Salem Chouaı̈b

21.9k total citations · 1 hit paper
328 papers, 17.2k citations indexed

About

Salem Chouaı̈b is a scholar working on Immunology, Oncology and Molecular Biology. According to data from OpenAlex, Salem Chouaı̈b has authored 328 papers receiving a total of 17.2k indexed citations (citations by other indexed papers that have themselves been cited), including 229 papers in Immunology, 118 papers in Oncology and 94 papers in Molecular Biology. Recurrent topics in Salem Chouaı̈b's work include Immune Cell Function and Interaction (119 papers), Immunotherapy and Immune Responses (74 papers) and T-cell and B-cell Immunology (58 papers). Salem Chouaı̈b is often cited by papers focused on Immune Cell Function and Interaction (119 papers), Immunotherapy and Immune Responses (74 papers) and T-cell and B-cell Immunology (58 papers). Salem Chouaı̈b collaborates with scholars based in France, United Arab Emirates and United States. Salem Chouaı̈b's co-authors include Muhammad Zaeem Noman, Bassam Janji, Meriem Hasmim, Fathia Mami‐Chouaib, Vincenzo Bronte, Philippe Dessen, Stéphane Terry, Saoussen Karray, Giacomo Desantis and Jérôme Thiery and has published in prestigious journals such as Proceedings of the National Academy of Sciences, Journal of Biological Chemistry and Journal of Clinical Investigation.

In The Last Decade

Salem Chouaı̈b

324 papers receiving 16.9k citations

Hit Papers

align trajectories

What are hit papers?

Hit papers significantly outperform the citation benchmark for their cohort. A paper qualifies if it has ≥500 total citations, achieves ≥1.5× the top-1% citation threshold for papers in the same subfield and year (this is the minimum needed to enter the top 1%, not the average within it), or reaches the top citation threshold in at least one of its specific research topics.

PD-L1 is a novel direct target of HIF-1α, and its blockade under hypoxia enhanced MDSC-mediated T cell activation

2014 1.7k citations Muhammad Zaeem Noman, Bassam Janji et al. profile →

Peers

Countries citing papers authored by Salem Chouaı̈b

Since Specialization

Citations

This map shows the geographic impact of Salem Chouaı̈b's research. It shows the number of citations coming from papers published by authors working in each country. You can also color the map by specialization and compare the number of citations received by Salem Chouaı̈b with the expected number of citations based on a country's size and research output (numbers larger than one mean the country cites Salem Chouaı̈b more than expected).

Fields of papers citing papers by Salem Chouaı̈b

Since Specialization

Physical SciencesHealth SciencesLife SciencesSocial Sciences

This network shows the impact of papers produced by Salem Chouaı̈b. Nodes represent research fields, and links connect fields that are likely to share authors. Colored nodes show fields that tend to cite the papers produced by Salem Chouaı̈b. The network helps show where Salem Chouaı̈b may publish in the future.

Co-authorship network of co-authors of Salem Chouaı̈b

This figure shows the co-authorship network connecting the top 25 collaborators of Salem Chouaı̈b. A scholar is included among the top collaborators of Salem Chouaı̈b based on the total number of citations received by their joint publications. Widths of edges represent the number of papers authors have co-authored together. Node borders signify the number of papers an author published with Salem Chouaı̈b. Salem Chouaı̈b is excluded from the visualization to improve readability, since they are connected to all nodes in the network.

All Works

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20 of 20 papers shown

#	Work	Indexed citations
1	Evofosfamide Enhances Sensitivity of Breast Cancer Cells to Apoptosis and Natural-Killer-Cell-Mediated Cytotoxicity Under Hypoxic Conditions 2025 ·Cancers ·Shubhankar Das, Goutham Hassan Venkatesh, Walid El‐Sayed, Raefa Abou Khouzam, (unknown), (unknown), (unknown), Rania Faouzi Zaarour, Salem Chouaı̈b	1
2	Hypoxia, a Targetable Culprit to Counter Pancreatic Cancer Resistance to Therapy 2023 ·Cancers ·Raefa Abou Khouzam, Jean‐Maríe Lehn, (unknown), Pierre–Alain Clavien, Michael B. Wallace, Michel Ducreux, Përparim Limani, Salem Chouaı̈b	17
3	Designing Organoid Models to Monitor Cancer Progression, Plasticity and Resistance: The Right Set Up for the Right Question 2022 ·Cancers ·(unknown), (unknown), (unknown), Stéphane Terry, Salem Chouaı̈b, Pierre Savagner	2
4	Cancer stem-like cells evade CD8⁺CD103⁺ tumor-resident memory T (T_RM) lymphocytes by initiating an epithelial-to-mesenchymal transition program in a human lung tumor model 2022 ·Journal for ImmunoTherapy of Cancer ·Stéphanie Corgnac, (unknown), Gwendoline Gros, (unknown), Stéphane Terry, Salem Chouaı̈b, Marc Deloger, Fathia Mami‐Chouaib	27
5	Tumor hypoxia: an important regulator of tumor progression or a potential modulator of tumor immunogenicity? 2021 ·OncoImmunology ·Goutham Hassan Venkatesh, Raefa Abou Khouzam, Walid El‐Sayed, (unknown), Stéphane Terry, Salem Chouaı̈b	18
6	Role of Hypoxia-Mediated Autophagy in Tumor Cell Death and Survival 2021 ·Cancers ·Rania Faouzi Zaarour, Bilal Azakir, (unknown), (unknown), (unknown), Agnete S. T. Engelsen, Jérôme Thiery, Colin Jamora, Salem Chouaı̈b	68
7	Nanomedicine-based cancer immunotherapy: recent trends and future perspectives 2021 ·Cancer Gene Therapy ·Vinoth‐Kumar Lakshmanan, (unknown), P. Gopinath, Shreesh Ojha, Sen Lian, Ajeet Kaushik, (unknown), (unknown), Sadras Panchatcharam Thyagarajan, Young Do Jung, Salem Chouaı̈b	60
8	Actin Cytoskeleton Remodeling Drives Breast Cancer Cell Escape from Natural Killer–Mediated Cytotoxicity 2018 ·Cancer Research ·(unknown), Hannah Wurzer, Coralie L. Guérin, Céline Hoffmann, Flora Moreau, Xianqing Mao, Joshua Brown-Clay, (unknown), (unknown), Monika Dieterle, Jean Paul Thiery, Salem Chouaı̈b, Guy Berchem, Bassam Janji, Clément Thomas	104
9	Human preprocalcitonin self-antigen generates TAP-dependent and -independent epitopes triggering optimised T-cell responses toward immune-escaped tumours 2018 ·Nature Communications ·Aurélie Durgeau, Yasemin Virk, Gwendoline Gros, (unknown), Stéphanie Corgnac, (unknown), Jérôme Salmon, Julien Adam, Vincent de Montpréville, Pierre Validire, Soldano Ferrone, Salem Chouaı̈b, Alexander Eggermont, Jean‐Charles Soria, François A. Lemonnier, Éric Tartour, Nathalie Chaput, Benjamin Besse, Fathia Mami‐Chouaib	21
10	Cutting Edge: NANOG Activates Autophagy under Hypoxic Stress by Binding to BNIP3L Promoter 2017 ·The Journal of Immunology ·Meriem Hasmim, Bassam Janji, Mehdi Khaled, Muhammad Zaeem Noman, Fawzia Louache, Didier Bordereaux, (unknown), Véronique Baud, Fathia Mami‐Chouaib, Salem Chouaı̈b	31
11	TCR-engineered T cells to treat tumors: Seeing but not touching? 2016 ·Seminars in Immunology ·Reno Debets, Emmanuel Donnadieu, Salem Chouaı̈b, George Coukos	57
12	Fas Ligand Deficiency Impairs Tumor Immunity by Promoting an Accumulation of Monocytic Myeloid-Derived Suppressor Cells 2015 ·Cancer Research ·Sanam Peyvandi, Stéphanie Buart, Boubekeur Samah, Marie Vétizou, Yanyan Zhang, (unknown), (unknown), Salem Chouaı̈b, Karim Benihoud, Fawzia Louache, Saoussen Karray	25
13	Tumor-Promoting Effects of Myeloid-Derived Suppressor Cells Are Potentiated by Hypoxia-Induced Expression of miR-210 2015 ·Cancer Research ·Muhammad Zaeem Noman, Bassam Janji, Shijun Hu, Joseph C. Wu, Fabio Martelli, Vincenzo Bronte, Salem Chouaı̈b	123
14	Epithelial-to-Mesenchymal Transition and Autophagy Induction in Breast Carcinoma Promote Escape from T-cell–Mediated Lysis 2013 ·Cancer Research ·Intissar Akalay, Bassam Janji, Meriem Hasmim, Muhammad Zaeem Noman, Fabrice André, Patricia de Crémoux, Philippe Bertheau, Cécile Badoual, Philippe Vielh, Annette K. Larsen, Michèle Sabbah, Tuan Zea Tan, (unknown), (unknown), Jean Paul Thiery, Fathia Mami‐Chouaib, Salem Chouaı̈b	247
15	Hypoxia-Inducible miR-210 Regulates the Susceptibility of Tumor Cells to Lysis by Cytotoxic T Cells 2012 ·Cancer Research ·Muhammad Zaeem Noman, Stéphanie Buart, Pedro Romero, (unknown), Bassam Janji, Bernard Mari, Fathia Mami‐Chouaib, Salem Chouaı̈b	160
16	Blocking Hypoxia-Induced Autophagy in Tumors Restores Cytotoxic T-Cell Activity and Promotes Regression 2011 ·Cancer Research ·Muhammad Zaeem Noman, Bassam Janji, Bożena Kamińska, Kris Van Moer, Sandrine Pierson, Piotr Przanowski, Stéphanie Buart, Guy Berchem, Pedro Romero, Fathia Mami‐Chouaib, Salem Chouaı̈b	204
17	Intratumoral Induction of CD103 Triggers Tumor-Specific CTL Function and CCR5-Dependent T-Cell Retention 2009 ·Cancer Research ·Katarzyna Franciszkiewicz, Audrey Le Floc’h, Abdelali Jalil, Frédéric Vigant, Thomas Robert, Isabelle Vergnon, Andrzej Maćkiewicz, Karim Benihoud, Pierre Validire, Salem Chouaı̈b, Christophe Combadière, Fathia Mami‐Chouaib	73
18	Human Renal Cancer Cells Express a Novel Membrane-Bound Interleukin-15 that Induces, in Response to the Soluble Interleukin-15 Receptor α Chain, Epithelial-to-Mesenchymal Transition 2009 ·Cancer Research ·(unknown), Julien Giron‐Michel, Yanhong Gu, Aurélie Perier, Massimo Giuliani, Anne Caignard, Aurore Devocelle, Silvano Ferrini, Marina Fabbi, Bernard Charpentier, Andreas Ludwig, Salem Chouaı̈b, Bruno Azzarone, Pierre Eid	40
19	ICAM-1 Has a Critical Role in the Regulation of Metastatic Melanoma Tumor Susceptibility to CTL Lysis by Interfering with PI3K/AKT Pathway 2008 ·Cancer Research ·Ahmed Hamaï, Franck Meslin, Houssem Benlalam, Abdelali Jalil, Maryam Mehrpour, Florence Faure, Yann Lécluse, (unknown), Marie‐Françoise Avril, Caroline Robert, Salem Chouaı̈b	61
20	Two opposite signaling outputs are driven by the KIR2DL1 receptor in human CD4+ T cells 2008 ·Blood ·(unknown), Abdelali Jalil, Sylvie Da Rocha, Christophe Pichon, Salem Chouaı̈b, Georges Bismuth, Anne Caignard	19

Rankless uses publication and citation data sourced from OpenAlex, an open and comprehensive bibliographic database. While OpenAlex provides broad and valuable coverage of the global research landscape, it—like all bibliographic datasets—has inherent limitations. These include incomplete records, variations in author disambiguation, differences in journal indexing, and delays in data updates. As a result, some metrics and network relationships displayed in Rankless may not fully capture the entirety of a scholar's output or impact.

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