Samir N. Kelada
- Molecular Biology
- Health, Toxicology and Mutagenesis top 10%
- Neurology top 10%
- Cellular and Molecular Neuroscience
- Nutrition and Dietetics top 10%
- Co-authors
- David L. EatonHarvey CheckowayPaola Costa‐MallenLucio G. CostaGary M. FranklinPhillip D. SwansonW. T. LongstrethNathaniel Rothman
- Topics
- Parkinson's Disease Mechanisms and Treatments (5 papers)Glutathione Transferases and Polymorphisms (3 papers)Nuclear Receptors and Signaling (3 papers)
- Partner nations
- United StatesItaly
In The Last Decade
Samir N. Kelada
10 papers receiving 498 citations
Peers
Comparison fields: 5 of 87
- Molecular Biology 192
- Health, Toxicology and Mutagenesis 143
- Neurology 106
- Cellular and Molecular Neuroscience 99
- Nutrition and Dietetics 94
Countries citing papers authored by Samir N. Kelada
This map shows the geographic impact of Samir N. Kelada's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Samir N. Kelada with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Samir N. Kelada more than expected).
Fields of papers citing papers by Samir N. Kelada
This network shows the impact of papers produced by Samir N. Kelada. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Samir N. Kelada. The network helps show where Samir N. Kelada may publish in the future.
Co-authorship network of co-authors of Samir N. Kelada
This figure shows the co-authorship network connecting the top 25 collaborators of Samir N. Kelada. A scholar is included among the top collaborators of Samir N. Kelada based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Samir N. Kelada. Samir N. Kelada is excluded from the visualization to improve readability, since they are connected to all nodes in the network.
All Works
| # | Work | Indexed citations |
|---|---|---|
| 1 | 44 | |
| 2 | 26 | |
| 3 | 44 | |
| 4 | 9 | |
| 5 | 3 | |
| 6 | 60 | |
| 7 | 88 | |
| 8 | 36 | |
| 9 | 160 | |
| 10 | The glutathione S-transferase-mu and -theta genotypes in the etiology of prostate cancer: genotype-environment interactions with smoking. | 59 |
About Samir N. Kelada
Samir N. Kelada is a scholar working on Neurology, Cellular and Molecular Neuroscience and Clinical Biochemistry, having authored 10 papers that have together received 529 indexed citations. Recurring topics across this work include Parkinson's Disease Mechanisms and Treatments (5 papers), Glutathione Transferases and Polymorphisms (3 papers) and Nuclear Receptors and Signaling (3 papers). The work is most often cited by research in Health, Toxicology and Mutagenesis (143 citations), Neurology (106 citations) and Nutrition and Dietetics (94 citations). Samir N. Kelada has collaborated with scholars based in United States and Italy. Frequent co-authors include David L. Eaton, Harvey Checkoway, Paola Costa‐Mallen, Lucio G. Costa, Gary M. Franklin, Phillip D. Swanson, W. T. Longstreth, Nathaniel Rothman, Sophia Wang and Muin J. Khoury. Their work appears in journals such as American Journal of Epidemiology, Environmental Health Perspectives and Human Molecular Genetics.
Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.