	3.3k 1.0×	1.9k 1.2×	926 0.9×	674 0.6×	878 1.1×	63	5.0k
Steffen Roßner Germany	35	2.6k 0.8×	2.0k 1.2×	814 0.8×	1.4k 1.3×	840 1.0×	114	4.9k

Countries citing papers authored by Sarah L. Cole

Since Specialization

Citations

This map shows the geographic impact of Sarah L. Cole's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sarah L. Cole with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sarah L. Cole more than expected).

Fields of papers citing papers by Sarah L. Cole

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sarah L. Cole. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sarah L. Cole. The network helps show where Sarah L. Cole may publish in the future.

Co-authorship network of co-authors of Sarah L. Cole

This figure shows the co-authorship network connecting the top 25 collaborators of Sarah L. Cole. A scholar is included among the top collaborators of Sarah L. Cole based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sarah L. Cole. Sarah L. Cole is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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14 of 14 papers shown

Chadwick, Wayne, Stuart Maudsley, William M. Hull, et al.. (2023). The oDGal Mouse: A Novel, Physiologically Relevant Rodent Model of Sporadic Alzheimer’s Disease. International Journal of Molecular Sciences. 24(8). 6953–6953. 6 indexed citations

Sadleir, Katherine R., William A. Eimer, Sarah L. Cole, & Robert Vassar. (2015). Aβ reduction in BACE1 heterozygous null 5XFAD mice is associated with transgenic APP level. Molecular Neurodegeneration. 10(1). 1–1. 168 indexed citations

Cole, Sarah L. & Robert Vassar. (2008). Linking vascular disorders and Alzheimer's disease: Potential involvement of BACE1. Neurobiology of Aging. 30(10). 1535–1544. 32 indexed citations

Cole, Sarah L. & Robert Vassar. (2008). The Role of Amyloid Precursor Protein Processing by BACE1, the β-Secretase, in Alzheimer Disease Pathophysiology. Journal of Biological Chemistry. 283(44). 29621–29625. 212 indexed citations

O’Connor, Tracy, Katherine R. Sadleir, Erika Maus, et al.. (2008). Phosphorylation of the Translation Initiation Factor eIF2α Increases BACE1 Levels and Promotes Amyloidogenesis. Neuron. 60(6). 988–1009. 344 indexed citations

Liu, Qiang, Celina Zerbinatti, Juan Zhang, et al.. (2007). Amyloid Precursor Protein Regulates Brain Apolipoprotein E and Cholesterol Metabolism through Lipoprotein Receptor LRP1. Neuron. 56(1). 66–78. 306 indexed citations

Cole, Sarah L. & Robert Vassar. (2007). The Alzheimer's disease Beta-secretase enzyme, BACE1. Molecular Neurodegeneration. 2(1). 22–22. 412 indexed citations

Oakley, Holly D., Sarah L. Cole, Sreemathi Logan, et al.. (2006). Intraneuronal β-Amyloid Aggregates, Neurodegeneration, and Neuron Loss in Transgenic Mice with Five Familial Alzheimer's Disease Mutations: Potential Factors in Amyloid Plaque Formation. Journal of Neuroscience. 26(40). 10129–10140. 2544 indexed citations breakdown →

Cole, Sarah L. & Robert Vassar. (2006). Isoprenoids and Alzheimer's disease: A complex relationship. Neurobiology of Disease. 22(2). 209–222. 63 indexed citations

10.

Ohno, Masuo, Sarah L. Cole, Marina V. Yasvoina, et al.. (2006). BACE1 gene deletion prevents neuron loss and memory deficits in 5XFAD APP/PS1 transgenic mice. Neurobiology of Disease. 26(1). 134–145. 238 indexed citations

11.

Cole, Sarah L., et al.. (2005). Statins Cause Intracellular Accumulation of Amyloid Precursor Protein, β-Secretase-cleaved Fragments, and Amyloid β-Peptide via an Isoprenoid-dependent Mechanism. Journal of Biological Chemistry. 280(19). 18755–18770. 120 indexed citations

12.

Cole, Sarah L., et al.. (2004). O4-02-08 A tale of two statins: the effects of lovastatin and simvastatin on APP metabolism. Neurobiology of Aging. 25. S77–S77. 2 indexed citations

13.

Cole, Sarah L. & Marcus Schindler. (2000). Characterisation of somatostatin sst2 receptor splice variants. Journal of Physiology-Paris. 94(3-4). 217–237. 21 indexed citations

14.

Sirica, Alphonse E., et al.. (1994). A unique rat model of bile ductular hyperplasia in which liver is almost totally replaced with well-differentiated bile ductules.. PubMed. 144(6). 1257–68. 30 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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