Sara M. Reyna

1.9k total citations
24 papers, 1.6k citations indexed

About

Sara M. Reyna is a scholar working on Molecular Biology, Epidemiology and Physiology. According to data from OpenAlex, Sara M. Reyna has authored 24 papers receiving a total of 1.6k indexed citations (citations by other indexed papers that have themselves been cited), including 10 papers in Molecular Biology, 9 papers in Epidemiology and 7 papers in Physiology. Recurrent topics in Sara M. Reyna's work include Adipose Tissue and Metabolism (7 papers), Adipokines, Inflammation, and Metabolic Diseases (7 papers) and Metabolism, Diabetes, and Cancer (5 papers). Sara M. Reyna is often cited by papers focused on Adipose Tissue and Metabolism (7 papers), Adipokines, Inflammation, and Metabolic Diseases (7 papers) and Metabolism, Diabetes, and Cancer (5 papers). Sara M. Reyna collaborates with scholars based in United States, France and Italy. Sara M. Reyna's co-authors include Nicolas Musi, Ralph A. DeFronzo, Eugênio Cersósimo, Dawn K. Coletta, Christopher P. Jenkinson, Apiradee Sriwijitkamol, Phyllis A. Eagan, Paula K. Shireman, Linda M. McManus and Joel Michalek and has published in prestigious journals such as Journal of Biological Chemistry, The Journal of Immunology and Molecular and Cellular Biology.

In The Last Decade

Sara M. Reyna

23 papers receiving 1.5k citations

Peers — A (Enhanced Table)

Peers by citation overlap · career bar shows stage (early→late) cites · hero ref

Name h Career Trend Papers Cites
Sara M. Reyna United States 17 863 452 346 298 198 24 1.6k
Olivier Bourron France 20 527 0.6× 276 0.6× 232 0.7× 318 1.1× 282 1.4× 61 1.5k
Qi‐Qun Tang China 29 790 0.9× 691 1.5× 598 1.7× 157 0.5× 188 0.9× 59 1.8k
Bounleut Phanavanh United States 16 828 1.0× 578 1.3× 539 1.6× 148 0.5× 284 1.4× 23 1.7k
Phi Villageois France 17 856 1.0× 732 1.6× 463 1.3× 183 0.6× 71 0.4× 25 1.7k
Ioannis Habeos Greece 21 925 1.1× 423 0.9× 297 0.9× 201 0.7× 92 0.5× 36 1.9k
Kyoungmin Park United States 25 730 0.8× 295 0.7× 209 0.6× 212 0.7× 139 0.7× 48 1.7k
Lubna Al‐Khalili Sweden 19 951 1.1× 625 1.4× 227 0.7× 217 0.7× 52 0.3× 31 1.4k
Tetsuya Hosooka Japan 21 820 1.0× 470 1.0× 299 0.9× 272 0.9× 137 0.7× 44 1.5k
María Gabriela Morales Chile 26 1.1k 1.3× 407 0.9× 114 0.3× 286 1.0× 80 0.4× 42 1.8k
Mengliu Yang China 30 933 1.1× 767 1.7× 802 2.3× 380 1.3× 119 0.6× 94 2.5k

Countries citing papers authored by Sara M. Reyna

Since Specialization
Citations

This map shows the geographic impact of Sara M. Reyna's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Sara M. Reyna with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Sara M. Reyna more than expected).

Fields of papers citing papers by Sara M. Reyna

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Sara M. Reyna. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Sara M. Reyna. The network helps show where Sara M. Reyna may publish in the future.

Co-authorship network of co-authors of Sara M. Reyna

This figure shows the co-authorship network connecting the top 25 collaborators of Sara M. Reyna. A scholar is included among the top collaborators of Sara M. Reyna based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Sara M. Reyna. Sara M. Reyna is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

20 of 20 papers shown
1.
Zhu, Lin, Sara M. Reyna, Puntip Tantiwong, et al.. (2024). Short-term HIIT impacts HDL function differently in lean, obese, and diabetic subjects. Frontiers in Physiology. 15. 1423989–1423989. 1 indexed citations
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Reyna, Sara M., et al.. (2022). 205-LB: ERK Is Required for LPS-Induced TLR4 Internalization in Macrophages. Diabetes. 71(Supplement_1). 1 indexed citations
4.
Acevedo, Daniel, et al.. (2018). Role of ERK in IRF3-Mediated Immune Responses. Diabetes. 67(Supplement_1). 1 indexed citations
5.
Acevedo, Daniel, et al.. (2015). Nigella sativa influences GLUT4 through the AMPK pathway. The FASEB Journal. 29(S1). 3 indexed citations
6.
Muscogiuri, Giovanna, Adam B. Salmon, Cristina Aguayo‐Mazzucato, et al.. (2013). Genetic Disruption of SOD1 Gene Causes Glucose Intolerance and Impairs β-Cell Function. Diabetes. 62(12). 4201–4207. 37 indexed citations
7.
Reyna, Sara M., Puntip Tantiwong, Eugênio Cersósimo, et al.. (2013). Short-Term Exercise Training Improves Insulin Sensitivity but Does Not Inhibit Inflammatory Pathways in Immune Cells from Insulin-Resistant Subjects. Journal of Diabetes Research. 2013. 1–8. 22 indexed citations
8.
Reyna, Sara M., Jose C. Granados, Sung‐Jen Wei, et al.. (2012). Inhibition of Neddylation Represses Lipopolysaccharide-induced Proinflammatory Cytokine Production in Macrophage Cells. Journal of Biological Chemistry. 287(42). 35756–35767. 81 indexed citations
9.
Reyna, Sara M., et al.. (2011). Effects of insulin and oral anti‐diabetic agents on glucose metabolism, vascular dysfunction and skeletal muscle inflammation in type 2 diabetic subjects. Diabetes/Metabolism Research and Reviews. 27(4). 373–382. 30 indexed citations
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Guigas, Bruno, Kei Sakamoto, Nellie Taleux, et al.. (2008). Beyond AICA riboside: In search of new specific AMP‐activated protein kinase activators. IUBMB Life. 61(1). 18–26. 68 indexed citations
12.
Reyna, Sara M., Sangeeta Ghosh, Puntip Tantiwong, et al.. (2008). Elevated Toll-Like Receptor 4 Expression and Signaling in Muscle From Insulin-Resistant Subjects. Diabetes. 57(10). 2595–2602. 314 indexed citations
13.
Ochoa, Oscar, Dongxu Sun, Sara M. Reyna, et al.. (2007). Delayed angiogenesis and VEGF production in CCR2−/− mice during impaired skeletal muscle regeneration. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology. 293(2). R651–R661. 142 indexed citations
14.
Shireman, Paula K., et al.. (2006). MCP-1 Parallels Inflammatory and Regenerative Responses in Ischemic Muscle. Journal of Surgical Research. 134(1). 145–157. 52 indexed citations
15.
Contreras‐Shannon, Veronica, Oscar Ochoa, Sara M. Reyna, et al.. (2006). Fat accumulation with altered inflammation and regeneration in skeletal muscle of CCR2−/− mice following ischemic injury. American Journal of Physiology-Cell Physiology. 292(2). C953–C967. 135 indexed citations
16.
Luo, Moulun, et al.. (2006). Quantification of phosphorylation of insulin receptor substrate-1 by HPLC-ESI-MS/MS. Journal of the American Society for Mass Spectrometry. 17(4). 562–567. 20 indexed citations
17.
Luo, Moulun, Sara M. Reyna, Lishan Wang, et al.. (2005). Identification of Insulin Receptor Substrate 1 Serine/Threonine Phosphorylation Sites Using Mass Spectrometry Analysis: Regulatory Role of Serine 1223. Endocrinology. 146(10). 4410–4416. 47 indexed citations
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Tracy, Steven, et al.. (2000). Group B coxsackievirus myocarditis and pancreatitis: Connection between viral virulence phenotypes in mice. Journal of Medical Virology. 62(1). 70–81. 109 indexed citations
20.
Reyna, Sara M. & Keith A. Krolick. (2000). Chemokine Production by Rat Myocytes Exposed to Interferon-γ. Clinical Immunology. 94(2). 105–113. 25 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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