Samir N. Kelada

733 total citations
10 papers, 529 citations indexed

About

Samir N. Kelada is a scholar working on Molecular Biology, Cellular and Molecular Neuroscience and Neurology. According to data from OpenAlex, Samir N. Kelada has authored 10 papers receiving a total of 529 indexed citations (citations by other indexed papers that have themselves been cited), including 5 papers in Molecular Biology, 5 papers in Cellular and Molecular Neuroscience and 5 papers in Neurology. Recurrent topics in Samir N. Kelada's work include Parkinson's Disease Mechanisms and Treatments (5 papers), Glutathione Transferases and Polymorphisms (3 papers) and Nuclear Receptors and Signaling (3 papers). Samir N. Kelada is often cited by papers focused on Parkinson's Disease Mechanisms and Treatments (5 papers), Glutathione Transferases and Polymorphisms (3 papers) and Nuclear Receptors and Signaling (3 papers). Samir N. Kelada collaborates with scholars based in United States and Italy. Samir N. Kelada's co-authors include David L. Eaton, Harvey Checkoway, Paola Costa‐Mallen, Lucio G. Costa, Gary M. Franklin, Phillip D. Swanson, W. T. Longstreth, Nathaniel Rothman, Sophia Wang and Muin J. Khoury and has published in prestigious journals such as American Journal of Epidemiology, Environmental Health Perspectives and Human Molecular Genetics.

In The Last Decade

Samir N. Kelada

10 papers receiving 498 citations

Peers

Samir N. Kelada
Rima Woods United States
F. Trippi Italy
H. A. Peters United States
Mitchell R. Emerson United States
Yu‐Hsuan Chuang United States
Sára Bech Denmark
Rima Woods United States
Samir N. Kelada
Citations per year, relative to Samir N. Kelada Samir N. Kelada (= 1×) peers Rima Woods

Countries citing papers authored by Samir N. Kelada

Since Specialization
Citations

This map shows the geographic impact of Samir N. Kelada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Samir N. Kelada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Samir N. Kelada more than expected).

Fields of papers citing papers by Samir N. Kelada

Since Specialization
Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Samir N. Kelada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Samir N. Kelada. The network helps show where Samir N. Kelada may publish in the future.

Co-authorship network of co-authors of Samir N. Kelada

This figure shows the co-authorship network connecting the top 25 collaborators of Samir N. Kelada. A scholar is included among the top collaborators of Samir N. Kelada based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Samir N. Kelada. Samir N. Kelada is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

10 of 10 papers shown
1.
Kelada, Samir N., Harvey Checkoway, Sharon L. R. Kardia, et al.. (2006). 5′ and 3′ region variability in the dopamine transporter gene ( SLC6A3 ), pesticide exposure and Parkinson's disease risk: a hypothesis-generating study. Human Molecular Genetics. 15(20). 3055–3062. 44 indexed citations
2.
Costa‐Mallen, Paola, Samir N. Kelada, Lucio G. Costa, & Harvey Checkoway. (2005). Characterization of the in vitro transcriptional activity of polymorphic alleles of the human monoamine oxidase-B gene. Neuroscience Letters. 383(1-2). 171–175. 26 indexed citations
3.
Kelada, Samir N., Paola Costa‐Mallen, Harvey Checkoway, et al.. (2005). Dopamine transporter (SLC6A3) 5′ region haplotypes significantly affect transcriptional activity in vitro but are not associated with Parkinson's disease. Pharmacogenetics and Genomics. 15(9). 659–668. 44 indexed citations
4.
Costa‐Mallen, Paola, Zahra Afsharinejad, Samir N. Kelada, et al.. (2004). DNA sequence analysis of monoamine oxidase B gene coding and promoter regions in Parkinson's disease cases and unrelated controls. Movement Disorders. 19(1). 76–83. 9 indexed citations
5.
Costa, Lucio G., Samir N. Kelada, Paola Costa‐Mallen, et al.. (2004). Paraoxonase 2 (PON2) polymorphisms and Parkinson's disease. Neuroscience Research Communications. 34(3). 130–135. 3 indexed citations
6.
Kelada, Samir N., Patricia L. Stapleton, Federico M. Farin, et al.. (2003). Glutathione S-transferase M1, T1, and P1 Polymorphisms and Parkinson's Disease. Neuroscience Letters. 337(1). 5–8. 60 indexed citations
7.
Kelada, Samir N., David L. Eaton, Sophia Wang, Nathaniel Rothman, & Muin J. Khoury. (2003). The role of genetic polymorphisms in environmental health.. Environmental Health Perspectives. 111(8). 1055–1064. 88 indexed citations
8.
Kelada, Samir N., Paola Costa‐Mallen, Lucio G. Costa, et al.. (2002). Gender Difference in the Interaction of Smoking and Monoamine Oxidase B Intron 13 Genotype in Parkinson’s Disease. NeuroToxicology. 23(4-5). 515–519. 36 indexed citations
9.
Kelada, Samir N.. (2001). delta-Aminolevulinic Acid Dehydratase Genotype and Lead Toxicity: A HuGE Review. American Journal of Epidemiology. 154(1). 1–13. 160 indexed citations
10.
Kelada, Samir N., Sharon L. R. Kardia, A. H. C. Walker, et al.. (2000). The glutathione S-transferase-mu and -theta genotypes in the etiology of prostate cancer: genotype-environment interactions with smoking.. PubMed. 9(12). 1329–34. 59 indexed citations

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

Explore authors with similar magnitude of impact

Rankless by CCL
2026